Insulin receptor substrate-1 deficiency drives a proinflammatory phenotype in KRAS mutant lung adenocarcinoma. - Wikidata - awgldk - TriplyDB

Insulin receptor substrate-1 deficiency drives a proinflammatory phenotype in KRAS mutant lung adenocarcinoma.

Insulin receptor substrate-1 deficiency drives a proinflammatory phenotype in KRAS mutant lung adenocarcinoma.

http://www.wikidata.org/entity/Q37161785

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The MicroRNA miR-191 Supports T Cell Survival Following Common γ Chain Signaling Ablation of insulin receptor substrates 1 and 2 suppresses Kras-driven lung tumorigenesis.

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Insulin receptor substrate-1 deficiency drives a proinflammatory phenotype in KRAS mutant lung adenocarcinoma.

http://www.wikidata.org/entity/Q37161785

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 20 July 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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Insulin receptor substrate-1 d ...... AS mutant lung adenocarcinoma.

@en

Insulin receptor substrate-1 d ...... AS mutant lung adenocarcinoma.

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type

label

Insulin receptor substrate-1 d ...... AS mutant lung adenocarcinoma.

@en

Insulin receptor substrate-1 d ...... AS mutant lung adenocarcinoma.

@nl

prefLabel

Insulin receptor substrate-1 d ...... AS mutant lung adenocarcinoma.

@en

Insulin receptor substrate-1 d ...... AS mutant lung adenocarcinoma.

@nl

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PNAS.1601989113

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Proceedings of the National Academy of Sciences of the United States of America

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Insulin receptor substrate-1 d ...... AS mutant lung adenocarcinoma.

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A McGarry Houghton

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Heather E Metz

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Jay K Kolls

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Julia Kargl

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Julie Randolph-Habecker

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Kyoung-Hee Kim

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Morris F White

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Shira R Abberbock

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Stephanie E Busch

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Sue E Knoblaugh

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Hallmarks of Cancer: The Next Generation

Phosphatidylinositol 3'-kinase is activated by association with IRS-1 during insulin stimulation

Neutralizing tumor-promoting chronic inflammation: a magic bullet?

Co-occurring genomic alterations define major subsets of KRAS-mutant lung adenocarcinoma with distinct biology, immune profiles, and therapeutic vulnerabilities.

Comprehensive molecular profiling of lung adenocarcinoma.

Neutrophil elastase-mediated degradation of IRS-1 accelerates lung tumor growth

IL-22BP is regulated by the inflammasome and modulates tumorigenesis in the intestine

Subcellular localization of insulin receptor substrate family proteins associated with phosphatidylinositol 3-kinase activity and alterations in lipolysis in primary mouse adipocytes from IRS-1 null mice

STATs in cancer inflammation and immunity: a leading role for STAT3

Cancer statistics, 2009

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10.1073/PNAS.1601989113

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2016-07-20T00:00:00Z

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27439864

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