NF-kappaB p50 plays distinct roles in the establishment and control of murine gammaherpesvirus 68 latency.
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The Role of Gammaherpesviruses in Cancer PathogenesisInterplay of Murine Gammaherpesvirus 68 with NF-kappaB Signaling of the HostMurine gamma-herpesvirus 68 hijacks MAVS and IKKbeta to initiate lytic replicationCharacterization of omental immune aggregates during establishment of a latent gammaherpesvirus infectionTracking murine gammaherpesvirus 68 infection of germinal center B cells in vivoIdentification of alternative transcripts encoding the essential murine gammaherpesvirus lytic transactivator RTAInvolvement of TLR2 in recognition of acute gammaherpesvirus-68 infection.Inhibition of NF-kappaB signaling reduces virus load and gammaherpesvirus-induced pulmonary fibrosisViral latency and its regulation: lessons from the gamma-herpesviruses.Histone deacetylases and the nuclear receptor corepressor regulate lytic-latent switch gene 50 in murine gammaherpesvirus 68-infected macrophagesMurine Gammaherpesvirus 68 Pathogenesis Is Independent of Caspase-1 and Caspase-11 in Mice and Impairs Interleukin-1β Production upon Extrinsic Stimulation in Culture.Tiled microarray identification of novel viral transcript structures and distinct transcriptional profiles during two modes of productive murine gammaherpesvirus 68 infection.Infection-Induced Retrotransposon-Derived Noncoding RNAs Enhance Herpesviral Gene Expression via the NF-κB PathwayAtaxia telangiectasia mutated kinase controls chronic gammaherpesvirus infection.The absence of M1 leads to increased establishment of murine gammaherpesvirus 68 latency in IgD-negative B cells.Ablation of STAT3 in the B Cell Compartment Restricts Gammaherpesvirus Latency In Vivo.Is murine gammaherpesvirus-68 (MHV-68) a suitable immunotoxicological model for examining immunomodulatory drug-associated viral recrudescence?RTA Occupancy of the Origin of Lytic Replication during Murine Gammaherpesvirus 68 Reactivation from B Cell Latency.Use of a virus-encoded enzymatic marker reveals that a stable fraction of memory B cells expresses latency-associated nuclear antigen throughout chronic gammaherpesvirus infection.A DNA-sensing-independent role of a nuclear RNA helicase, DHX9, in stimulation of NF-κB-mediated innate immunity against DNA virus infection
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NF-kappaB p50 plays distinct roles in the establishment and control of murine gammaherpesvirus 68 latency.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 04 March 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
NF-kappaB p50 plays distinct r ...... e gammaherpesvirus 68 latency.
@en
NF-kappaB p50 plays distinct r ...... e gammaherpesvirus 68 latency.
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type
label
NF-kappaB p50 plays distinct r ...... e gammaherpesvirus 68 latency.
@en
NF-kappaB p50 plays distinct r ...... e gammaherpesvirus 68 latency.
@nl
prefLabel
NF-kappaB p50 plays distinct r ...... e gammaherpesvirus 68 latency.
@en
NF-kappaB p50 plays distinct r ...... e gammaherpesvirus 68 latency.
@nl
P2860
P356
P1433
P1476
NF-kappaB p50 plays distinct r ...... e gammaherpesvirus 68 latency.
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P2093
Christopher M Collins
Samuel H Speck
P2860
P304
P356
10.1128/JVI.00111-09
P407
P577
2009-03-04T00:00:00Z