Genetic modulation of apoptotic pathways fails to alter disease course in tripeptidyl-peptidase 1 deficient mice.
about
Parent-reported benefits of flupirtine in juvenile neuronal ceroid lipofuscinosis (Batten disease; CLN3) are not supported by quantitative data.Therapeutic approaches to the challenge of neuronal ceroid lipofuscinoses.Chronic Enzyme Replacement to the Brain of a Late Infantile Neuronal Ceroid Lipofuscinosis Mouse Has Differential Effects on Phenotypes of Disease.A zebrafish model of CLN2 disease is deficient in tripeptidyl peptidase 1 and displays progressive neurodegeneration accompanied by a reduction in proliferation.
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Genetic modulation of apoptotic pathways fails to alter disease course in tripeptidyl-peptidase 1 deficient mice.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 04 February 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Genetic modulation of apoptoti ...... yl-peptidase 1 deficient mice.
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Genetic modulation of apoptoti ...... yl-peptidase 1 deficient mice.
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type
label
Genetic modulation of apoptoti ...... yl-peptidase 1 deficient mice.
@en
Genetic modulation of apoptoti ...... yl-peptidase 1 deficient mice.
@nl
prefLabel
Genetic modulation of apoptoti ...... yl-peptidase 1 deficient mice.
@en
Genetic modulation of apoptoti ...... yl-peptidase 1 deficient mice.
@nl
P2093
P2860
P1433
P1476
Genetic modulation of apoptoti ...... yl-peptidase 1 deficient mice.
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P2093
David E Sleat
Kwi-Hye Kim
Ora Bernard
Peter Lobel
P2860
P356
10.1016/J.NEULET.2009.01.072
P407
P577
2009-02-04T00:00:00Z