PRIP promotes tumor formation through enhancing serum-responsive factor-mediated FOS expression
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Transcription coactivator PBP/MED1-deficient hepatocytes are not susceptible to diethylnitrosamine-induced hepatocarcinogenesis in the mouse.Lens fiber cell differentiation and denucleation are disrupted through expression of the N-terminal nuclear receptor box of NCOA6 and result in p53-dependent and p53-independent apoptosis.Coactivators in PPAR-Regulated Gene ExpressionThe nuclear import of oncoprotein hepatitis B X-interacting protein depends on interacting with c-Fos and phosphorylation of both proteins in breast cancer cells.Steroid Hormone Receptor Coregulators in Endocrine Cancers.
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PRIP promotes tumor formation through enhancing serum-responsive factor-mediated FOS expression
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 26 March 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
PRIP promotes tumor formation ...... factor-mediated FOS expression
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PRIP promotes tumor formation ...... actor-mediated FOS expression.
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type
label
PRIP promotes tumor formation ...... factor-mediated FOS expression
@en
PRIP promotes tumor formation ...... actor-mediated FOS expression.
@nl
prefLabel
PRIP promotes tumor formation ...... factor-mediated FOS expression
@en
PRIP promotes tumor formation ...... actor-mediated FOS expression.
@nl
P2093
P2860
P356
P1476
PRIP promotes tumor formation ...... factor-mediated FOS expression
@en
P2093
Yi-Jun Zhu
Yiwei Tony Zhu
P2860
P304
14485-14492
P356
10.1074/JBC.M900935200
P407
P577
2009-03-26T00:00:00Z