Brain nuclear factor-kappa B activation contributes to neurohumoral excitation in angiotensin II-induced hypertension - Wikidata - awgldk - TriplyDB

Brain nuclear factor-kappa B activation contributes to neurohumoral excitation in angiotensin II-induced hypertension

Brain nuclear factor-kappa B activation contributes to neurohumoral excitation in angiotensin II-induced hypertension

http://www.wikidata.org/entity/Q37193534

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Autonomic-Immune-Vascular Interaction: An Emerging Concept for Neurogenic Hypertension Effects of tempol and redox-cycling nitroxides in models of oxidative stress.Anti-hypertensive Herbs and their Mechanisms of Action: Part I Brain inflammation and hypertension: the chicken or the egg?Neuroinflammatory basis of metabolic syndrome Brain mineralocorticoid receptors in cognition and cardiovascular homeostasis Neuroinflammation in overnutrition-induced diseases Autonomic nervous system and immune system interactions The inextricable role of the kidney in hypertension Chronic estrogen exposure affects gene expression in the rostral ventrolateral medulla of young and aging rats: Possible role in hypertension Attenuation of renal excretory responses to ANG II during inhibition of superoxide dismutase in anesthetized rats.The central renin-angiotensin system and sympathetic nerve activity in chronic heart failure.Role of the hypothalamic PVN in the regulation of renal sympathetic nerve activity and blood flow during hyperthermia and in heart failure Silencing nox4 in the paraventricular nucleus improves myocardial infarction-induced cardiac dysfunction by attenuating sympathoexcitation and periinfarct apoptosis.Obesity induces neuroinflammation mediated by altered expression of the renin-angiotensin system in mouse forebrain nuclei.Brain microglial cytokines in neurogenic hypertension HDAC inhibition attenuates inflammatory, hypertrophic, and hypertensive responses in spontaneously hypertensive rats.Angiotensin II-induced upregulation of AT(1) receptor expression: sequential activation of NF-kappaB and Elk-1 in neurons.Blockade of brain angiotensin II AT1 receptors ameliorates stress, anxiety, brain inflammation and ischemia: Therapeutic implications.Detraining differentially preserved beneficial effects of exercise on hypertension: effects on blood pressure, cardiac function, brain inflammatory cytokines and oxidative stress.Inhibition of TNF in the brain reverses alterations in RAS components and attenuates angiotensin II-induced hypertension Agonistic antibody to angiotensin II type 1 receptor accelerates atherosclerosis in ApoE-/- mice.Inflammatory links between obesity and metabolic disease.Paraventricular nucleus corticotrophin releasing hormone contributes to sympathoexcitation via interaction with neurotransmitters in heart failure.Toll-like receptor 4 inhibition within the paraventricular nucleus attenuates blood pressure and inflammatory response in a genetic model of hypertension Toll-like receptor 4 promotes autonomic dysfunction, inflammation and microglia activation in the hypothalamic paraventricular nucleus: role of endoplasmic reticulum stress.Hypothalamic inflammation: a double-edged sword to nutritional diseases.Hypothalamic paraventricular nucleus activation contributes to neurohumoral excitation in rats with heart failure NF-κB in the paraventricular nucleus modulates neurotransmitters and contributes to sympathoexcitation in heart failure Angiotensin II-induced hypertension is modulated by nuclear factor-κBin the paraventricular nucleus.Chronic NF-κB blockade improves renal angiotensin II type 1 receptor functions and reduces blood pressure in Zucker diabetic rats.ACE2 overexpression in the paraventricular nucleus attenuates angiotensin II-induced hypertension Angiotensin II AT(1) receptor blockers ameliorate inflammatory stress: a beneficial effect for the treatment of brain disorders.Activation of central PPAR-γ attenuates angiotensin II-induced hypertension.The central mechanism underlying hypertension: a review of the roles of sodium ions, epithelial sodium channels, the renin-angiotensin-aldosterone system, oxidative stress and endogenous digitalis in the brain.Central blockade of salusin β attenuates hypertension and hypothalamic inflammation in spontaneously hypertensive rats.Endoplasmic reticulum and oxidant stress mediate nuclear factor-κB activation in the subfornical organ during angiotensin II hypertension Neuroinflammation and oxidative stress in rostral ventrolateral medulla contribute to neurogenic hypertension induced by systemic inflammation Cross talk between AT1 receptors and Toll-like receptor 4 in microglia contributes to angiotensin II-derived ROS production in the hypothalamic paraventricular nucleus Immunohistochemical Localization of AT1a, AT1b, and AT2 Angiotensin II Receptor Subtypes in the Rat Adrenal, Pituitary, and Brain with a Perspective Commentary.

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Brain nuclear factor-kappa B activation contributes to neurohumoral excitation in angiotensin II-induced hypertension

http://www.wikidata.org/entity/Q37193534

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 25 February 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Brain nuclear factor-kappa B a ...... tensin II-induced hypertension

@en

Brain nuclear factor-kappa B a ...... ensin II-induced hypertension.

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type

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Brain nuclear factor-kappa B a ...... tensin II-induced hypertension

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Brain nuclear factor-kappa B a ...... ensin II-induced hypertension.

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prefLabel

Brain nuclear factor-kappa B a ...... tensin II-induced hypertension

@en

Brain nuclear factor-kappa B a ...... ensin II-induced hypertension.

@nl

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Cardiovascular Research

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Brain nuclear factor-kappa B a ...... tensin II-induced hypertension

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Carrie Elks

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Jin-Ping Zheng

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Joseph Francis

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Srinivas Sriramula

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Ying Ma

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Yu-Ming Kang

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Zhi-Ming Yang

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P2860

Site-specific phosphorylation of IkappaBalpha by a novel ubiquitination-dependent protein kinase activity

Cardiovascular and renal sympathetic activation by blood-borne TNF-alpha in rat: the role of central prostaglandins

Mitogen-activated protein kinases mediate upregulation of hypothalamic angiotensin II type 1 receptors in heart failure rats

Transcription factor NF-kappaB as a potential biomarker for oxidative stress.

Angiotensin II-mediated hypertension in the rat increases vascular superoxide production via membrane NADH/NADPH oxidase activation. Contribution to alterations of vasomotor tone.

The hypothalamus and hypertension.

Regulation of blood pressure by the type 1A angiotensin II receptor gene.

Angiotensin II as a pro-inflammatory mediator.

Brain renin-angiotensin system dysfunction in hypertension: recent advances and perspectives.

The IKKbeta subunit of IkappaB kinase (IKK) is essential for nuclear factor kappaB activation and prevention of apoptosis.

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503-512

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scholarly article

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10.1093/CVR/CVP073

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3

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82

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24043728

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19246475

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arterial hypertension

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2682616

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