Genetic manipulation of periostin expression reveals a role in cardiac hypertrophy and ventricular remodeling.
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Gq/11-mediated signaling and hypertrophy in mice with cardiac-specific transgenic expression of regulator of G-protein signaling 2Periostin shows increased evolutionary plasticity in its alternatively spliced regionDefects in tendon, ligament, and enthesis in response to genetic alterations in key proteoglycans and glycoproteins: a reviewIntegrated genomic approaches implicate osteoglycin (Ogn) in the regulation of left ventricular massPeriostin and TGF-β-induced protein: Two peas in a pod?Searching novel diagnostic markers and targets for therapy of CKDHarnessing Hippo in the heart: Hippo/Yap signaling and applications to heart regeneration and rejuvenationOrigin of cardiomyocytes in the adult heartIntrapericardial delivery of gelfoam enables the targeted delivery of Periostin peptide after myocardial infarction by inducing fibrin clot formationSystems biology-opportunities and challenges: the application of proteomics to study the cardiovascular extracellular matrixPhysiological Implications of Myocardial Scar StructurePeriostin, a member of a novel family of vitamin K-dependent proteins, is expressed by mesenchymal stromal cellsCollagen XIV is important for growth and structural integrity of the myocardiumAngiotensin II increases periostin expression via Ras/p38 MAPK/CREB and ERK1/2/TGF-β1 pathways in cardiac fibroblastsLack of periostin leads to suppression of Notch1 signaling and calcific aortic valve diseaseSystemic injection of AAV9 carrying a periostin promoter targets gene expression to a myofibroblast-like lineage in mouse hearts after reperfused myocardial infarction.Periostin promotes fibrosis and predicts progression in patients with idiopathic pulmonary fibrosis.Cardiac fibroblast: the renaissance cell.Genome-wide profiling identifies epithelial cell genes associated with asthma and with treatment response to corticosteroidsAblation of periostin inhibits post-infarction myocardial regeneration in neonatal mice mediated by the phosphatidylinositol 3 kinase/glycogen synthase kinase 3β/cyclin D1 signalling pathway.Molecular and physiological characterization of RV remodeling in a murine model of pulmonary stenosis.Transcriptome profiles of carcinoma-in-situ and invasive non-small cell lung cancer as revealed by SAGECardiac fibroblasts are essential for the adaptive response of the murine heart to pressure overload.Periostin and Discoidin Domain Receptor 1: New Biomarkers or Targets for Therapy of Renal Disease.Myofibroblasts and the extracellular matrix network in post-myocardial infarction cardiac remodelingPeriostin associates with Notch1 precursor to maintain Notch1 expression under a stress condition in mouse cellsExpression profile of matricellular proteins in hypertrophied right ventricle of monocrotaline-induced pulmonary hypertensive rats.Delayed re-epithelialization in periostin-deficient mice during cutaneous wound healing.Fibroblast deletion of ROCK2 attenuates cardiac hypertrophy, fibrosis, and diastolic dysfunctionALMS1-deficient fibroblasts over-express extra-cellular matrix components, display cell cycle delay and are resistant to apoptosis.Endometrial gene expression in early pregnancy: lessons from human ectopic pregnancy.Genetic lineage tracing defines myofibroblast origin and function in the injured heart.Periostin promotes a fibroblastic lineage pathway in atrioventricular valve progenitor cellsDifferential protein expression and basal lamina remodeling in human heart failurePreserved heart function and maintained response to cardiac stresses in a genetic model of cardiomyocyte-targeted deficiency of cyclooxygenase-2.Periostin advances atherosclerotic and rheumatic cardiac valve degeneration by inducing angiogenesis and MMP production in humans and rodents.Inhibition of periostin expression protects against the development of renal inflammation and fibrosis.The Notch pathway controls fibrotic and regenerative repair in the adult heart.Cardiac fibrosis in mice with hypertrophic cardiomyopathy is mediated by non-myocyte proliferation and requires Tgf-β.Neonatal periostin knockout mice are protected from hyperoxia-induced alveolar simplication.
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Genetic manipulation of periostin expression reveals a role in cardiac hypertrophy and ventricular remodeling.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 14 June 2007
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Genetic manipulation of perios ...... hy and ventricular remodeling.
@en
Genetic manipulation of perios ...... hy and ventricular remodeling.
@nl
type
label
Genetic manipulation of perios ...... hy and ventricular remodeling.
@en
Genetic manipulation of perios ...... hy and ventricular remodeling.
@nl
prefLabel
Genetic manipulation of perios ...... hy and ventricular remodeling.
@en
Genetic manipulation of perios ...... hy and ventricular remodeling.
@nl
P2093
P2860
P1433
P1476
Genetic manipulation of perios ...... hy and ventricular remodeling.
@en
P2093
Angela Lorts
Eric W Brunskill
Gerald W Dorn
Jaime Melendez
Jeffrey Robbins
Michael Hambleton
Michelle A Sargent
Robert A Kaiser
Simon J Conway
P2860
P304
P356
10.1161/CIRCRESAHA.107.149047
P577
2007-06-14T00:00:00Z