The aging brain and cognition: contribution of vascular injury and aβ to mild cognitive dysfunction. - Wikidata - awgldk - TriplyDB

The aging brain and cognition: contribution of vascular injury and aβ to mild cognitive dysfunction.

The aging brain and cognition: contribution of vascular injury and aβ to mild cognitive dysfunction.

http://www.wikidata.org/entity/Q37166455

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Amyloid Proteins and Their Role in Multiple Sclerosis. Considerations in the Use of Amyloid-PET Imaging Human isoprenoid synthase enzymes as therapeutic targets Role of HIV in amyloid metabolism Pathophysiologic relationship between Alzheimer's disease, cerebrovascular disease, and cardiovascular risk: A review and synthesis Apolipoprotein E-low density lipoprotein receptor interaction affects spatial memory retention and brain ApoE levels in an isoform-dependent manner.White matter abnormalities associated with Alzheimer's disease and mild cognitive impairment: a critical review of MRI studies.Advances in MRI biomarkers for the diagnosis of Alzheimer's disease.Diabetes and elevated hemoglobin A1c levels are associated with brain hypometabolism but not amyloid accumulation.Association between neuropathology and brain volume in the Framingham Heart Study.Aggregate effects of vascular risk factors on cerebrovascular changes in autopsy-confirmed Alzheimer's disease Cognitive health begins at conception: addressing dementia as a lifelong and preventable condition HMGCR is a genetic modifier for risk, age of onset and MCI conversion to Alzheimer's disease in a three cohorts study.Vascular and amyloid pathologies are independent predictors of cognitive decline in normal elderly.Clinical and imaging features of mixed Alzheimer and vascular pathologies.Mortality in mild cognitive impairment varies by subtype, sex, and lifestyle factors: the Mayo Clinic Study of Aging.Review of 'the potential role of arterial stiffness in the pathogenesis of Alzheimer's disease'.Humic Acid Increases Amyloid β-Induced Cytotoxicity by Induction of ER Stress in Human SK-N-MC Neuronal Cells Amyloid burden, cortical thickness, and cognitive function in the Wisconsin Registry for Alzheimer's Prevention Glio-vascular changes during ageing in wild-type and Alzheimer's disease-like APP/PS1 mice.Cerebral microvascular rather than parenchymal amyloid-β protein pathology promotes early cognitive impairment in transgenic mice Effects of amyloid and vascular markers on cognitive decline in subcortical vascular dementia.Cerebral white matter lesions - associations with Aβ isoforms and amyloid PET Multimorbidity and neuroimaging biomarkers among cognitively normal persons Much of late life cognitive decline is not due to common neurodegenerative pathologies.Contribution of cerebrovascular disease in autopsy confirmed neurodegenerative disease cases in the National Alzheimer's Coordinating Centre.Effects of rs3846662 Variants on HMGCR mRNA and Protein Levels and on Markers of Alzheimer's Disease Pathology.Neurotoxic saboteurs: straws that break the hippo's (hippocampus) back drive cognitive impairment and Alzheimer's Disease.Alzheimer disease therapy--moving from amyloid-β to tau.Pathogenesis and treatment of vascular cognitive impairment.Cognitive screening in persons with chronic diseases in primary care: challenges and recommendations for practice.Impact of multiple pathologies on the threshold for clinically overt dementia.Cerebral hypoperfusion and glucose hypometabolism: Key pathophysiological modulators promote neurodegeneration, cognitive impairment, and Alzheimer's disease.Disordered APP metabolism and neurovasculature in trauma and aging: Combined risks for chronic neurodegenerative disorders.Detectable Neuropsychological Differences in Early Preclinical Alzheimer's Disease: A Meta-Analysis.Cytochrome P450 eicosanoids in cerebrovascular function and disease.Association Between Midlife Vascular Risk Factors and Estimated Brain Amyloid Deposition.Memory Binding Test Predicts Incident Amnestic Mild Cognitive Impairment.'Two-level' measurements of processing speed as cognitive markers in the differential diagnosis of DSM-5 mild neurocognitive disorders (NCD).Cerebrovascular pathology and Alzheimer's disease.Imaging Vascular Disease and Amyloid in the Aging Brain: Implications for Treatment

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The aging brain and cognition: contribution of vascular injury and aβ to mild cognitive dysfunction.

http://www.wikidata.org/entity/Q37166455

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article científic

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article scientifique

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articolo scientifico

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bilimsel makale

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scientific article published on April 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

The aging brain and cognition: ...... to mild cognitive dysfunction.

@en

The aging brain and cognition: ...... to mild cognitive dysfunction.

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type

label

The aging brain and cognition: ...... to mild cognitive dysfunction.

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The aging brain and cognition: ...... to mild cognitive dysfunction.

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prefLabel

The aging brain and cognition: ...... to mild cognitive dysfunction.

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The aging brain and cognition: ...... to mild cognitive dysfunction.

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2013.JAMANEUROL.405

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The aging brain and cognition: ...... to mild cognitive dysfunction.

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Bruce R Reed

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Cindee M Madison

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Dan M Mungas

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Helena C Chui

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Nerses Sanossian

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Roxana Dhada

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Stephen Kriger

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Wendy J Mack

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William J Jagust

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P2860

The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

The diagnosis of dementia due to Alzheimer's disease: recommendations from the National Institute on Aging-Alzheimer's Association workgroups on diagnostic guidelines for Alzheimer's disease

The diagnosis of mild cognitive impairment due to Alzheimer's disease: recommendations from the National Institute on Aging-Alzheimer's Association workgroups on diagnostic guidelines for Alzheimer's disease

Hypothetical model of dynamic biomarkers of the Alzheimer's pathological cascade

Neuroanatomical substrates of age-related cognitive decline

Restoration of MRI data for intensity non-uniformities using local high order intensity statistics.

Pittsburgh compound B imaging and prediction of progression from cognitive normality to symptomatic Alzheimer disease

Conversion of amyloid positive and negative MCI to AD over 3 years: an 11C-PIB PET study.

Longitudinal cognitive decline is associated with fibrillar amyloid-beta measured by [11C]PiB

Increased metabolic vulnerability in early-onset Alzheimer's disease is not related to amyloid burden

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488-495

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scholarly article

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10.1001/2013.JAMANEUROL.405

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P407

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4

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70

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Charles DeCarli

Michael W. Weiner

Natalie L Marchant

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2013-04-01T00:00:00Z

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23400560

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3771392

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