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Mechanisms of chronic central neuropathic pain after spinal cord injuryTramadol and propentofylline coadministration exerted synergistic effects on rat spinal nerve ligation-induced neuropathic painBrief, low frequency stimulation of rat peripheral C-fibres evokes prolonged microglial-induced central sensitization in adults but not in neonatesCellular and Molecular Mechanisms of PainProteomic analysis uncovers novel actions of the neurosecretory protein VGF in nociceptive processing.Harnessing pain heterogeneity and RNA transcriptome to identify blood-based pain biomarkers: a novel correlational study design and bioinformatics approach in a graded chronic constriction injury model.From cytokines to toll-like receptors and beyond - current knowledge and future research needs in irritable bowel syndrome.Medication-overuse headache and opioid-induced hyperalgesia: A review of mechanisms, a neuroimmune hypothesis and a novel approach to treatment.Fractalkine attenuates excito-neurotoxicity via microglial clearance of damaged neurons and antioxidant enzyme heme oxygenase-1 expression2020 Foresight: Envisioning Therapeutic Innovations for Pain.Exploring the neuroimmunopharmacology of opioids: an integrative review of mechanisms of central immune signaling and their implications for opioid analgesia.Behavioral evidence for the differential regulation of p-p38 MAPK and p-NF-κB in rats with trigeminal neuropathic pain.Low level laser therapy alters satellite glial cell expression and reverses nociceptive behavior in rats with neuropathic pain.Examination of sex and minocycline treatment on acute morphine-induced analgesia and inflammatory gene expression along the pain pathway in Sprague-Dawley rats.Gabapentin reduces CX3CL1 signaling and blocks spinal microglial activation in monoarthritic ratsA functional relationship between trigeminal astroglial activation and NR1 expression in a rat model of temporomandibular joint inflammation.Opioids and the gastrointestinal tract - a case of narcotic bowel syndrome and literature review.Spinal mitochondrial-derived peroxynitrite enhances neuroimmune activation during morphine hyperalgesia and antinociceptive tolerance.Fractalkine mediates inflammatory pain through activation of satellite glial cellsExpression and function of CCL2/CCR2 in rat micturition reflexes and somatic sensitivity with urinary bladder inflammationReactive microglia after taste nerve injury: comparison to nerve injury models of chronic pain.Cooperative phagocytes: resident microglia and bone marrow immigrants remove dead photoreceptors in retinal lesions.Multi-targeted neuroprotection by the HSV-2 gene ICP10PK includes robust bystander activity through PI3-K/Akt and/or MEK/ERK-dependent neuronal release of vascular endothelial growth factor and fractalkine.Glial responses after chorda tympani nerve injury.Current challenges and future prospects in management of neuropathic pain.Central sensitization: a generator of pain hypersensitivity by central neural plasticity.Neuropathic pain: a maladaptive response of the nervous system to damage.Long-term control of neuropathic pain in a non-viral gene therapy paradigm.Developmental Changes in Pain and Spinal Immune Gene Expression after Radicular Trauma in the Rat.Chemokines, neuronal-glial interactions, and central processing of neuropathic pain.TNF-alpha and neuropathic pain--a review.Neuron-Microglia Dialogue and Hippocampal Neurogenesis in the Aged Brain.Expression of ATP receptors in the rat dorsal root ganglion and spinal cord.Spinal 5-HT(3) receptor activation induces behavioral hypersensitivity via a neuronal-glial-neuronal signaling cascadeGlial Attenuation With Ibudilast in the Treatment of Medication Overuse Headache: A Double-Blind, Randomized, Placebo-Controlled Pilot Trial of Efficacy and Safety.Chemokines in neuron-glial cell interaction and pathogenesis of neuropathic pain.Spinal 5-HT3 receptors mediate descending facilitation and contribute to behavioral hypersensitivity via a reciprocal neuron-glial signaling cascadeGliopathy ensures persistent inflammation and chronic pain after spinal cord injury.Neural mobilization reverses behavioral and cellular changes that characterize neuropathic pain in rats.The liberation of fractalkine in the dorsal horn requires microglial cathepsin S.
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P2860
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 10 June 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
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name
Glia in pathological pain: a role for fractalkine
@en
Glia in pathological pain: a role for fractalkine.
@nl
type
label
Glia in pathological pain: a role for fractalkine
@en
Glia in pathological pain: a role for fractalkine.
@nl
prefLabel
Glia in pathological pain: a role for fractalkine
@en
Glia in pathological pain: a role for fractalkine.
@nl
P2093
P2860
P1476
Glia in pathological pain: a role for fractalkine
@en
P2093
E D Milligan
E M Sloane
L R Watkins
P2860
P304
P356
10.1016/J.JNEUROIM.2008.04.011
P577
2008-06-10T00:00:00Z