NKCC1 and AE3 appear to accumulate chloride in embryonic motoneurons.
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Mechanisms of GABAergic homeostatic plasticityLoss of Slc4a1b chloride/bicarbonate exchanger function protects mechanosensory hair cells from aminoglycoside damage in the zebrafish mutant persephoneDifferences in cortical versus subcortical GABAergic signaling: a candidate mechanism of electroclinical uncoupling of neonatal seizures.Circadian modulation of the Cl(-) equilibrium potential in the rat suprachiasmatic nuclei.Pharmacological manipulation of GABA-driven activity in ovo disrupts the development of dendritic morphology but not the maturation of spinal cord network activity.Contribution of the potassium-chloride co-transporter KCC2 to the modulation of lumbar spinal networks in mice.GABAergic synaptic scaling in embryonic motoneurons is mediated by a shift in the chloride reversal potential.Reversal of behavioral deficits and synaptic dysfunction in mice overexpressing neuregulin 1Non-cell-autonomous factor induces the transition from excitatory to inhibitory GABA signaling in retina independent of activityActivity blockade and GABAA receptor blockade produce synaptic scaling through chloride accumulation in embryonic spinal motoneurons and interneuronsChloride Accumulators NKCC1 and AE2 in Mouse GnRH Neurons: Implications for GABAA Mediated Excitation.Refuting the challenges of the developmental shift of polarity of GABA actions: GABA more exciting than ever!In vivo synaptic scaling is mediated by GluA2-lacking AMPA receptors in the embryonic spinal cord.Calcium dependence of damage to mouse motor nerve terminals following oxygen/glucose deprivation.Postnatal maturation of GABAergic transmission in the rat basolateral amygdalaCarbonic anhydrases CA4 and CA14 both enhance AE3-mediated Cl--HCO3- exchange in hippocampal neurons.GABAergic depolarization during early cortical development and implications for anticonvulsive therapy in neonates.Maturation of the GABAergic transmission in normal and pathologic motoneurons.The effects of endocannabinoid signaling on network activity in developing and motor circuits.GABAA receptor-mediated tonic depolarization in developing neural circuits.Anion transport and GABA signaling.Depolarizing GABA and developmental epilepsies.SLC26A11 (KBAT) in Purkinje Cells Is Critical for Inhibitory Transmission and Contributes to Locomotor CoordinationGlobal gene expression analysis of rodent motor neurons following spinal cord injury associates molecular mechanisms with development of postinjury spasticity.Elevated intracellular Na+ concentrations in developing spinal neurons.Regulation of synaptic scaling by action potential-independent miniature neurotransmission.Both NKCC1 and anion exchangers contribute to Cl⁻ accumulation in postnatal forebrain neuronal progenitors.Activity-dependent plasticity in the isolated embryonic avian brainstem following manipulations of rhythmic spontaneous neural activity.Cystic fibrosis transmembrane conductance regulator modulates synaptic chloride homeostasis in motoneurons of the rat spinal cord during neonatal development.
P2860
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P2860
NKCC1 and AE3 appear to accumulate chloride in embryonic motoneurons.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
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2008年學術文章
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name
NKCC1 and AE3 appear to accumulate chloride in embryonic motoneurons.
@en
NKCC1 and AE3 appear to accumulate chloride in embryonic motoneurons.
@nl
type
label
NKCC1 and AE3 appear to accumulate chloride in embryonic motoneurons.
@en
NKCC1 and AE3 appear to accumulate chloride in embryonic motoneurons.
@nl
prefLabel
NKCC1 and AE3 appear to accumulate chloride in embryonic motoneurons.
@en
NKCC1 and AE3 appear to accumulate chloride in embryonic motoneurons.
@nl
P2860
P356
P1476
NKCC1 and AE3 appear to accumulate chloride in embryonic motoneurons
@en
P2093
Carlos Gonzalez-Islas
Nikolai Chub
P2860
P304
P356
10.1152/JN.90986.2008
P407
P50
P577
2008-11-26T00:00:00Z