Suppressed hindlimb perfusion in Rac2-/- and Nox2-/- mice does not result from impaired collateral growth. - Wikidata - awgldk - TriplyDB

Suppressed hindlimb perfusion in Rac2-/- and Nox2-/- mice does not result from impaired collateral growth.

Suppressed hindlimb perfusion in Rac2-/- and Nox2-/- mice does not result from impaired collateral growth.

http://www.wikidata.org/entity/Q37138949

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Nox2 and p47(phox) modulate compensatory growth of primary collateral arteries.Vascular growth responses to chronic arterial occlusion are unaffected by myeloid specific focal adhesion kinase (FAK) deletion.Mechanisms of Amplified Arteriogenesis in Collateral Artery Segments Exposed to Reversed Flow Direction.Despite normal arteriogenic and angiogenic responses, hind limb perfusion recovery and necrotic and fibroadipose tissue clearance are impaired in matrix metalloproteinase 9-deficient mice Trans-illuminated laser speckle imaging of collateral artery blood flow in ischemic mouse hindlimb.Laser speckle flowmetry method for measuring spatial and temporal hemodynamic alterations throughout large microvascular networks.Myoglobin overexpression inhibits reperfusion in the ischemic mouse hindlimb through impaired angiogenesis but not arteriogenesis.Marvels, mysteries, and misconceptions of vascular compensation to peripheral artery occlusion.Polymerase δ-interacting protein 2 promotes postischemic neovascularization of the mouse hindlimb Temporal patterns of blood flow and nitric oxide synthase expression affect macrophage accumulation and proliferation during collateral growth Chemokine-coupled β2 integrin-induced macrophage Rac2-Myosin IIA interaction regulates VEGF-A mRNA stability and arteriogenesis.Chronic hindlimb ischemia impairs functional vasodilation and vascular reactivity in mouse feed arteries.Impaired compensation to femoral artery ligation in diet-induced obese mice is primarily mediated via suppression of collateral growth by Nox2 and p47phox.Novel method to assess arterial insufficiency in rodent hind limb.Molecular basis for impaired collateral artery growth in the spontaneously hypertensive rat: insight from microarray analysis NADPH oxidases as a source of oxidative stress and molecular target in ischemia/reperfusion injury.Optimization of Vascular Casting for Three-Dimensional Fluorescence Cryo-Imaging of Collateral Vessels in the Ischemic Rat Hindlimb.DNA Methyltransferase 1-Dependent DNA Hypermethylation Constrains Arteriogenesis by Augmenting Shear Stress Set Point.MicroRNA-146a Regulates Perfusion Recovery in Response to Arterial Occlusion via Arteriogenesis.

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Suppressed hindlimb perfusion in Rac2-/- and Nox2-/- mice does not result from impaired collateral growth.

http://www.wikidata.org/entity/Q37138949

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on 16 January 2009

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Suppressed hindlimb perfusion ...... om impaired collateral growth.

@en

Suppressed hindlimb perfusion ...... om impaired collateral growth.

@nl

type

label

Suppressed hindlimb perfusion ...... om impaired collateral growth.

@en

Suppressed hindlimb perfusion ...... om impaired collateral growth.

@nl

prefLabel

Suppressed hindlimb perfusion ...... om impaired collateral growth.

@en

Suppressed hindlimb perfusion ...... om impaired collateral growth.

@nl

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AJPHEART.00772.2008

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American Journal of Physiology - Heart and Circulatory Physiology

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Suppressed hindlimb perfusion ...... om impaired collateral growth.

@en

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Carlos A Labarrere

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David A Ingram

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Joseph L Unthank

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Laura S Haneline

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Mary C Dinauer

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Matthew A Ziegler

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Matthew R Distasi

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Mervin C Yoder

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Michael C Dalsing

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Michael P Murphy

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P2860

Identification of a conserved Rac-binding site on NADPH oxidases supports a direct GTPase regulatory mechanism

Delayed arteriogenesis in hypercholesterolemic mice

Deficiency of the hematopoietic cell-specific Rho family GTPase Rac2 is characterized by abnormalities in neutrophil function and host defense

Novel role of gp91(phox)-containing NAD(P)H oxidase in vascular endothelial growth factor-induced signaling and angiogenesis

Mechanisms of angiogenesis and arteriogenesis

Ultrasound-microbubble-induced neovascularization in mouse skeletal muscle.

Contribution of arteriogenesis and angiogenesis to postocclusive hindlimb perfusion in mice.

Blood monocyte concentration is critical for enhancement of collateral artery growth.

Bone marrow transplantation abolishes inhibition of arteriogenesis in placenta growth factor (PlGF) -/- mice.

Role of gp91phox (Nox2)-containing NAD(P)H oxidase in angiogenesis in response to hindlimb ischemia.

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H877-86

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scholarly article

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10.1152/AJPHEART.00772.2008

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296

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2009-01-16T00:00:00Z

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23798352

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19151256

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2660231

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