Loss of SEC-23 in Caenorhabditis elegans causes defects in oogenesis, morphogenesis, and extracellular matrix secretion.
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The function and expansion of the Patched- and Hedgehog-related homologs in C. elegansFunctional genomic analysis of C. elegans moltingThe Caenorhabditis elegans epidermis as a model skin. II: differentiation and physiological rolesWorming our way in and out of the Caenorhabditis elegans germline and developing embryoThe ArfGEF GBF-1 Is Required for ER Structure, Secretion and Endocytic Transport in C. elegansCUTI-1: A novel tetraspan protein involved in C. elegans CUTicle formation and epithelial integrityFunctional genomics of hsp-90 in parasitic and free-living nematodes.Genetics coupled to quantitative intact proteomics links heritable aphid and endosymbiont protein expression to circulative polerovirus transmission.MLT-10 defines a family of DUF644 and proline-rich repeat proteins involved in the molting cycle of Caenorhabditis elegans.Cranio-lenticulo-sutural dysplasia associated with defects in collagen secretion.The C terminus of collagen SQT-3 has complex and essential functions in nematode collagen assembly.C. elegans NIMA-related kinases NEKL-2 and NEKL-3 are required for the completion of molting.Extracellular leucine-rich repeat proteins are required to organize the apical extracellular matrix and maintain epithelial junction integrity in C. elegansMechanistic insights and identification of two novel factors in the C. elegans NMD pathway.The [corrected] SEC23-SEC31 [corrected] interface plays critical role for export of procollagen from the endoplasmic reticulumThe V0-ATPase mediates apical secretion of exosomes containing Hedgehog-related proteins in Caenorhabditis elegansSEC23B is required for the maintenance of murine professional secretory tissues.The genetic basis of a craniofacial disease provides insight into COPII coat assembly.Negative regulation of Caenorhabditis elegans epidermal damage responses by death-associated protein kinase.An ER-resident membrane protein complex regulates nicotinic acetylcholine receptor subunit composition at the synapse.Selective targeting of ER exit sites supports axon development.Inherited hematological disorders due to defects in coat protein (COP)II complex.Molting in C. elegans.Requirement of the Caenorhabditis elegans RapGEF pxf-1 and rap-1 for epithelial integrityCOPII-coated membranes function as transport carriers of intracellular procollagen I.Conserved Ankyrin Repeat Proteins and Their NIMA Kinase Partners Regulate Extracellular Matrix Remodeling and Intracellular Trafficking in Caenorhabditis elegans.Heritable Skeletal Disorders Arising from Defects in Processing and Transport of Type I Procollagen from the ER: Perspectives on Possible Therapeutic Approaches.Actin organization and endocytic trafficking are controlled by a network linking NIMA-related kinases to the CDC-42-SID-3/ACK1 pathway.
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Loss of SEC-23 in Caenorhabditis elegans causes defects in oogenesis, morphogenesis, and extracellular matrix secretion.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on 07 August 2003
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Loss of SEC-23 in Caenorhabdit ...... xtracellular matrix secretion.
@en
Loss of SEC-23 in Caenorhabdit ...... xtracellular matrix secretion.
@nl
type
label
Loss of SEC-23 in Caenorhabdit ...... xtracellular matrix secretion.
@en
Loss of SEC-23 in Caenorhabdit ...... xtracellular matrix secretion.
@nl
prefLabel
Loss of SEC-23 in Caenorhabdit ...... xtracellular matrix secretion.
@en
Loss of SEC-23 in Caenorhabdit ...... xtracellular matrix secretion.
@nl
P2093
P2860
P921
P356
P1476
Loss of SEC-23 in Caenorhabdit ...... xtracellular matrix secretion.
@en
P2093
Brett Roberts
Caroline Clucas
Iain L Johnstone
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P304
P356
10.1091/MBC.E03-03-0162
P577
2003-08-07T00:00:00Z