New insights into the function of the Wilms tumor suppressor gene WT1 in podocytes.
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PINCH1 is transcriptional regulator in podocytes that interacts with WT1 and represses podocalyxin expressionOverexpression of VEGF165b in podocytes reduces glomerular permeability.New perspectives on the renal slit diaphragm protein podocin.A novel WT1 gene mutation in a three-generation family with progressive isolated focal segmental glomerulosclerosis.Molecular genetic analysis of podocyte genes in focal segmental glomerulosclerosis--a review.Renal malformations associated with mutations of developmental genes: messages from the clinic.Blockade of Wnt/β-catenin signaling by paricalcitol ameliorates proteinuria and kidney injury.Intrauterine growth restriction leads to a dysregulation of Wilms' tumour supressor gene 1 (WT1) and to early podocyte alterations.Hepatocyte growth factor signaling ameliorates podocyte injury and proteinuria.Mutual antagonism of Wilms' tumor 1 and β-catenin dictates podocyte health and disease.A novel missense mutation of Wilms' Tumor 1 causes autosomal dominant FSGS.Progress in pathogenesis of proteinuria.19q13.11 cryptic deletion: description of two new cases and indication for a role of WTIP haploinsufficiency in hypospadiasDecreased transcriptional activity of calcium-sensing receptor gene promoter 1 is associated with calcium nephrolithiasis.Ubiquitin specific protease 18 (Usp18) is a WT1 transcriptional targetA biomimetic gelatin-based platform elicits a pro-differentiation effect on podocytes through mechanotransduction.Glomerular diseases: genetic causes and future therapeutics.WT1 in disease: shifting the epithelial-mesenchymal balance.One hundred ways to kill a podocyte.Wilms Tumor Suppressor, WT1, Cooperates with MicroRNA-26a and MicroRNA-101 to Suppress Translation of the Polycomb Protein, EZH2, in Mesenchymal Stem Cells.Adoptive transfer of genetically engineered WT1-specific cytotoxic T lymphocytes does not induce renal injury.Genetic Syndromes Affecting Kidney Development.Hormone supplying renal cell sheet in vivo produced by tissue engineering technology.GSK3β inactivation in podocytes results in decreased phosphorylation of p70S6K accompanied by cytoskeletal rearrangements and inhibited motility.Nephron, Wilms' tumor-1 (WT1), and synaptopodin expression in developing podocytes of miceHsf-1 affects podocyte markers NPHS1, NPHS2 and WT1 in a transgenic mouse model of TTRVal30Met-related amyloidosis.Discordant expression of a new WT1 gene mutation in a family with monozygotic twins presenting with congenital nephrotic syndrome.Genotype-phenotype analysis of pediatric patients with WT1 glomerulopathy.
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New insights into the function of the Wilms tumor suppressor gene WT1 in podocytes.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 02 April 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
New insights into the function of the Wilms tumor suppressor gene WT1 in podocytes.
@en
New insights into the function of the Wilms tumor suppressor gene WT1 in podocytes.
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type
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New insights into the function of the Wilms tumor suppressor gene WT1 in podocytes.
@en
New insights into the function of the Wilms tumor suppressor gene WT1 in podocytes.
@nl
prefLabel
New insights into the function of the Wilms tumor suppressor gene WT1 in podocytes.
@en
New insights into the function of the Wilms tumor suppressor gene WT1 in podocytes.
@nl
P2093
P2860
P1476
New insights into the function of the Wilms tumor suppressor gene WT1 in podocytes.
@en
P2093
Avril A Morrison
Michael R Ladomery
Moin A Saleem
Rebecca L Viney
P2860
P356
10.1152/AJPRENAL.00597.2007
P577
2008-04-02T00:00:00Z