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Genetic Susceptibility to Fungal Infections: What is in the Genes?

Genetic Susceptibility to Fungal Infections: What is in the Genes?

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New Horizons in Antifungal Therapy.Acidic mammalian chitinase negatively affects immune responses during acute and chronic Aspergillus fumigatus exposure.

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Genetic Susceptibility to Fungal Infections: What is in the Genes?

http://www.wikidata.org/entity/Q37183884

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 29 March 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Genetic Susceptibility to Fungal Infections: What is in the Genes?

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Genetic Susceptibility to Fungal Infections: What is in the Genes?

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type

label

Genetic Susceptibility to Fungal Infections: What is in the Genes?

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Genetic Susceptibility to Fungal Infections: What is in the Genes?

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prefLabel

Genetic Susceptibility to Fungal Infections: What is in the Genes?

@en

Genetic Susceptibility to Fungal Infections: What is in the Genes?

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S40588-016-0037-3

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Current clinical microbiology reports

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Genetic Susceptibility to Fungal Infections: What is in the Genes?

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Melissa D Johnson

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Stacey A Maskarinec

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TLR4 mutations are associated with endotoxin hyporesponsiveness in humans

Single-nucleotide polymorphisms (SNPs) in human beta-defensin 1: high-throughput SNP assays and association with Candida carriage in type I diabetics and nondiabetic controls

Autosomal dominant and sporadic monocytopenia with susceptibility to mycobacteria, fungi, papillomaviruses, and myelodysplasia

Human dectin-1 deficiency and mucocutaneous fungal infections

Pathogenesis of Aspergillus fumigatus in Invasive Aspergillosis

A homozygous CARD9 mutation in a family with susceptibility to fungal infections

The beta-glucan receptor dectin-1 recognizes specific morphologies of Aspergillus fumigatus

STAT3 and the Hyper-IgE syndrome: Clinical presentation, genetic origin, pathogenesis, novel findings and remaining uncertainties

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Genetically-determined hyperfunction of the S100B/RAGE axis is a risk factor for aspergillosis in stem cell transplant recipients

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s40588-016-0037-3

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81-91

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scholarly article

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10.1007/S40588-016-0037-3

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