Methamphetamine preconditioning: differential protective effects on monoaminergic systems in the rat brain
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The effects of methamphetamine self-administration on cortical monoaminergic deficits induced by subsequent high-dose methamphetamine administrationsMethamphetamine preconditioning alters midbrain transcriptional responses to methamphetamine-induced injury in the rat striatumMethamphetamine self-administration is associated with persistent biochemical alterations in striatal and cortical dopaminergic terminals in the ratChronic methamphetamine administration causes differential regulation of transcription factors in the rat midbrainMethamphetamine causes differential alterations in gene expression and patterns of histone acetylation/hypoacetylation in the rat nucleus accumbensChronic methamphetamine exposure suppresses the striatal expression of members of multiple families of immediate early genes (IEGs) in the rat: normalization by an acute methamphetamine injectionMethamphetamine sensitization attenuates the febrile and neuroinflammatory response to a subsequent peripheral immune stimulusMethamphetamine self-administration attenuates hippocampal serotonergic deficits: role of brain-derived neurotrophic factor.Selective vulnerability in striosomes and in the nigrostriatal dopaminergic pathway after methamphetamine administration : early loss of TH in striosomes after methamphetamine.A neurotoxic regimen of methamphetamine exacerbates the febrile and neuroinflammatory response to a subsequent peripheral immune stimulus.Methamphetamine preconditioning causes differential changes in striatal transcriptional responses to large doses of the drug.Long-term protective effects of methamphetamine preconditioning against single-day methamphetamine toxic challenges.Methamphetamine blunts Ca(2+) currents and excitatory synaptic transmission through D1/5 receptor-mediated mechanisms in the mouse medial prefrontal cortexPrior methamphetamine self-administration attenuates serotonergic deficits induced by subsequent high-dose methamphetamine administrations.Role of oxidative stress in methamphetamine-induced dopaminergic toxicity mediated by protein kinase Cδ.Methamphetamine addiction: involvement of CREB and neuroinflammatory signaling pathways.Chronic methamphetamine self-administration disrupts cortical control of cognition.Comparison of single-dose and extended methamphetamine administration on reversal learning in rats.The Main Molecular Mechanisms Underlying Methamphetamine- Induced Neurotoxicity and Implications for Pharmacological Treatment.
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P2860
Methamphetamine preconditioning: differential protective effects on monoaminergic systems in the rat brain
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 24 February 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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Methamphetamine preconditionin ...... ergic systems in the rat brain
@en
Methamphetamine preconditionin ...... rgic systems in the rat brain.
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type
label
Methamphetamine preconditionin ...... ergic systems in the rat brain
@en
Methamphetamine preconditionin ...... rgic systems in the rat brain.
@nl
prefLabel
Methamphetamine preconditionin ...... ergic systems in the rat brain
@en
Methamphetamine preconditionin ...... rgic systems in the rat brain.
@nl
P2093
P2860
P1476
Methamphetamine preconditionin ...... ergic systems in the rat brain
@en
P2093
Bruce Ladenheim
Fidelis E Atianjoh
Irina N Krasnova
Jean Lud Cadet
Michael T McCoy
Ning-Sheng Cai
P2860
P2888
P304
P356
10.1007/S12640-009-9026-0
P577
2009-02-24T00:00:00Z