ADAM30 Downregulates APP-Linked Defects Through Cathepsin D Activation in Alzheimer's Disease.
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Mice are not Men: ADAM30 Findings Emphasize a Broader Look Towards Murine Alzheimer's Disease ModelsGenome-wide, high-content siRNA screening identifies the Alzheimer's genetic risk factor FERMT2 as a major modulator of APP metabolism.MT5-MMP Promotes Alzheimer's Pathogenesis in the Frontal Cortex of 5xFAD Mice and APP Trafficking in vitroThe Metalloprotease Meprin β Is an Alternative β-Secretase of APP.Autophagy and Alzheimer's Disease: From Molecular Mechanisms to Therapeutic Implications.Autophagy mediates glucose starvation-induced glioblastoma cell quiescence and chemoresistance through coordinating cell metabolism, cell cycle, and survival.
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ADAM30 Downregulates APP-Linked Defects Through Cathepsin D Activation in Alzheimer's Disease.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 02 June 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
ADAM30 Downregulates APP-Linke ...... vation in Alzheimer's Disease.
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ADAM30 Downregulates APP-Linke ...... vation in Alzheimer's Disease.
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type
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ADAM30 Downregulates APP-Linke ...... vation in Alzheimer's Disease.
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ADAM30 Downregulates APP-Linke ...... vation in Alzheimer's Disease.
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ADAM30 Downregulates APP-Linke ...... vation in Alzheimer's Disease.
@en
ADAM30 Downregulates APP-Linke ...... vation in Alzheimer's Disease.
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ADAM30 Downregulates APP-Linke ...... vation in Alzheimer's Disease.
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Amandine Flaig
Anne-Marie Ayral
Bart Dermaut
Charlotte Bauer
Claudine Berr
David Mann
Elisabeth Werkmeister
Fanny Eysert
Florence Leroux
Florie Demiautte
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P304
P356
10.1016/J.EBIOM.2016.06.002
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2016-06-02T00:00:00Z
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P818
1906.07511