Growth factors and vitamin E modify neuronal glutamate toxicity
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Neurotrophin-mediated potentiation of neuronal injury.Protection against oxidative stress-induced neuronal cell death--a novel role for RU486.Risk factors for Alzheimer's disease: role of multiple antioxidants, non-steroidal anti-inflammatory and cholinergic agents alone or in combination in prevention and treatment.Inhibition of PC12 cell redox activity is a specific, early indicator of the mechanism of beta-amyloid-mediated cell deathOxidative Stress in Alzheimer's Disease: Should We Keep Trying Antioxidant Therapies?Researching glutamate - induced cytotoxicity in different cell lines: a comparative/collective analysis/study.The neuroprotective effects of tocotrienol rich fraction and alpha tocopherol against glutamate injury in astrocytesDifferentiation renders susceptibility to excitotoxicity in HT22 neuronsIron and ferroptosis: A still ill-defined liaison.Excitotoxicity effects of glutamate on human neuroblastoma SH-SY5Y cells via oxidative damage.Gene transfer of glial cell-derived neurotrophic factor and cardiotrophin-1 protects PC12 cells from injury: involvement of the phosphatidylinositol 3-kinase and mitogen-activated protein kinase kinase pathways.Tyrphostins protect neuronal cells from oxidative stress.Specificity of resistance to oxidative stress.Neurotoxic effects of apolipoprotein E4 are mediated via dysregulation of calcium homeostasis.Propofol hemisuccinate protects neuronal cells from oxidative injury.The up-regulation of endosomal-lysosomal components in amyloid beta-resistant cells.Resistance to the apoptotic effect of aggregated amyloid-beta peptide in several different cell types including neuronal- and hepatoma-derived cell lines.The serpin-enzyme complex receptor recognizes soluble, nontoxic amyloid-beta peptide but not aggregated, cytotoxic amyloid-beta peptide.CGP37157, an inhibitor of the mitochondrial Na+/Ca2+ exchanger, protects neurons from excitotoxicity by blocking voltage-gated Ca2+ channels.Cellular Pharmacological Effects of the Traditional Japanese Kampo Medicine Yokukansan on Brain Cells.Hypoxia regulates glutamate metabolism and membrane transport in rat PC12 cells.Isoliquiritigenin is a novel NMDA receptor antagonist in kampo medicine yokukansan.The acute neurotoxic effect of beta-amyloid on mature cultures of rat hippocampal neurons is attenuated by the anti-oxidant U-78517F.In Vitro Neuroprotective and Anti-Inflammatory Activities of Natural and Semi-Synthetic Spirosteroid Analogues.Oxytosis/Ferroptosis-(Re-) Emerging Roles for Oxidative Stress-Dependent Non-apoptotic Cell Death in Diseases of the Central Nervous System.Medicinal herbs and antioxidants: potential of Rhinacanthus nasutus for disease treatment?
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P2860
Growth factors and vitamin E modify neuronal glutamate toxicity
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on September 1992
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Growth factors and vitamin E modify neuronal glutamate toxicity
@en
Growth factors and vitamin E modify neuronal glutamate toxicity.
@nl
type
label
Growth factors and vitamin E modify neuronal glutamate toxicity
@en
Growth factors and vitamin E modify neuronal glutamate toxicity.
@nl
prefLabel
Growth factors and vitamin E modify neuronal glutamate toxicity
@en
Growth factors and vitamin E modify neuronal glutamate toxicity.
@nl
P2093
P2860
P356
P1476
Growth factors and vitamin E modify neuronal glutamate toxicity
@en
P2093
P2860
P304
P356
10.1073/PNAS.89.17.8264
P407
P577
1992-09-01T00:00:00Z