H2-K(b) and H2-D(b) regulate cerebellar long-term depression and limit motor learning.
about
Impaired locomotor learning and altered cerebellar synaptic plasticity in pep-19/PCP4-null miceEndurance Exercise as an "Endogenous" Neuro-enhancement Strategy to Facilitate Motor LearningMajor histocompatibility complex class I molecules protect motor neurons from astrocyte-induced toxicity in amyotrophic lateral sclerosis.Learning and memory ... and the immune system.Rett syndrome and other autism spectrum disorders--brain diseases of immune malfunction?Slow angled-descent forepaw grasping (SLAG): an innate behavioral task for identification of individual experimental mice possessing functional visionDevelopmental lead effects on behavior and brain gene expression in male and female BALB/cAnNTac miceA saturation hypothesis to explain both enhanced and impaired learning with enhanced plasticity.A lack of immune system genes causes loss in high frequency hearing but does not disrupt cochlear synapse maturation in mice.Neuronal MHC class I molecules are involved in excitatory synaptic transmission at the hippocampal mossy fiber synapses of marmoset monkeys.MHC class I molecules are present both pre- and postsynaptically in the visual cortex during postnatal development and in adulthood.Functional deficiency of MHC class I enhances LTP and abolishes LTD in the nucleus accumbens of mice.Dynamic gene expression in the human cerebral cortex distinguishes children from adults.MHC class I immune proteins are critical for hippocampus-dependent memory and gate NMDAR-dependent hippocampal long-term depression.Activity-dependent regulation of MHC class I expression in the developing primary visual cortex of the common marmoset monkey.Novel roles for immune molecules in neural development: implications for neurodevelopmental disorders.Characterization of signaling function and expression of HLA class I molecules in medulloblastoma.Major histocompatibility complex I in brain development and schizophreniaThe major histocompatibility complex and autism spectrum disorder.Neuronal MHC Class I Expression Is Regulated by Activity Driven Calcium Signaling.Neuroprotection from stroke in the absence of MHCI or PirB.Neuroglial expression of the MHCI pathway and PirB receptor is upregulated in the hippocampus with advanced aging.Major histocompatibility complex class I proteins in brain development and plasticity.Human ataxias: a genetic dissection of inositol triphosphate receptor (ITPR1)-dependent signaling.Expression and alternative splicing of classical and nonclassical MHCI genes in the hippocampus and neuromuscular junction.MHCI requires MEF2 transcription factors to negatively regulate synapse density during development and in disease.Efficient subtractive cloning of genes activated by lipopolysaccharide and interferon γ in primary-cultured cortical cells of newborn miceAge-related changes in cerebellar and hypothalamic function accompany non-microglial immune gene expression, altered synapse organization, and excitatory amino acid neurotransmission deficits.A TAP1 null mutation leads to an enlarged olfactory bulb and supernumerary, ectopic olfactory glomeruli.The Major Histocompatibility Complex (MHC) in Schizophrenia: A Review.Classic major histocompatibility complex class I molecules: new actors at the neuromuscular junction.Plasticity in the injured brain: more than molecules matter.Neuronal MHC-I expression and its implications in synaptic function, axonal regeneration and Parkinson's and other brain diseasesThe nervous and the immune systems: conspicuous physiological analogies.Neurons and T cells: Understanding this interaction for inflammatory neurological diseases.CTLA4 blockade elicits paraneoplastic neurological disease in a mouse model.CNS-wide Sexually Dimorphic Induction of the Major Histocompatibility Complex 1 Pathway With Aging.The effects of immune protein CD3ζ development and degeneration of retinal neurons after optic nerve injury.MHC class I: an unexpected role in neuronal plasticity.Neuronal major histocompatibility complex class I molecules are implicated in the generation of asymmetries in hippocampal circuitry.
P2860
Q24634111-CB24D2E2-A676-414A-8F2E-F353AFE73965Q26772814-250E4456-8630-49F6-8165-F12AFCA5B51BQ30277241-AC2CFBBE-C7D7-4F27-92D8-8A50CD992CFDQ30410489-DB0A057A-4E67-4BB8-AF30-45B2ADD58B54Q30426525-97479CDD-8AA6-432D-88C9-65FB7DEBD389Q30449582-C0553AB0-7E42-4E5F-8A6D-B060F3385119Q30525483-1D98B6AF-A0C2-4701-809F-F9AEA59FBFC9Q33364858-AA9DC4D1-EF84-432A-9812-038093A2EE90Q33577742-6D9C0119-DED8-4918-8E0C-F3536C43A698Q34030854-35BB2B56-D5BC-4367-AAFE-BAAF0007E8D3Q34165584-48F2A2B7-4A5F-43D0-8074-16B78B7921E7Q34275910-10E89D19-4E8F-4849-B722-E0814EC0E7D3Q34292595-8B300DC4-B746-4CCD-9AD9-EFF97C7DDEC7Q34365149-71E93684-1F1F-42FB-9E6A-69F977F85CB4Q34503911-1F5AB733-2422-44FC-9D98-D5BCC102F9F6Q34689823-B495BED3-7914-4096-9FBE-40AFA2B34C8EQ34993398-E3C5D414-D364-4A2B-9443-C618109C7DCBQ35163148-1516FFD4-0A78-4F6A-BE48-549998DF170DQ35193880-C227E2DB-A068-4D04-910B-8F81CFB3E4BBQ35742741-7A0D1010-F834-478F-8E3E-A98D86C7A216Q35857345-F8C58DA7-7A00-4061-A427-99CDB8CEE90EQ36149926-79FA7366-9289-40EF-9D5B-757C62D2682AQ36384439-CDF89DE4-AB90-4DD7-809E-0D02BE81C631Q36384774-2436CEEB-C238-4338-8460-35E9CA432F96Q36767935-C02B12EF-9D9B-4DD5-A535-951F99A63A02Q37128147-58AFE38F-532B-4A49-BC0F-D9753D12BEE4Q37296697-A2699F7F-9890-41C6-A2B1-54DADDDD7990Q37361058-C073FA24-7D2D-4A7B-AA0B-ED8C0DAFFC1CQ37400776-55BBE274-B408-4B6D-B9E7-73C5C48874A1Q37626366-45EF73C3-D59B-4176-ABF7-F8A819744D92Q37829178-FEB9E826-AA77-48C6-9F0C-6106C2677E29Q38113758-E92B822F-E8EC-4E28-B2D9-382C69EB0F12Q38263500-29FA2BF6-56DB-436C-A2DC-3E394F224C59Q38268332-1BEB75D8-6B8B-4039-9371-2B8C521252A0Q38582200-3DA8741E-C6D2-44FE-A6D7-C45C9C2B7FF3Q39413457-3205D8DD-2BF6-4D40-B35D-1236CFDE89B3Q40087375-9D9D6282-4604-49DC-8F7D-D12B60CE563DQ41862244-375C3DE8-417F-4B3A-85B2-6AB72285F968Q42115670-6F74CA30-6865-4579-8745-E98A8D3D4268Q42441852-84B66B1C-B558-497B-92EB-8F55F01668D9
P2860
H2-K(b) and H2-D(b) regulate cerebellar long-term depression and limit motor learning.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 03 April 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
H2-K
@nl
H2-K(b) and H2-D(b) regulate cerebellar long-term depression and limit motor learning.
@en
type
label
H2-K
@nl
H2-K(b) and H2-D(b) regulate cerebellar long-term depression and limit motor learning.
@en
prefLabel
H2-K
@nl
H2-K(b) and H2-D(b) regulate cerebellar long-term depression and limit motor learning.
@en
P2093
P2860
P356
P1476
H2-K(b) and H2-D(b) regulate cerebellar long-term depression and limit motor learning
@en
P2093
Akash Datwani
Carla J Shatz
Michael J McConnell
P2860
P304
P356
10.1073/PNAS.0902018106
P407
P577
2009-04-03T00:00:00Z