Aquaporin 4-specific T cells and NMO-IgG cause primary retinal damage in experimental NMO/SD - Wikidata - awgldk - TriplyDB

Aquaporin 4-specific T cells and NMO-IgG cause primary retinal damage in experimental NMO/SD

Aquaporin 4-specific T cells and NMO-IgG cause primary retinal damage in experimental NMO/SD

http://www.wikidata.org/entity/Q37160320

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Complement-independent retinal pathology produced by intravitreal injection of neuromyelitis optica immunoglobulin G.Marked central nervous system pathology in CD59 knockout rats following passive transfer of Neuromyelitis optica immunoglobulin G.Microstructural visual system changes in AQP4-antibody-seropositive NMOSD.Deletional tolerance prevents AQP4-directed autoimmunity in mice.Müller cells and retinal axons can be primary targets in experimental neuromyelitis optica spectrum disorder.T cells targeting neuromyelitis optica autoantigen aquaporin-4 cause paralysis and visual system injury.Foveal thinning in neuromyelitis optica: A sign of retinal astrocytopathy?Induction of Paralysis and Visual System Injury in Mice by T Cells Specific for Neuromyelitis Optica Autoantigen Aquaporin-4.Retinal glymphatic system: an explanation for transient retinal layer volume changes?Mechanisms for lesion localization in neuromyelitis optica spectrum disorders.Detection Methods for Autoantibodies in Suspected Autoimmune Encephalitis Differences in clinical features between optic neuritis in neuromyelitis optica spectrum disorders and in multiple sclerosis

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Aquaporin 4-specific T cells and NMO-IgG cause primary retinal damage in experimental NMO/SD

http://www.wikidata.org/entity/Q37160320

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article científic

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scientific article published on 08 August 2016

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Aquaporin 4-specific T cells a ...... damage in experimental NMO/SD

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Aquaporin 4-specific T cells a ...... damage in experimental NMO/SD

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Aquaporin 4-specific T cells a ...... damage in experimental NMO/SD

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Aquaporin 4-specific T cells a ...... damage in experimental NMO/SD.

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Acta neuropathologica communications

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Aquaporin 4-specific T cells a ...... damage in experimental NMO/SD

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Bleranda Zeka

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Charlotte Dahle

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Fritz Leutmezer

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Ichiro Nakashima

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Kathrin Schanda

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Kazuo Fujihara

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Maria Hastermann

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Nathalie Kaufmann

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Paulus Rommer

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Tatsuro Misu

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P2860

Blood supply of the optic nerve head and its role in optic atrophy, glaucoma, and oedema of the optic disc

IgG marker of optic-spinal multiple sclerosis binds to the aquaporin-4 water channel

A serum autoantibody marker of neuromyelitis optica: distinction from multiple sclerosis

What is the Thalamus in Zebrafish?

Neuromyelitis optica and multiple sclerosis: Seeing differences through optical coherence tomography.

A role for humoral mechanisms in the pathogenesis of Devic's neuromyelitis optica.

Novel rodent models for macular research

Aquaporin-4 water channel protein in the rat retina and optic nerve: polarized expression in Müller cells and fibrous astrocytes.

Aquaporin 4-specific T cells in neuromyelitis optica exhibit a Th17 bias and recognize Clostridium ABC transporter

Highly encephalitogenic aquaporin 4-specific T cells and NMO-IgG jointly orchestrate lesion location and tissue damage in the CNS.

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