Caffeic acid phenethyl ester suppresses the proliferation of human prostate cancer cells through inhibition of p70S6K and Akt signaling networks. - Wikidata - awgldk - TriplyDB

Caffeic acid phenethyl ester suppresses the proliferation of human prostate cancer cells through inhibition of p70S6K and Akt signaling networks.

Caffeic acid phenethyl ester suppresses the proliferation of human prostate cancer cells through inhibition of p70S6K and Akt signaling networks.

http://www.wikidata.org/entity/Q37130094

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Recent progress of propolis for its biological and chemical compositions and its botanical origin Caffeic Acid phenethyl ester is a potential therapeutic agent for oral cancer Dietary polyphenols in prevention and treatment of prostate cancer Caffeic acid derivatives inhibit the growth of colon cancer: involvement of the PI3-K/Akt and AMPK signaling pathways Androgen suppresses the proliferation of androgen receptor-positive castration-resistant prostate cancer cells via inhibition of Cdk2, CyclinA, and Skp2.Difference in protein expression profile and chemotherapy drugs response of different progression stages of LNCaP sublines and other human prostate cancer cells Elevation of soluble guanylate cyclase suppresses proliferation and survival of human breast cancer cells.Propolis: A Complex Natural Product with a Plethora of Biological Activities That Can Be Explored for Drug Development.Caffeic acid phenethyl ester induced cell cycle arrest and growth inhibition in androgen-independent prostate cancer cells via regulation of Skp2, p53, p21Cip1 and p27Kip1.The DIONESUS algorithm provides scalable and accurate reconstruction of dynamic phosphoproteomic networks to reveal new drug targets.AKT3 promotes prostate cancer proliferation cells through regulation of Akt, B-Raf, and TSC1/TSC2.Caffeic Acid phenethyl ester inhibits oral cancer cell metastasis by regulating matrix metalloproteinase-2 and the mitogen-activated protein kinase pathway.Caffeic Acid phenethyl ester as a potential treatment for advanced prostate cancer targeting akt signaling Caffeic acid phenethyl ester suppresses proliferation and survival of TW2.6 human oral cancer cells via inhibition of Akt signaling Cholestane-3β, 5α, 6β-triol suppresses proliferation, migration, and invasion of human prostate cancer cells Caffeic acid phenethyl ester suppresses melanoma tumor growth by inhibiting PI3K/AKT/XIAP pathway.CAPE suppresses migration and invasion of prostate cancer cells via activation of non-canonical Wnt signaling.In vivo and in vitro antıneoplastic actions of caffeic acid phenethyl ester (CAPE): therapeutic perspectives.Bioactivity and chemical synthesis of caffeic acid phenethyl ester and its derivatives.Emerging Adjuvant Therapy for Cancer: Propolis and its Constituents.Genome Expression Profiling-Based Identification and Administration Efficacy of Host-Directed Antimicrobial Drugs against Respiratory Infection by Nontypeable Haemophilus influenzae.Saurolactam Inhibits Proliferation, Migration, and Invasion of Human Osteosarcoma Cells.Identification and validation of genetic variants that influence transcription factor and cell signaling protein levels.Microwestern Arrays for Systems-Level Analysis of SH2 Domain-Containing Proteins.Caffeic acid phenethyl ester synergistically enhances docetaxel and paclitaxel cytotoxicity in prostate cancer cells.Caffeic acid-conjugated chitosan derivatives and their anti-tumor activity.Dietary Intervention by Phytochemicals and Their Role in Modulating Coding and Non-Coding Genes in Cancer.Caffeic acid phenethyl ester and therapeutic potentials Engineering E. coli for caffeic acid biosynthesis from renewable sugars.Antiproliferative and Apoptotic Effects of Shemamruthaa, a Herbal Preparation, in 7,12-Dimethylbenz(a)Anthracene-Induced Breast Cancer Rats.Caffeic acid phenethyl ester upregulates N-myc downstream regulated gene 1 via ERK pathway to inhibit human oral cancer cell growth in vitro and in vivo.Caffeic acid production enhancement by engineering a phenylalanine over-producing Escherichia coli strain.Caffeic Acid Phenethyl Ester Induces N-myc Downstream Regulated Gene 1 to Inhibit Cell Proliferation and Invasion of Human Nasopharyngeal Cancer Cells.

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Caffeic acid phenethyl ester suppresses the proliferation of human prostate cancer cells through inhibition of p70S6K and Akt signaling networks.

http://www.wikidata.org/entity/Q37130094

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on May 2012

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Caffeic acid phenethyl ester s ...... 6K and Akt signaling networks.

@en

Caffeic acid phenethyl ester s ...... 6K and Akt signaling networks.

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type

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Caffeic acid phenethyl ester s ...... 6K and Akt signaling networks.

@en

Caffeic acid phenethyl ester s ...... 6K and Akt signaling networks.

@nl

prefLabel

Caffeic acid phenethyl ester s ...... 6K and Akt signaling networks.

@en

Caffeic acid phenethyl ester s ...... 6K and Akt signaling networks.

@nl

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1940-6207.CAPR-12-0004-T

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Cancer Prevention Research

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Caffeic acid phenethyl ester s ...... 6K and Akt signaling networks.

@en

P2093

Chih-Pin Chuu

http://www.w3.org/2001/XMLSchema#string

Hui-Ping Lin

http://www.w3.org/2001/XMLSchema#string

John M Kokontis

http://www.w3.org/2001/XMLSchema#string

Mark F Ciaccio

http://www.w3.org/2001/XMLSchema#string

Richard A Hiipakka

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Richard Baker Jones

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Ronald J Hause

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Shutsung Liao

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P2860

Protein kinase SGK mediates survival signals by phosphorylating the forkhead transcription factor FKHRL1 (FOXO3a)

TFM-Explorer: mining cis-regulatory regions in genomes

Caffeic acid phenethyl ester is a potent and specific inhibitor of activation of nuclear transcription factor NF-kappa B

TRED: a Transcriptional Regulatory Element Database and a platform for in silico gene regulation studies

Twist, a master regulator of morphogenesis, plays an essential role in tumor metastasis

The phosphatidylinositol 3'-kinase pathway is a dominant growth factor-activated cell survival pathway in LNCaP human prostate carcinoma cells

Multisite phosphorylation by Cdk2 and GSK3 controls cyclin E degradation

Inhibition of tumor growth and progression of LNCaP prostate cancer cells in athymic mice by androgen and liver X receptor agonist

Glycogen synthase kinase-3beta regulates cyclin D1 proteolysis and subcellular localization

Antiproliferative effect of liver X receptor agonists on LNCaP human prostate cancer cells

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788-797

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scholarly article

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10.1158/1940-6207.CAPR-12-0004-T

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5

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5

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2012-05-01T00:00:00Z

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22562408

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4962698

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