Autoantibodies in scurfy mice and IPEX patients recognize keratin 14. - Wikidata - awgldk - TriplyDB

Autoantibodies in scurfy mice and IPEX patients recognize keratin 14.

Autoantibodies in scurfy mice and IPEX patients recognize keratin 14.

http://www.wikidata.org/entity/Q37158241

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The Biology of Autoimmune Response in the Scurfy Mice that Lack the CD4+Foxp3+ Regulatory T-Cells IL-2-controlled expression of multiple T cell trafficking genes and Th2 cytokines in the regulatory T cell-deficient scurfy mice: implication to multiorgan inflammation and control of skin and lung inflammation From IPEX syndrome to FOXP3 mutation: a lesson on immune dysregulation.A self-reactive TCR drives the development of Foxp3+ regulatory T cells that prevent autoimmune disease.Regulatory T cell-deficient scurfy mice develop systemic autoimmune features resembling lupus-like disease.Fatal autoimmunity results from the conditional deletion of Snai2 and Snai3 Immune dysregulation, polyendocrinopathy, enteropathy, x-linked syndrome: a paradigm of immunodeficiency with autoimmunity γδ T Cells Protect the Liver and Lungs of Mice from Autoimmunity Induced by Scurfy Lymphocytes B cells are critical for autoimmune pathology in Scurfy mice.Severe Developmental B Lymphopoietic Defects in Foxp3-Deficient Mice are Refractory to Adoptive Regulatory T Cell Therapy.Altered expression of keratin 14 in lesional epidermis of autoimmune skin diseases.Genetic disorders with immune dysregulation.

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Autoantibodies in scurfy mice and IPEX patients recognize keratin 14.

http://www.wikidata.org/entity/Q37158241

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 11 February 2010

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Autoantibodies in scurfy mice and IPEX patients recognize keratin 14.

@en

Autoantibodies in scurfy mice and IPEX patients recognize keratin 14.

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type

label

Autoantibodies in scurfy mice and IPEX patients recognize keratin 14.

@en

Autoantibodies in scurfy mice and IPEX patients recognize keratin 14.

@nl

prefLabel

Autoantibodies in scurfy mice and IPEX patients recognize keratin 14.

@en

Autoantibodies in scurfy mice and IPEX patients recognize keratin 14.

@nl

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Journal of Investigative Dermatology

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Autoantibodies in scurfy mice and IPEX patients recognize keratin 14.

@en

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Deborah D Glass

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Ethan M Shevach

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Eva N Huter

http://www.w3.org/2001/XMLSchema#string

Kannan Natarajan

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Troy R Torgerson

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P2860

Mouse differentiation-specific keratins 1 and 10 require a preexisting keratin scaffold to form a filament network

Clinical and molecular features of the immunodysregulation, polyendocrinopathy, enteropathy, X linked (IPEX) syndrome

Foxp3 programs the development and function of CD4+CD25+ regulatory T cells

X-linked neonatal diabetes mellitus, enteropathy and endocrinopathy syndrome is the human equivalent of mouse scurfy

The immune dysregulation, polyendocrinopathy, enteropathy, X-linked syndrome (IPEX) is caused by mutations of FOXP3

Humoral autoimmune responses to the squamous cell carcinoma antigen protein family in psoriasis.

Regulatory T cells in the control of immune pathology.

Immunologic tolerance maintained by CD25+ CD4+ regulatory T cells: their common role in controlling autoimmunity, tumor immunity, and transplantation tolerance.

IPEX, FOXP3 and regulatory T-cells: a model for autoimmunity.

CD4+ CD25+ suppressor T cells: more questions than answers.

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1391-1399

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scholarly article

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10.1038/JID.2010.16

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2010-02-11T00:00:00Z

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20147963

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3767981

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