Depletion of γ-catenin by Histone Deacetylase Inhibition Confers Elimination of CML Stem Cells in Combination with Imatinib
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The antihelminthic drug niclosamide effectively inhibits the malignant phenotypes of uveal melanoma in vitro and in vivo.Cooperative effect of chidamide and chemotherapeutic drugs induce apoptosis by DNA damage accumulation and repair defects in acute myeloid leukemia stem and progenitor cells.Anthelmintic niclosamide suppresses transcription of BCR-ABL fusion oncogene via disabling Sp1 and induces apoptosis in imatinib-resistant CML cells harboring T315I mutant.Dual cyclin-dependent kinase 4/6 inhibition by PD-0332991 induces apoptosis and senescence in oesophageal squamous cell carcinoma cells.
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Depletion of γ-catenin by Histone Deacetylase Inhibition Confers Elimination of CML Stem Cells in Combination with Imatinib
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 12 August 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Depletion of γ-catenin by Hist ...... s in Combination with Imatinib
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Depletion of γ-catenin by Hist ...... in Combination with Imatinib.
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type
label
Depletion of γ-catenin by Hist ...... s in Combination with Imatinib
@en
Depletion of γ-catenin by Hist ...... in Combination with Imatinib.
@nl
prefLabel
Depletion of γ-catenin by Hist ...... s in Combination with Imatinib
@en
Depletion of γ-catenin by Hist ...... in Combination with Imatinib.
@nl
P2093
P2860
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Depletion of γ-catenin by Hist ...... s in Combination with Imatinib
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P2093
Jingxuan Pan
Sheng Jiang
Xiaohui Zhu
Yingying Shen
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P304
P356
10.7150/THNO.16139
P577
2016-08-12T00:00:00Z