Pin1-Nanog expression in human glioma is correlated with advanced tumor progression. - Wikidata - awgldk - TriplyDB

Pin1-Nanog expression in human glioma is correlated with advanced tumor progression.

Pin1-Nanog expression in human glioma is correlated with advanced tumor progression.

http://www.wikidata.org/entity/Q37181702

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Inhibition of stearoyl-coA desaturase selectively eliminates tumorigenic Nanog-positive cells: improving the safety of iPS cell transplantation to myocardium Deregulated proliferation and differentiation in brain tumors.Androgen/androgen receptor axis maintains and promotes cancer cell stemness through direct activation of Nanog transcription in hepatocellular carcinoma.Regulation of stem cell function by protein ubiquitylation SAHA and/or MG132 reverse the aggressive phenotypes of glioma cells: An in vitro and vivo study.Juglone reduces growth and migration of U251 glioblastoma cells and disrupts angiogenesis.ING5-mediated antineuroblastoma effects of suberoylanilide hydroxamic acid

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Pin1-Nanog expression in human glioma is correlated with advanced tumor progression.

http://www.wikidata.org/entity/Q37181702

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 23 May 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Pin1-Nanog expression in human glioma is correlated with advanced tumor progression.

@en

Pin1-Nanog expression in human glioma is correlated with advanced tumor progression.

@nl

type

label

Pin1-Nanog expression in human glioma is correlated with advanced tumor progression.

@en

Pin1-Nanog expression in human glioma is correlated with advanced tumor progression.

@nl

prefLabel

Pin1-Nanog expression in human glioma is correlated with advanced tumor progression.

@en

Pin1-Nanog expression in human glioma is correlated with advanced tumor progression.

@nl

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Oncology Reports

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Pin1-Nanog expression in human glioma is correlated with advanced tumor progression.

@en

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Chao-Shi Niu

http://www.w3.org/2001/XMLSchema#string

Chuan-Dong Cheng

http://www.w3.org/2001/XMLSchema#string

Yang Yang

http://www.w3.org/2001/XMLSchema#string

P2860

Pin1 down-regulates transforming growth factor-beta (TGF-beta) signaling by inducing degradation of Smad proteins

The essential mitotic peptidyl-prolyl isomerase Pin1 binds and regulates mitosis-specific phosphoproteins

Exciting new advances in neuro-oncology: the avenue to a cure for malignant glioma

Structural and functional analysis of the mitotic rotamase Pin1 suggests substrate recognition is phosphorylation dependent

Prolyl isomerase Pin1 acts as a switch to control the degree of substrate ubiquitylation.

Identification of a cancer stem cell in human brain tumors

The homeoprotein Nanog is required for maintenance of pluripotency in mouse epiblast and ES cells

Human F-box protein hCdc4 targets cyclin E for proteolysis and is mutated in a breast cancer cell line

Nanog and transcriptional networks in embryonic stem cell pluripotency

Repression of Nanog gene transcription by Tcf3 limits embryonic stem cell self-renewal

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560-566

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scholarly article

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10.3892/OR.2013.2481

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2

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30

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2013-05-23T00:00:00Z

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236939637

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23708493

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