Genetic manipulation of periostin expression reveals a role in cardiac hypertrophy and ventricular remodeling. - Wikidata - awgldk - TriplyDB

Genetic manipulation of periostin expression reveals a role in cardiac hypertrophy and ventricular remodeling.

Genetic manipulation of periostin expression reveals a role in cardiac hypertrophy and ventricular remodeling.

http://www.wikidata.org/entity/Q37187381

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Gq/11-mediated signaling and hypertrophy in mice with cardiac-specific transgenic expression of regulator of G-protein signaling 2 Periostin shows increased evolutionary plasticity in its alternatively spliced region Defects in tendon, ligament, and enthesis in response to genetic alterations in key proteoglycans and glycoproteins: a review Integrated genomic approaches implicate osteoglycin (Ogn) in the regulation of left ventricular mass Periostin and TGF-β-induced protein: Two peas in a pod?Searching novel diagnostic markers and targets for therapy of CKD Harnessing Hippo in the heart: Hippo/Yap signaling and applications to heart regeneration and rejuvenation Origin of cardiomyocytes in the adult heart Intrapericardial delivery of gelfoam enables the targeted delivery of Periostin peptide after myocardial infarction by inducing fibrin clot formation Systems biology-opportunities and challenges: the application of proteomics to study the cardiovascular extracellular matrix Physiological Implications of Myocardial Scar Structure Periostin, a member of a novel family of vitamin K-dependent proteins, is expressed by mesenchymal stromal cells Collagen XIV is important for growth and structural integrity of the myocardium Angiotensin II increases periostin expression via Ras/p38 MAPK/CREB and ERK1/2/TGF-β1 pathways in cardiac fibroblasts Lack of periostin leads to suppression of Notch1 signaling and calcific aortic valve disease Systemic injection of AAV9 carrying a periostin promoter targets gene expression to a myofibroblast-like lineage in mouse hearts after reperfused myocardial infarction.Periostin promotes fibrosis and predicts progression in patients with idiopathic pulmonary fibrosis.Cardiac fibroblast: the renaissance cell.Genome-wide profiling identifies epithelial cell genes associated with asthma and with treatment response to corticosteroids Ablation of periostin inhibits post-infarction myocardial regeneration in neonatal mice mediated by the phosphatidylinositol 3 kinase/glycogen synthase kinase 3β/cyclin D1 signalling pathway.Molecular and physiological characterization of RV remodeling in a murine model of pulmonary stenosis.Transcriptome profiles of carcinoma-in-situ and invasive non-small cell lung cancer as revealed by SAGE Cardiac fibroblasts are essential for the adaptive response of the murine heart to pressure overload.Periostin and Discoidin Domain Receptor 1: New Biomarkers or Targets for Therapy of Renal Disease.Myofibroblasts and the extracellular matrix network in post-myocardial infarction cardiac remodeling Periostin associates with Notch1 precursor to maintain Notch1 expression under a stress condition in mouse cells Expression profile of matricellular proteins in hypertrophied right ventricle of monocrotaline-induced pulmonary hypertensive rats.Delayed re-epithelialization in periostin-deficient mice during cutaneous wound healing.Fibroblast deletion of ROCK2 attenuates cardiac hypertrophy, fibrosis, and diastolic dysfunction ALMS1-deficient fibroblasts over-express extra-cellular matrix components, display cell cycle delay and are resistant to apoptosis.Endometrial gene expression in early pregnancy: lessons from human ectopic pregnancy.Genetic lineage tracing defines myofibroblast origin and function in the injured heart.Periostin promotes a fibroblastic lineage pathway in atrioventricular valve progenitor cells Differential protein expression and basal lamina remodeling in human heart failure Preserved heart function and maintained response to cardiac stresses in a genetic model of cardiomyocyte-targeted deficiency of cyclooxygenase-2.Periostin advances atherosclerotic and rheumatic cardiac valve degeneration by inducing angiogenesis and MMP production in humans and rodents.Inhibition of periostin expression protects against the development of renal inflammation and fibrosis.The Notch pathway controls fibrotic and regenerative repair in the adult heart.Cardiac fibrosis in mice with hypertrophic cardiomyopathy is mediated by non-myocyte proliferation and requires Tgf-β.Neonatal periostin knockout mice are protected from hyperoxia-induced alveolar simplication.

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Genetic manipulation of periostin expression reveals a role in cardiac hypertrophy and ventricular remodeling.

http://www.wikidata.org/entity/Q37187381

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 14 June 2007

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Genetic manipulation of perios ...... hy and ventricular remodeling.

@en

Genetic manipulation of perios ...... hy and ventricular remodeling.

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type

label

Genetic manipulation of perios ...... hy and ventricular remodeling.

@en

Genetic manipulation of perios ...... hy and ventricular remodeling.

@nl

prefLabel

Genetic manipulation of perios ...... hy and ventricular remodeling.

@en

Genetic manipulation of perios ...... hy and ventricular remodeling.

@nl

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CIRCRESAHA.107.149047

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Circulation Research

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Genetic manipulation of perios ...... hy and ventricular remodeling.

@en

P2093

Angela Lorts

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Eric W Brunskill

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Gerald W Dorn

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Jaime Melendez

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Jeffrey Robbins

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Jian Xu

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Michael Hambleton

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Michelle A Sargent

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Robert A Kaiser

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Simon J Conway

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P2860

Periostin secreted by epithelial ovarian carcinoma is a ligand for alpha(V)beta(3) and alpha(V)beta(5) integrins and promotes cell motility

Osteoblast-specific factor 2: cloning of a putative bone adhesion protein with homology with the insect protein fasciclin I

Beta IG-H3, a novel secretory protein inducible by transforming growth factor-beta, is present in normal skin and promotes the adhesion and spreading of dermal fibroblasts in vitro

Identification and characterization of a novel protein, periostin, with restricted expression to periosteum and periodontal ligament and increased expression by transforming growth factor beta

Periostin as a novel factor responsible for ventricular dilation

Phosphatidylinositol-3-kinase signaling mediates vascular smooth muscle cell expression of periostin in vivo and in vitro.

Expression and characterization of murine osteoblast-specific factor 2 (OSF-2) in a baculovirus expression system

periostin null mice exhibit dwarfism, incisor enamel defects, and an early-onset periodontal disease-like phenotype.

Myocyte-restricted focal adhesion kinase deletion attenuates pressure overload-induced hypertrophy

Calcineurin/NFAT coupling participates in pathological, but not physiological, cardiac hypertrophy.

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313-321

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scholarly article

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10.1161/CIRCRESAHA.107.149047

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3

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101

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Jeffery D. Molkentin

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2007-06-14T00:00:00Z

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17569887

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2680305

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