Loss of SOCS3 in myeloid cells prolongs survival in a syngeneic model of glioma. - Wikidata - awgldk - TriplyDB

Loss of SOCS3 in myeloid cells prolongs survival in a syngeneic model of glioma.

Loss of SOCS3 in myeloid cells prolongs survival in a syngeneic model of glioma.

http://www.wikidata.org/entity/Q37190394

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Dysregulation of SOCS-Mediated Negative Feedback of Cytokine Signaling in Carcinogenesis and Its Significance in Cancer Treatment.Developing immunotherapeutic strategies to target brain tumors.SOCS molecules: the growing players in macrophage polarization and function.

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P2860

Loss of SOCS3 in myeloid cells prolongs survival in a syngeneic model of glioma.

http://www.wikidata.org/entity/Q37190394

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 08 March 2016

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Loss of SOCS3 in myeloid cells prolongs survival in a syngeneic model of glioma.

@en

Loss of SOCS3 in myeloid cells prolongs survival in a syngeneic model of glioma.

@nl

type

label

Loss of SOCS3 in myeloid cells prolongs survival in a syngeneic model of glioma.

@en

Loss of SOCS3 in myeloid cells prolongs survival in a syngeneic model of glioma.

@nl

prefLabel

Loss of SOCS3 in myeloid cells prolongs survival in a syngeneic model of glioma.

@en

Loss of SOCS3 in myeloid cells prolongs survival in a syngeneic model of glioma.

@nl

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ONCOTARGET.7992

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P1476

Loss of SOCS3 in myeloid cells prolongs survival in a syngeneic model of glioma

@en

P2093

Amber L Rowse

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Etty N Benveniste

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Hongwei Qin

http://www.w3.org/2001/XMLSchema#string

Magda Gerigk

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Margaret P Marks

http://www.w3.org/2001/XMLSchema#string

Samuel C Fehling

http://www.w3.org/2001/XMLSchema#string

P2860

Immunotherapy for brain cancer: recent progress and future promise

Foxp3 programs the development and function of CD4+CD25+ regulatory T cells

Coordinated regulation of myeloid cells by tumours

Characteristics of the alternative phenotype of microglia/macrophages and its modulation in experimental gliomas

STATs in cancer inflammation and immunity: a leading role for STAT3

Immunocompetent murine models for the study of glioblastoma immunotherapy

Proinflammatory-activated glioma cells induce a switch in microglial polarization and activation status, from a predominant M2b phenotype to a mixture of M1 and M2a/B polarized cells.

Therapeutic modulators of STAT signalling for human diseases

Involvement of the janus kinase/signal transducer and activator of transcription signaling pathway in multiple sclerosis and the animal model of experimental autoimmune encephalomyelitis.

SOCS3 deficiency promotes M1 macrophage polarization and inflammation.

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20621-20635

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scholarly article

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10.18632/ONCOTARGET.7992

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15

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7

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Braden C McFarland

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2016-04-01T00:00:00Z

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297900641

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26967393

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4991480

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