Fractalkine suppression during hepatic encephalopathy promotes neuroinflammation in mice. - Wikidata - awgldk - TriplyDB

Fractalkine suppression during hepatic encephalopathy promotes neuroinflammation in mice.

Fractalkine suppression during hepatic encephalopathy promotes neuroinflammation in mice.

http://www.wikidata.org/entity/Q37209413

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Bile Acid-Mediated Sphingosine-1-Phosphate Receptor 2 Signaling Promotes Neuroinflammation during Hepatic Encephalopathy in Mice.Recent advances in hepatic encephalopathy.Role of the indoleamine-2,3-dioxygenase/kynurenine pathway of tryptophan metabolism in behavioral alterations in a hepatic encephalopathy rat model.Tissue-specific Role of CX3CR1 Expressing Immune Cells and Their Relationships with Human Disease.FXR-Mediated Cortical Cholesterol Accumulation Contributes to the Pathogenesis of Type A Hepatic Encephalopathy.

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Fractalkine suppression during hepatic encephalopathy promotes neuroinflammation in mice.

http://www.wikidata.org/entity/Q37209413

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on 26 August 2016

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Fractalkine suppression during hepatic encephalopathy promotes neuroinflammation in mice.

@en

Fractalkine suppression during hepatic encephalopathy promotes neuroinflammation in mice.

@nl

type

label

Fractalkine suppression during hepatic encephalopathy promotes neuroinflammation in mice.

@en

Fractalkine suppression during hepatic encephalopathy promotes neuroinflammation in mice.

@nl

prefLabel

Fractalkine suppression during hepatic encephalopathy promotes neuroinflammation in mice.

@en

Fractalkine suppression during hepatic encephalopathy promotes neuroinflammation in mice.

@nl

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S12974-016-0674-8

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Journal of Neuroinflammation

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Fractalkine suppression during hepatic encephalopathy promotes neuroinflammation in mice.

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Adam Brown

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Gabriel Frampton

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Matthew McMillin

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Sarah Andry

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Sharon DeMorrow

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Stephanie Grant

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P2860

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Current concepts in the assessment and treatment of hepatic encephalopathy

Chronic neuroinflammation and cognitive impairment following transient global cerebral ischemia: role of fractalkine/CX3CR1 signaling

Role for neuronally derived fractalkine in mediating interactions between neurons and CX3CR1-expressing microglia

Changes of liver enzymes and bilirubin during ischemic stroke: mechanisms and possible significance.

Neuronal CCL2 is upregulated during hepatic encephalopathy and contributes to microglia activation and neurological decline.

Hepatic encephalopathy--definition, nomenclature, diagnosis, and quantification: final report of the working party at the 11th World Congresses of Gastroenterology, Vienna, 1998.

Proliferation in the Alzheimer hippocampus is due to microglia, not astroglia, and occurs at sites of amyloid deposition.

Results of a prospective study of acute liver failure at 17 tertiary care centers in the United States.

CX3CL1 reduces neurotoxicity and microglial activation in a rat model of Parkinson's disease.

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