HDL, ABC transporters, and cholesterol efflux: implications for the treatment of atherosclerosis.
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Diverse roles of macrophages in atherosclerosis: from inflammatory biology to biomarker discoveryAntagonism of miR-33 in mice promotes reverse cholesterol transport and regression of atherosclerosismicroRNAs and cholesterol metabolismThe role of ANGPTL3 in controlling lipoprotein metabolismMIF and CXCL12 in Cardiovascular Diseases: Functional Differences and SimilaritiesRole of gut microbiota in the modulation of atherosclerosis-associated immune responseEmerging role of mast cells and macrophages in cardiovascular and metabolic diseasesAnti-atherogenic mechanisms of high density lipoprotein: effects on myeloid cellsMicroRNA modulation of cholesterol homeostasisPost-transcriptional regulation in metabolic diseasesDouble Superhelix Model of High Density LipoproteinHigh-Density Lipoproteins: Nature's Multifunctional NanoparticlesWill Lipidation of ApoA1 through Interaction with ABCA1 at the Intestinal Level Affect the Protective Functions of HDL?Niacin Therapy, HDL Cholesterol, and Cardiovascular Disease: Is the HDL Hypothesis Defunct?Cholesterol, inflammation and innate immunityImpaired cholesterol efflux in senescent macrophages promotes age-related macular degenerationImmune cell screening of a nanoparticle library improves atherosclerosis therapyThe dynamin chemical inhibitor dynasore impairs cholesterol trafficking and sterol-sensitive genes transcription in human HeLa cells and macrophagesMicroRNA-33 and the SREBP host genes cooperate to control cholesterol homeostasisAtherosclerosis in LDLR-knockout mice is inhibited, but not reversed, by the PPARgamma ligand pioglitazoneComparative transcriptional network modeling of three PPAR-α/γ co-agonists reveals distinct metabolic gene signatures in primary human hepatocytesMacrophages in the pathogenesis of atherosclerosisInteraction of methylation-related genetic variants with circulating fatty acids on plasma lipids: a meta-analysis of 7 studies and methylation analysis of 3 studies in the Cohorts for Heart and Aging Research in Genomic Epidemiology consortium.Loss of ABCG1 influences regulatory T cell differentiation and atherosclerosis.Macrophage-independent regulation of reverse cholesterol transport by liver X receptors.Phenotypic polarization of macrophages in atherosclerosisLiver LXRα expression is crucial for whole body cholesterol homeostasis and reverse cholesterol transport in miceNuclear receptors as drug targets for metabolic disease.Evaluating the relationship between biomarkers of potential harm and biomarkers of tobacco exposure among current, past, and nonsmokers: data from the National Health and Nutrition Examination Survey 2007-2012.Enhanced hepatic apoA-I secretion and peripheral efflux of cholesterol and phospholipid in CD36 null miceCongruency between biophysical data from multiple platforms and molecular dynamics simulation of the double-super helix model of nascent high-density lipoprotein.A novel role for c-Jun N-terminal kinase and phosphoinositide 3-kinase in the liver X receptor-mediated induction of macrophage gene expressionAbsence of myeloid COX-2 attenuates acute inflammation but does not influence development of atherosclerosis in apolipoprotein E null miceHumans with atherosclerosis have impaired ABCA1 cholesterol efflux and enhanced high-density lipoprotein oxidation by myeloperoxidase.The ability to promote efflux via ABCA1 determines the capacity of serum specimens with similar high-density lipoprotein cholesterol to remove cholesterol from macrophages.The TNF-like protein 1A-death receptor 3 pathway promotes macrophage foam cell formation in vitro.Modifying apolipoprotein A-I by malondialdehyde, but not by an array of other reactive carbonyls, blocks cholesterol efflux by the ABCA1 pathwayModest diet-induced weight loss reduces macrophage cholesterol efflux to plasma of patients with metabolic syndrome.miR-33 links SREBP-2 induction to repression of sterol transporters.Extracellular cholesterol-rich microdomains generated by human macrophages and their potential function in reverse cholesterol transport.
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HDL, ABC transporters, and cholesterol efflux: implications for the treatment of atherosclerosis.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on May 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
HDL, ABC transporters, and cho ...... treatment of atherosclerosis.
@en
HDL, ABC transporters, and cho ...... treatment of atherosclerosis.
@nl
type
label
HDL, ABC transporters, and cho ...... treatment of atherosclerosis.
@en
HDL, ABC transporters, and cho ...... treatment of atherosclerosis.
@nl
prefLabel
HDL, ABC transporters, and cho ...... treatment of atherosclerosis.
@en
HDL, ABC transporters, and cho ...... treatment of atherosclerosis.
@nl
P2093
P1433
P1476
HDL, ABC transporters, and cho ...... treatment of atherosclerosis.
@en
P2093
Alan R Tall
Naoki Terasaka
Tamara Pagler
P304
P356
10.1016/J.CMET.2008.03.001
P577
2008-05-01T00:00:00Z