Blockade of lymphotoxin-beta receptor signaling reduces aspects of Sjögren's syndrome in salivary glands of non-obese diabetic mice - Wikidata - awgldk - TriplyDB

Blockade of lymphotoxin-beta receptor signaling reduces aspects of Sjögren's syndrome in salivary glands of non-obese diabetic mice

Blockade of lymphotoxin-beta receptor signaling reduces aspects of Sjögren's syndrome in salivary glands of non-obese diabetic mice

http://www.wikidata.org/entity/Q37207114

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Recent advances in primary Sjogren's syndrome Ectopic lymphoid follicles: inducible centres for generating antigen-specific immune responses within tissues Control of CD8 T-Cell Infiltration into Tumors by Vasculature and Microenvironment The clinical relevance of animal models in Sjögren's syndrome: the interferon signature from mouse to man The Lymphotoxin Network: orchestrating a type I interferon response to optimize adaptive immunity Epidemiology of Sjögren's Syndrome-from an Oral Perspective Ectopic Tertiary Lymphoid Tissue in Inflammatory Bowel Disease: Protective or Provocateur?Ectopic lymphoid neogenesis in rheumatic autoimmune diseases.Effector lymphocyte-induced lymph node-like vasculature enables naive T-cell entry into tumours and enhanced anti-tumour immunity Peripheral tissue homing receptor control of naïve, effector, and memory CD8 T cell localization in lymphoid and non-lymphoid tissues.Genome-wide DNA methylation patterns in naive CD4+ T cells from patients with primary Sjögren's syndrome.The development of inducible bronchus-associated lymphoid tissue depends on IL-17.Sjögren's syndrome: studying the disease in mice.Lymphotoxin-beta receptor blockade reduces CXCL13 in lacrimal glands and improves corneal integrity in the NOD model of Sjögren's syndrome Inducible tertiary lymphoid structures, autoimmunity, and exocrine dysfunction in a novel model of salivary gland inflammation in C57BL/6 mice Analysis of novel Sjogren's syndrome autoantibodies in patients with dry eyes.Early BAFF receptor blockade mitigates murine Sjögren's syndrome: Concomitant targeting of CXCL13 and the BAFF receptor prevents salivary hypofunction B Cell in Autoimmune Diseases.CXCL13 is elevated in Sjögren's syndrome in mice and humans and is implicated in disease pathogenesis Role of the Lymphotoxin/LIGHT System in the Development and Maintenance of Reticular Networks and Vasculature in Lymphoid Tissues Disturbance of cytokine networks in Sjögren's syndrome.LTβR and CD40: working together in dendritic cells to optimize immune responses.Effects of CXCL13 inhibition on lymphoid follicles in models of autoimmune disease.Classification criteria and treatment modalities in primary Sjögren's syndrome.Biologic treatment in Sjögren's syndrome.Beyond TNF: TNF superfamily cytokines as targets for the treatment of rheumatic diseases.Identification of Novel CD4+ T Cell Subsets in the Target Tissue of Sjögren's Syndrome and Their Differential Regulation by the Lymphotoxin/LIGHT Signaling Axis.Innate lymphoid cells in autoimmunity: emerging regulators in rheumatic diseases.Lymphotoxin signalling in tertiary lymphoid structures and immunotherapy.Pulmonary dendritic cells: thinking globally, acting locally.Strategies to inhibit alloantibody production in alloprimed murine recipients of hematopoietic stem cell grafts.Tertiary Lymphoid Structures in Cancer: Drivers of Antitumor Immunity, Immunosuppression, or Bystander Sentinels in Disease?Immune Cell Infiltration and Tertiary Lymphoid Structures as Determinants of Antitumor Immunity.Non-proliferating plasma cells detected in the salivary glands and bone marrow of autoimmune NOD.B10.H2b mice, a model for primary Sjögren's syndrome.MyD88 signaling causes autoimmune sialadenitis through formation of high endothelial venules and upregulation of LTβ receptor-mediated signaling

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Blockade of lymphotoxin-beta receptor signaling reduces aspects of Sjögren's syndrome in salivary glands of non-obese diabetic mice

http://www.wikidata.org/entity/Q37207114

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 18 February 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Blockade of lymphotoxin-beta r ...... nds of non-obese diabetic mice

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Blockade of lymphotoxin-beta r ...... ds of non-obese diabetic mice.

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Blockade of lymphotoxin-beta r ...... nds of non-obese diabetic mice

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Blockade of lymphotoxin-beta r ...... ds of non-obese diabetic mice.

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Blockade of lymphotoxin-beta r ...... nds of non-obese diabetic mice

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Blockade of lymphotoxin-beta r ...... ds of non-obese diabetic mice.

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Arthritis Research and Therapy

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Blockade of lymphotoxin-beta r ...... nds of non-obese diabetic mice

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Adrian Papandile

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Anne Isine Bolstad

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Kathrine Skarstein

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Margaret K Gatumu

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Roy A Fava

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P2860

LIGHT, a new member of the TNF superfamily, and lymphotoxin alpha are ligands for herpesvirus entry mediator

A lymphotoxin-beta-specific receptor

Characterization of surface lymphotoxin forms. Use of specific monoclonal antibodies and soluble receptors

Lymphotoxin and an associated 33-kDa glycoprotein are expressed on the surface of an activated human T cell hybridoma

Abnormal development of peripheral lymphoid organs in mice deficient in lymphotoxin

Abnormal development of secondary lymphoid tissues in lymphotoxin beta-deficient mice

Distinct roles in lymphoid organogenesis for lymphotoxins alpha and beta revealed in lymphotoxin beta-deficient mice

Lymphotoxin/light, lymphoid microenvironments and autoimmune disease.

A role for surface lymphotoxin in experimental autoimmune encephalomyelitis independent of LIGHT.

Lymphotoxin beta receptor signaling is required for inflammatory lymphangiogenesis in the thyroid.

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10.1186/AR2617

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1

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Jeffrey L. Browning

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2688257

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