Pathophysiology of septic acute kidney injury: what do we really know?
about
Role of inflammation and infection in the pathogenesis of human acute liver failure: Clinical implications for monitoring and therapyA Pathophysiological Insight into Sepsis and Its Correlation with Postmortem DiagnosisSeptic acute kidney injury: the culprit is inflammatory apoptosis rather than ischemic necrosisRenal oxygenation and haemodynamics in acute kidney injury and chronic kidney diseaseMicrocirculatory dysfunction and tissue oxygenation in critical illnessTLR2, TLR4 and the MYD88 signaling pathway are crucial for neutrophil migration in acute kidney injury induced by sepsisAnimal models of sepsis and sepsis-induced kidney injuryHemodynamic variables and progression of acute kidney injury in critically ill patients with severe sepsis: data from the prospective observational FINNAKI study.Renal haemodynamic, microcirculatory, metabolic and histopathological responses to peritonitis-induced septic shock in pigs.Platelet Consumption and Filter Clotting Using Two Different Membrane Sizes during Continuous Venovenous Haemodiafiltration in the Intensive Care Unit.Association of inflammatory and endothelial cell activation biomarkers with acute kidney injury after sepsis.Molecular differences in susceptibility of the kidney to sepsis-induced kidney injuryUrinary output and fractional excretion of sodium and urea as indicators of transient versus intrinsic acute kidney injury during early sepsis.Prevention of acute kidney injury and protection of renal function in the intensive care unit: update 2017 : Expert opinion of the Working Group on Prevention, AKI section, European Society of Intensive Care Medicine.Injurious mechanical ventilation causes kidney apoptosis and dysfunction during sepsis but not after intra-tracheal acid instillation: an experimental studyUrinary cystatin C is diagnostic of acute kidney injury and sepsis, and predicts mortality in the intensive care unit.Searching for mechanisms that matter in early septic acute kidney injury: an experimental studyUrine biochemistry in septic and non-septic acute kidney injury: a prospective observational studyPrognosis of AKI in malignant diseases with and without sepsis.Renal neutrophil gelatinase associated lipocalin expression in lipopolysaccharide-induced acute kidney injury in the rat.A multicentre study of acute kidney injury in severe sepsis and septic shock: association with inflammatory phenotype and HLA genotypeSuppression of mitochondrial biogenesis through toll-like receptor 4-dependent mitogen-activated protein kinase kinase/extracellular signal-regulated kinase signaling in endotoxin-induced acute kidney injury.Multiple organ dysfunction syndrome in humans and animals.A clinically applicable porcine model of septic and ischemia/reperfusion-induced shock and multiple organ injury.Role of surfactant proteins A and D in sepsis-induced acute kidney injury.Septic versus non-septic acute kidney injury in critically ill patients: characteristics and clinical outcomes.Modulation of early inflammatory response by different balanced and non-balanced colloids and crystalloids in a rodent model of endotoxemiaSirt1 deletion leads to enhanced inflammation and aggravates endotoxin-induced acute kidney injuryComputational approaches for translational clinical research in disease progression.Relation between mean arterial pressure and renal function in the early phase of shock: a prospective, explorative cohort study.Resveratrol attenuates lipopolysaccharide-induced acute kidney injury by suppressing inflammation driven by macrophagesXenon Protects Against Septic Acute Kidney Injury via miR-21 Target Signaling Pathway.Hospital Admission following Acute Kidney Injury in Kidney Transplant Recipients Is Associated with a Negative Impact on Graft Function after 1-Year.Nicotinic acetylcholine receptor agonists attenuate septic acute kidney injury in mice by suppressing inflammation and proteasome activity.Development of oxidative stress in the peritubular capillary microenvironment mediates sepsis-induced renal microcirculatory failure and acute kidney injury.Inflammatory Kidney and Liver Tissue Response to Different Hydroxyethylstarch (HES) Preparations in a Rat Model of Early Sepsis.Identification of candidate serum biomarkers for severe septic shock-associated kidney injury via microarray.Apoptosis and acute kidney injuryFluid overload and survival in critically ill patients with acute kidney injury receiving continuous renal replacement therapy.Differentially expressed miRNAs in sepsis-induced acute kidney injury target oxidative stress and mitochondrial dysfunction pathways
P2860
Q26741218-DBA97CA7-3BBD-4627-87C3-DB16E011B08BQ26745458-8D5C9629-9BC5-4536-97DC-EF748CFA5130Q26853638-AE79F510-70C6-4928-A444-EBD39B7D0FB9Q26996101-EFE9151C-F81F-4056-A478-50715331F0DBQ27008088-050D2415-31CD-4887-A8D7-78F04DEF5C2CQ27302311-286CD7F6-788E-4401-B23D-39EA07363047Q30490536-A05162FB-ACA2-41F9-AF6B-14AF83D3FD29Q30713446-1C6BB8F1-52D9-4F94-BBC9-B0141554905CQ33395875-C1A90EA1-D1A6-4B42-A89A-CE83084BCF3AQ33415569-0EBF73DC-DCBA-478E-8360-DD444A6FF8E1Q33600169-8CA264F5-DE11-4975-980C-31BE9902A34DQ33749902-54908746-32C5-49DE-AAE9-7B4CB1AFFFEEQ33753534-33FBE2CF-6914-4D33-8B5D-17AE05589C09Q33843623-3F279D95-1A26-4522-B407-1BD6CD7B9175Q33991892-14500261-1897-4590-9A18-8A5BBE304B8FQ34026824-CD091487-A818-4E24-9D58-5077CF1A8531Q34059264-A725347B-410B-4F8C-818A-9DC288B18677Q34093502-4BBAADE2-4634-4A5C-B29D-CFFB94A6B088Q34245768-D756BFA7-1DF9-4D6B-876D-E2DF680E9368Q34260466-39C729D2-1C79-4166-9AA2-EC8BAE956F2EQ34305100-876F30CB-DF96-4D05-8B3C-928D125E11F2Q34453628-737DEF49-5EF1-4094-BDFA-F52A4F28FB4CQ34661567-D5320964-9670-40BE-ACE9-32CC16F49BEDQ34699749-86DEF61F-6102-459D-A945-835E89775C56Q34725649-2770CCC3-9992-476E-A536-E4246F5DD78FQ35006028-560E0E6A-4833-4384-BA35-AD761DED5AF7Q35141682-B7D80DA1-EDFB-4C3C-B4D0-5312FA3D2988Q35181466-33C554D7-A3AC-4947-8A33-57FFF7EA0ED5Q35388120-D132E37A-EDED-49B7-9144-43660C02CC6DQ35558284-25DF8B2A-53C0-4934-9FA4-4302AA37CCD7Q35572350-B679DE6C-174D-45DB-808A-424E5C70F7E7Q35742994-65B5AFF8-EAE6-4E88-BBCA-C00AC89E7434Q35791488-C72E0796-489B-4A55-B1B4-C7C64D348C7BQ35941896-AD21416A-AB84-4E18-8692-F6E14CBE27F8Q35951888-13185FE4-E2A2-4629-8499-84E9D26E9360Q35960658-A54F0FCA-B691-467D-BA25-266F39DD8024Q36073104-E34AE28B-2866-45DD-BC4A-EE9DFCAA17B0Q36224335-7AD252F7-9B8C-462E-9173-448D9190521FQ36280023-E2557AAB-D8DC-4EB7-AF35-7526934B6AFFQ36308978-15296BE2-7A41-4152-9CE9-B94503B3A5BA
P2860
Pathophysiology of septic acute kidney injury: what do we really know?
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on April 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Pathophysiology of septic acute kidney injury: what do we really know?
@en
Pathophysiology of septic acute kidney injury: what do we really know?
@nl
type
label
Pathophysiology of septic acute kidney injury: what do we really know?
@en
Pathophysiology of septic acute kidney injury: what do we really know?
@nl
prefLabel
Pathophysiology of septic acute kidney injury: what do we really know?
@en
Pathophysiology of septic acute kidney injury: what do we really know?
@nl
P2093
P1476
Pathophysiology of septic acute kidney injury: what do we really know?
@en
P2093
Christoph Langenberg
Sean M Bagshaw
Takao Saotome
P304
P356
10.1097/CCM.0B013E318168CCD5
P407
P433
P577
2008-04-01T00:00:00Z