Localized low-level re-expression of high-affinity mesolimbic nicotinic acetylcholine receptors restores nicotine-induced locomotion but not place conditioning.
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Molecular mechanisms underlying behaviors related to nicotine addictionTargeting the noradrenergic system for gender-sensitive medication development for tobacco dependenceMolecules and circuits involved in nicotine addiction: The many faces of smokingPreclinical evidence that activation of mesolimbic alpha 6 subunit containing nicotinic acetylcholine receptors supports nicotine addiction phenotypeLow dose nicotine and antagonism of β2 subunit containing nicotinic acetylcholine receptors have similar effects on affective behavior in miceMouse models for studying genetic influences on factors determining smoking cessation success in humans.Alpha-conotoxin MII-sensitive nicotinic acetylcholine receptors in the nucleus accumbens shell regulate progressive ratio responding maintained by nicotine.Peroxisome proliferator-activated receptors-alpha modulate dopamine cell activity through nicotinic receptors.Genetically expressed HIV-1 viral proteins attenuate nicotine-induced behavioral sensitization and alter mesocorticolimbic ERK and CREB signaling in rats.Nicotine decreases food intake through activation of POMC neurons.Differential expression of the beta4 neuronal nicotinic receptor subunit affects tolerance development and nicotinic binding sites following chronic nicotine treatmentAn autoradiographic survey of mouse brain nicotinic acetylcholine receptors defined by null mutantsDecreased α4β2 nicotinic receptor number in the absence of mRNA changes suggests post-transcriptional regulation in the spontaneously hypertensive rat model of ADHDAnxiolytic-like and anxiogenic-like effects of nicotine are regulated via diverse action at β2*nicotinic acetylcholine receptorsIntra-ventral tegmental area HIV-1 Tat1-86 attenuates nicotine-mediated locomotor sensitization and alters mesocorticolimbic ERK and CREB signaling in ratsNicotinic receptor contributions to smoking: insights from human studies and animal modelsα4β2 nicotinic acetylcholine receptors on dopaminergic neurons mediate nicotine reward and anxiety relief.Diverse strategies targeting α7 homomeric and α6β2* heteromeric nicotinic acetylcholine receptors for smoking cessation.Menthol disrupts nicotine's psychostimulant properties in an age and sex-dependent manner in C57BL/6J mice.Repeated in vivo exposure of cocaine induces long-lasting synaptic plasticity in hypocretin/orexin-producing neurons in the lateral hypothalamus in mice.Role of α4- and α6-containing nicotinic receptors in the acquisition and maintenance of nicotine self-administration.
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P2860
Localized low-level re-expression of high-affinity mesolimbic nicotinic acetylcholine receptors restores nicotine-induced locomotion but not place conditioning.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 11 December 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Localized low-level re-express ...... on but not place conditioning.
@en
Localized low-level re-express ...... on but not place conditioning.
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type
label
Localized low-level re-express ...... on but not place conditioning.
@en
Localized low-level re-express ...... on but not place conditioning.
@nl
prefLabel
Localized low-level re-express ...... on but not place conditioning.
@en
Localized low-level re-express ...... on but not place conditioning.
@nl
P2093
P2860
P1476
Localized low-level re-express ...... ion but not place conditioning
@en
P2093
D H Brunzell
J M Lindstrom
J M McIntosh
Y S Mineur
P2860
P304
P356
10.1111/J.1601-183X.2008.00468.X
P407
P577
2008-12-11T00:00:00Z