Inactivation of GSK-3beta by metallothionein prevents diabetes-related changes in cardiac energy metabolism, inflammation, nitrosative damage, and remodeling
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Interplay of oxidative, nitrosative/nitrative stress, inflammation, cell death and autophagy in diabetic cardiomyopathyEndoplasmic reticulum stress and diabetic cardiomyopathySpotlights on immunological effects of reactive nitrogen species: When inflammation says nitric oxideMetallothionein plays a prominent role in the prevention of diabetic nephropathy by sulforaphane via up-regulation of Nrf2Curcumin alleviates diabetic cardiomyopathy in experimental diabetic ratsKnockdown of GSK3β increases basal autophagy and AMPK signalling in nutrient-laden human aortic endothelial cellsPPARalpha: energy combustion, hypolipidemia, inflammation and cancerNRF2 Plays a Critical Role in Both Self and EGCG Protection against Diabetic Testicular Damage.Cardiac-specific overexpression of HIF-1{alpha} prevents deterioration of glycolytic pathway and cardiac remodeling in streptozotocin-induced diabetic mice.Zinc is essential for the transcription function of Nrf2 in human renal tubule cells in vitro and mouse kidney in vivo under the diabetic condition.Azelnidipine protects myocardium in hyperglycemia-induced cardiac damageCannabidiol attenuates cardiac dysfunction, oxidative stress, fibrosis, and inflammatory and cell death signaling pathways in diabetic cardiomyopathy.Targeting GSK-3 family members in the heart: a very sharp double-edged sword.Drp1-mediated mitochondrial abnormalities link to synaptic injury in diabetes model.Electric Pulse Stimulation of Myotubes as an In Vitro Exercise Model: Cell-Mediated and Non-Cell-Mediated EffectsCannabinoid 1 receptor promotes cardiac dysfunction, oxidative stress, inflammation, and fibrosis in diabetic cardiomyopathyActivin A levels are associated with abnormal glucose regulation in patients with myocardial infarction: potential counteracting effects of activin A on inflammationMagnolia bioactive constituent 4-O-methylhonokiol prevents the impairment of cardiac insulin signaling and the cardiac pathogenesis in high-fat diet-induced obese mice.FGF21 deletion exacerbates diabetic cardiomyopathy by aggravating cardiac lipid accumulation.Protective Effect of Antenatal Antioxidant on Nicotine-Induced Heart Ischemia-Sensitive Phenotype in Rat OffspringCardiac overexpression of metallothionein rescues cold exposure-induced myocardial contractile dysfunction through attenuation of cardiac fibrosis despite cardiomyocyte mechanical anomalies.Prevention of diabetic nephropathy by sulforaphane: possible role of Nrf2 upregulation and activation.Heavy metal scavenger metallothionein mitigates deep hypothermia-induced myocardial contractile anomalies: role of autophagy.Curcumin Protects Neonatal Rat Cardiomyocytes against High Glucose-Induced Apoptosis via PI3K/Akt Signalling PathwayTaurine attenuates oxidative stress and alleviates cardiac failure in type I diabetic rats.Nutrient-sensing mTORC1: Integration of metabolic and autophagic signalsPoly(ADP-ribose) polymerase 1 inhibition protects cardiomyocytes from inflammation and apoptosis in diabetic cardiomyopathy.Insulin-like growth factor-1 improves diabetic cardiomyopathy through antioxidative and anti-inflammatory processes along with modulation of Akt/GSK-3β signaling in ratsC66 ameliorates diabetic nephropathy in mice by both upregulating NRF2 function via increase in miR-200a and inhibiting miR-21Sulforaphane Prevents Angiotensin II-Induced Testicular Cell Death via Activation of NRF2.Renal improvement by zinc in diabetic mice is associated with glucose metabolism signaling mediated by metallothionein and Akt, but not Akt2.Ischemic preconditioning: Interruption of various disorders.Diabetic inhibition of preconditioning- and postconditioning-mediated myocardial protection against ischemia/reperfusion injury.Zinc homeostasis in the metabolic syndrome and diabetes.Do multiple nuclear factor kappa B activation mechanisms explain its varied effects in the heart?Crosstalk between insulin and Toll-like receptor signaling pathways in the central nervous system.Diabetic cardiomyopathy and its prevention by nrf2: current status.Regulation by glycogen synthase kinase-3 of inflammation and T cells in CNS diseasesProteomic study on the protective mechanism of fibroblast growth factor 21 to ischemia-reperfusion injury.Pathophysiological Fundamentals of Diabetic Cardiomyopathy.
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Inactivation of GSK-3beta by metallothionein prevents diabetes-related changes in cardiac energy metabolism, inflammation, nitrosative damage, and remodeling
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 26 March 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
@cs
name
Inactivation of GSK-3beta by m ...... osative damage, and remodeling
@en
Inactivation of GSK-3beta by m ...... sative damage, and remodeling.
@nl
type
label
Inactivation of GSK-3beta by m ...... osative damage, and remodeling
@en
Inactivation of GSK-3beta by m ...... sative damage, and remodeling.
@nl
prefLabel
Inactivation of GSK-3beta by m ...... osative damage, and remodeling
@en
Inactivation of GSK-3beta by m ...... sative damage, and remodeling.
@nl
P2093
P2860
P356
P1433
P1476
Inactivation of GSK-3beta by m ...... osative damage, and remodeling
@en
P2093
P2860
P304
P356
10.2337/DB08-1697
P407
P577
2009-03-26T00:00:00Z