Advances in the genetics of rheumatoid arthritis point to subclassification into distinct disease subsets.
about
Effects of GSTM1 in rheumatoid arthritis; results from the Swedish EIRA studyCausal graph-based analysis of genome-wide association data in rheumatoid arthritisIdentification of BACH2 and RAD51B as rheumatoid arthritis susceptibility loci in a meta-analysis of genome-wide dataAnalysis of neuropeptide S receptor gene (NPSR1) polymorphism in rheumatoid arthritisTRAF1/C5 polymorphism is not associated with increased mortality in rheumatoid arthritis: two large longitudinal studies.Evaluation of the rheumatoid arthritis susceptibility loci HLA-DRB1, PTPN22, OLIG3/TNFAIP3, STAT4 and TRAF1/C5 in an inception cohort.Influence of HLA DRB1 alleles in the susceptibility of rheumatoid arthritis and the regulation of antibodies against citrullinated proteins and rheumatoid factorA systems genetics approach provides a bridge from discovered genetic variants to biological pathways in rheumatoid arthritis.Autophagy in antigen-presenting cells results in presentation of citrullinated peptides to CD4 T cells.Can anti-cyclic citrullinated peptide antibody-negative RA be subdivided into clinical subphenotypes?Genetics of serum concentration of IL-6 and TNFα in systemic lupus erythematosus and rheumatoid arthritis: a candidate gene analysis.Antibodies against cyclic citrullinated peptides of IgG, IgA and IgM isotype and rheumatoid factor of IgM and IgA isotype are increased in unaffected members of multicase rheumatoid arthritis families from northern Sweden.PAD4 is not essential for disease in the K/BxN murine autoantibody-mediated model of arthritis.ATP induces protein arginine deiminase 2-dependent citrullination in mast cells through the P2X7 purinergic receptorPharmacogenetics of rheumatoid arthritis: Potential targets from susceptibility genes and present therapies.The genetics of rheumatoid arthritis: risk and protection in different stages of the evolution of RA.Non-MHC risk alleles in rheumatoid arthritis and in the syntenic chromosome regions of corresponding animal models.Genetic markers of rheumatoid arthritis susceptibility in anti-citrullinated peptide antibody negative patients.Higher levels of autoantibodies targeting mutated citrullinated vimentin in patients with psoriatic arthritis than in patients with psoriasis vulgarisEffectiveness of four dynamic treatment strategies in patients with anticitrullinated protein antibody-negative rheumatoid arthritis: a randomised trial.Replication of Associations of Genetic Loci Outside the HLA Region With Susceptibility to Anti-Cyclic Citrullinated Peptide-Negative Rheumatoid Arthritis.Epidemiology of rheumatoid arthritis in tirana, Albania.Rheumatoid arthritis: Disease or syndrome?Fine mapping seronegative and seropositive rheumatoid arthritis to shared and distinct HLA alleles by adjusting for the effects of heterogeneity.Laboratory evaluation in rheumatic diseases.Polymorphisms of RAD51B are associated with rheumatoid arthritis and erosion in rheumatoid arthritis patients.Genetics of rheumatoid arthritis: what have we learned?New drug discovery strategies for rheumatoid arthritis: a niche for nonhuman primate models to address systemic complications in inflammatory arthritis.HLA and rheumatoid arthritis: how do they connect?Association of susceptible genetic markers and autoantibodies in rheumatoid arthritis.Personalized medicine in rheumatoid arthritis: is the glass half full or half empty?Cyclocurcumin, a curcumin derivative, exhibits immune-modulating ability and is a potential compound for the treatment of rheumatoid arthritis as predicted by the MM-PBSA method.Dipeptidyl Peptidase (DPP)-4 Inhibitor-Induced Arthritis/Arthralgia: A Review of Clinical Cases.Genetic susceptibility to rheumatoid arthritis and its implications for novel drug discovery.Fine-mapping the MHC locus in juvenile idiopathic arthritis (JIA) reveals genetic heterogeneity corresponding to distinct adult inflammatory arthritic diseasesEffect of ethanol extract of an ayurvedic preparation (Pathyadya Churna) on arthritis in rats.The increased ability to present citrullinated peptides is not unique to HLA-SE molecules: arginine-to-citrulline conversion also enhances peptide affinity for HLA-DQ molecules.The interaction between HLA shared epitope alleles and smoking and its contribution to autoimmunity against several citrullinated antigens.A spectrum of susceptibility to rheumatoid arthritis within HLA-DRB1: stratification by autoantibody status in a large UK population.Common and different genetic background for rheumatoid arthritis and coeliac disease.
P2860
Q28741719-FC1D3C85-8D05-4EC8-947E-8CE42A2AD6C6Q30501358-262E5338-D63F-40C9-B48E-FAF34E5D4BA9Q30666089-F19386A2-6FB2-4CC8-A92C-7723691671F4Q33533768-10DF696D-D904-4F47-AB92-DF5495B7FFFAQ33923265-C8FE5DE3-4086-4B1E-B244-4254B78EF7D3Q33923376-3206FB5A-163D-4458-8D36-5A0797AF2720Q33923415-9BED5340-A01C-4AE2-A352-2192A7A1C8EEQ34043155-2BBB2A8A-9087-457C-B891-B3C2DA52867EQ35627649-A26489A6-024F-4882-913D-48025F5FCD9FQ35840850-C6F2B309-9D79-465D-BC90-3B058FABB9EAQ35921093-3BEFEF57-3464-44E0-885A-C1D8D45B54BBQ36020173-923821CC-E6C8-4E11-A34B-1E5DBC2BEC70Q36245934-313654FA-B5FE-43D6-8A74-47B434E6D53BQ36303652-9F59D47D-5A7D-470C-BCE8-0D6C023F0036Q36441438-29397706-8E91-4AFE-8120-35B0B49339A5Q36456814-22A62CAE-C865-4518-AFBE-318F74B06895Q36464468-88A67CF9-E01F-4D8A-85CB-EBFCFDB86D4EQ36679775-8757ED41-034C-4E58-8A38-0D66FBBFE1A2Q36733774-C7975857-78DA-4967-81A9-50454D63C28FQ36773674-147198A9-F15A-4760-8926-790459E85C0CQ37046219-F6735162-64B8-401B-B832-4A0A6BC9AB43Q37160579-5D146F46-C12B-49C6-AA06-DD4CCB7E8BFBQ37358114-AA8F5195-85E6-45AF-9FAD-95460FC4D437Q37693227-71E8FFA7-ABEA-4596-A4FE-3159501FCF64Q37721695-FFE06016-A33C-46B0-AD99-7A8913DC7EAEQ37731382-B73C69A7-EC2B-4CD0-84E2-464312C960BEQ37873802-B0337C09-63DD-405A-B1F7-FDE80F4EB984Q37998089-0989D393-B4FD-47A7-87FB-0335B5705EC1Q38210610-728E9FEC-B560-4B7E-8A1B-AF22ADEDD258Q38246108-9D4ABBAD-293D-4C7F-99DF-1BB2FDBE040BQ38259809-59CA6304-38D6-40BB-BBA2-E8AA2921D074Q38710775-B80D6716-BB07-426D-AC8E-630129792F93Q38731788-3E93D2F2-948D-4590-B50D-C3B1C056CF89Q38856541-8E2EA45E-17B3-4019-81A0-1F7A6C3C1436Q39080731-B09B0D07-DB91-4A4D-A959-290B1D00858FQ39980396-E46EC653-A71A-4B92-B6D2-BA720B3206DCQ41661241-CB57BE1B-651D-4328-A779-DE3A1C54D550Q42598147-29DA51E5-C64E-40A2-BA13-8F5A0FAFE042Q42614760-3FE06E49-546B-48CE-A043-0CE16A0F7FA0Q42629408-3CF134C6-6787-4271-94EF-B91E285EEF7F
P2860
Advances in the genetics of rheumatoid arthritis point to subclassification into distinct disease subsets.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 31 March 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Advances in the genetics of rh ...... into distinct disease subsets.
@en
Advances in the genetics of rh ...... into distinct disease subsets.
@nl
type
label
Advances in the genetics of rh ...... into distinct disease subsets.
@en
Advances in the genetics of rh ...... into distinct disease subsets.
@nl
prefLabel
Advances in the genetics of rh ...... into distinct disease subsets.
@en
Advances in the genetics of rh ...... into distinct disease subsets.
@nl
P2860
P356
P1476
Advances in the genetics of rh ...... into distinct disease subsets.
@en
P2093
Tom W J Huizinga
P2860
P2888
P356
10.1186/AR2384
P577
2008-03-31T00:00:00Z