A large number of protein expression changes occur early in life and precede phenotype onset in a mouse model for huntington disease.
about
EFhd2, a Protein Linked to Alzheimer's Disease and Other Neurological DisordersMouse models of polyglutamine diseases: review and data table. Part IRecent advances in quantitative neuroproteomicsPossible involvement of self-defense mechanisms in the preferential vulnerability of the striatum in Huntington's diseaseEstablishment of a mouse model with misregulated chromosome condensation due to defective Mcph1 functionEnhanced neuronal glucose transporter expression reveals metabolic choice in a HD Drosophila modelOxygen consumption deficit in Huntington disease mouse brain under metabolic stressA role for huntington disease protein in dendritic RNA granulesProteomic Analysis of Dynein-Interacting Proteins in Amyotrophic Lateral Sclerosis Synaptosomes Reveals Alterations in the RNA-Binding Protein Staufen1.A mouse embryonic stem cell bank for inducible overexpression of human chromosome 21 genes.Mutant SOD1 knockdown in all cell types ameliorates disease in G85R SOD1 mice with a limited additional effect over knockdown restricted to motor neuronsA common gene expression signature in Huntington's disease patient brain regions.Delayed Onset and Reduced Cognitive Deficits through Pre-Conditioning with 3-Nitropropionic Acid is Dependent on Sex and CAG Repeat Length in the R6/2 Mouse Model of Huntington's Disease.SORLA-mediated trafficking of TrkB enhances the response of neurons to BDNF.Double NF1 inactivation affects adrenocortical function in NF1Prx1 mice and a human patientPoly-glutamine expanded huntingtin dramatically alters the genome wide binding of HSF1.A role of mitochondrial complex II defects in genetic models of Huntington's disease expressing N-terminal fragments of mutant huntingtinThe Role of Epigenetic Mechanisms in the Regulation of Gene Expression in the Nervous System.Proteomic analysis of mitochondrial dysfunction in neurodegenerative diseases.Energy deficit in Huntington disease: why it matters.Mitochondrial and metabolic-based protective strategies in Huntington's disease: the case of creatine and coenzyme Q.New insight into neurodegeneration: the role of proteomics.Challenges of Huntington's disease and quest for therapeutic biomarkers.Quantitative Proteomic Analysis Reveals Similarities between Huntington's Disease (HD) and Huntington's Disease-Like 2 (HDL2) Human Brains.Dietary anaplerotic therapy improves peripheral tissue energy metabolism in patients with Huntington's disease.Changes in the striatal proteome of YAC128Q mice exhibit gene-environment interactions between mutant huntingtin and manganese.PROTEOMER: A workflow-optimized laboratory information management system for 2-D electrophoresis-centered proteomics.Altered Aconitase 2 Activity in Huntington's Disease Peripheral Blood Cells and Mouse Model Striatum.Towards an Understanding of Energy Impairment in Huntington's Disease Brain.Proteomic analysis reveals distinctive protein profiles involved in CD8+ T cell-mediated murine autoimmune cholangitis.Early postnatal behavioral, cellular, and molecular changes in models of Huntington disease are reversible by HDAC inhibition
P2860
Q26750390-1D332859-D182-4DAC-8F66-E12F39DA2BB8Q26853025-A2FF2C73-700E-42E1-A9D6-EA5FFEE2404BQ27000073-193AEEF1-D562-4FEA-9960-02EE66173A77Q27011417-AAD9402B-7FD1-4043-8F3B-B6AA59DEBBFEQ27334403-D557B2DA-5AF0-4578-8192-907295B5352AQ28389710-FC1B627B-D0EA-4CEE-B7E9-7AB8789DD7B4Q28397230-E3D6588A-F7DC-4657-8AE7-C44141839A50Q28574690-0229D692-722B-44DF-91F9-4EC5D0777BA5Q30354368-F7520E13-02F9-457C-9A51-AC35E0425032Q33452667-5DC499E6-C092-4171-8454-09B9D2FE003AQ34036108-D6164C3C-0CFD-4C88-9EC7-DC372007C8B6Q34446889-B932FA71-7DF5-400F-94AD-0860773F2C88Q34520437-48215EFD-05B0-4A82-B0F0-CDBE260076CCQ34973554-EB9B8CFF-B222-4AF9-90F9-841FEFD5D842Q35182111-2F0DF426-0EBE-453C-A7D5-B65E7BB7854AQ36509945-76BACF39-E918-481E-81DA-63AD899D348CQ37153804-3A8833F6-76AE-42B3-84B2-1B35DC6125B4Q37444945-07DA2B11-6BB8-4C09-ADEA-AAC4E2D3628EQ37775036-8A4C93EE-EEFA-435B-801C-892D2DD023F6Q37834195-8B637FE4-6317-4988-A4F7-0A8FA3DBF1A5Q37965763-8E6B9FA3-A261-4832-8D0B-14E4D1E8F010Q38170010-CDA38FAB-9B16-4B10-823C-BA2F80BFDC68Q38257697-02F97861-9F45-4799-9B3E-C347BAEE624BQ41377417-0AB20D3C-8A24-4E1F-AF39-D1DF1ED755EDQ41734464-5D01BEC0-7A41-4823-8F7D-58A55219A76AQ42070989-F9B27C81-80E3-422C-B066-0705EB3E7E77Q45382929-5E0FCAD4-19A2-48FB-B3B1-DB6907E98B42Q47138180-9AE4D73F-F723-42F6-A30E-01CD709BE172Q47442924-C3EE7614-D8AB-46A8-86A4-13332BF3C859Q48156075-F80ECA92-5C70-401B-B0CB-7760AF5BCB5AQ58700269-5504A805-C6B0-4028-83BD-3D2F889D402D
P2860
A large number of protein expression changes occur early in life and precede phenotype onset in a mouse model for huntington disease.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
A large number of protein expr ...... model for huntington disease.
@en
A large number of protein expr ...... model for huntington disease.
@nl
type
label
A large number of protein expr ...... model for huntington disease.
@en
A large number of protein expr ...... model for huntington disease.
@nl
prefLabel
A large number of protein expr ...... model for huntington disease.
@en
A large number of protein expr ...... model for huntington disease.
@nl
P2093
P2860
P1476
A large number of protein expr ...... model for huntington disease.
@en
P2093
Andrea Koppelstätter
Andrew Strand
Ben Woodman
Claus Zabel
Daniela Hartl
Grit Nebrich
Joachim Klose
Maik A Wacker
Michael Rohe
P2860
P304
P356
10.1074/MCP.M800277-MCP200
P577
2008-11-30T00:00:00Z