Early BDNF treatment ameliorates cell loss in the entorhinal cortex of APP transgenic mice - Wikidata - awgldk - TriplyDB

Early BDNF treatment ameliorates cell loss in the entorhinal cortex of APP transgenic mice

Early BDNF treatment ameliorates cell loss in the entorhinal cortex of APP transgenic mice

http://www.wikidata.org/entity/Q37196325

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Potential therapeutic effects of neurotrophins for acute and chronic neurological diseases Behavioral assays with mouse models of Alzheimer's disease: practical considerations and guidelines.A γ-secretase inhibitor, but not a γ-secretase modulator, induced defects in BDNF axonal trafficking and signaling: evidence for a role for APP Longitudinal analysis of the behavioral phenotype in a novel transgenic rat model of early stages of Alzheimer's disease.Hippocampal neurogenesis and antidepressive therapy: shocking relations.Proneurogenic Group II mGluR antagonist improves learning and reduces anxiety in Alzheimer Aβ oligomer mouse.Nerve Growth Factor Gene Therapy: Activation of Neuronal Responses in Alzheimer Disease.IGF2 ameliorates amyloidosis, increases cholinergic marker expression and raises BMP9 and neurotrophin levels in the hippocampus of the APPswePS1dE9 Alzheimer's disease model mice.CXCR3 promotes plaque formation and behavioral deficits in an Alzheimer's disease model.Brain-derived neurotrophic factor ameliorates learning deficits in a rat model of Alzheimer's disease induced by aβ1-42 Proteome-wide characterization of signalling interactions in the hippocampal CA4/DG subfield of patients with Alzheimer's disease.BBB-Permeable, Neuroprotective, and Neurotrophic Polysaccharide, Midi-GAGR Decreased peripheral brain-derived neurotrophic factor levels in Alzheimer's disease: a meta-analysis study (N=7277).DNA Methylation and Tag SNPs of the BDNF Gene in Conversion of Amnestic Mild Cognitive Impairment into Alzheimer's Disease: A Cross-Sectional Cohort Study.Norepinephrine Protects against Amyloid-β Toxicity via TrkB.Physical activity delays hippocampal neurodegeneration and rescues memory deficits in an Alzheimer disease mouse model PPARγ-coactivator-1α gene transfer reduces neuronal loss and amyloid-β generation by reducing β-secretase in an Alzheimer's disease model.Impaired retrograde transport of axonal autophagosomes contributes to autophagic stress in Alzheimer's disease neurons.Brain-derived neurotrophic factor protects against tau-related neurodegeneration of Alzheimer's disease.Synaptic Dysfunction in Alzheimer's Disease and Glaucoma: From Common Degenerative Mechanisms Toward Neuroprotection.Viral vectors for therapy of neurologic diseases.Autophagy-mediated Regulation of BACE1 Protein Trafficking and Degradation.Consequences of brain-derived neurotrophic factor withdrawal in CNS neurons and implications in disease.Engineered BDNF producing cells as a potential treatment for neurologic disease.Neurotrophin Signaling and Stem Cells-Implications for Neurodegenerative Diseases and Stem Cell Therapy.Regulation of Synaptic Amyloid-β Generation through BACE1 Retrograde Transport in a Mouse Model of Alzheimer's Disease.Withdrawal of BDNF from hippocampal cultures leads to changes in genes involved in synaptic function.BDNF Val66Met predicts cognitive decline in the Wisconsin Registry for Alzheimer's Prevention.Loss of Neuroprotective Factors in Neurodegenerative Dementias: The End or the Starting Point?Oligodendrogenesis and myelinogenesis during postnatal development effect of glatiramer acetate.p75 neurotrophin receptor interacts with and promotes BACE1 localization in endosomes aggravating amyloidogenesis.Oral administration of inosine promotes recovery after experimental spinal cord injury in rat.MR-guided delivery of AAV2-BDNF into the entorhinal cortex of non-human primates.Protective effects of 7,8-dihydroxyflavone on neuropathological and neurochemical changes in a mouse model of Alzheimer's disease.Adeno-Associated Viral Vector (Serotype 2)-Nerve Growth Factor for Patients With Alzheimer Disease: A Randomized Clinical Trial.Physical positioning markedly enhances brain transduction after intrathecal AAV9 infusion

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Early BDNF treatment ameliorates cell loss in the entorhinal cortex of APP transgenic mice

http://www.wikidata.org/entity/Q37196325

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on September 2013

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Early BDNF treatment ameliorat ...... cortex of APP transgenic mice

@en

Early BDNF treatment ameliorat ...... cortex of APP transgenic mice.

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type

label

Early BDNF treatment ameliorat ...... cortex of APP transgenic mice

@en

Early BDNF treatment ameliorat ...... cortex of APP transgenic mice.

@nl

prefLabel

Early BDNF treatment ameliorat ...... cortex of APP transgenic mice

@en

Early BDNF treatment ameliorat ...... cortex of APP transgenic mice.

@nl

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JNEUROSCI.5195-12.2013

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Journal of Neuroscience

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Early BDNF treatment ameliorat ...... cortex of APP transgenic mice

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Alan H Nagahara

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Edward H Koo

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Imre Kovacs

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Ling Wang

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Mark H Tuszynski

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Michael Mateling

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Simone Eggert

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P2860

Efficient selection for high-expression transfectants with a novel eukaryotic vector

Neuropathological stageing of Alzheimer-related changes

A third-generation lentivirus vector with a conditional packaging system

Differential contribution of amygdala and hippocampus to cued and contextual fear conditioning

High-level neuronal expression of abeta 1-42 in wild-type human amyloid protein precursor transgenic mice: synaptotoxicity without plaque formation

Transgenesis by lentiviral vectors: lack of gene silencing in mammalian embryonic stem cells and preimplantation embryos.

Neuroprotective effects of brain-derived neurotrophic factor in rodent and primate models of Alzheimer's disease

Self-inactivating lentivirus vector for safe and efficient in vivo gene delivery.

BDNF and activity-dependent synaptic modulation

Distribution of intracerebral ventricularly administered neurotrophins in rat brain and its correlation with trk receptor expression.

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15596-15602

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scholarly article

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10.1523/JNEUROSCI.5195-12.2013

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39

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Eliezer Masliah

Edward Rockenstein

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2013-09-01T00:00:00Z

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257075229

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24068826

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3782628

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