Partial MHC class II constructs inhibit MIF/CD74 binding and downstream effects - Wikidata - awgldk - TriplyDB

Partial MHC class II constructs inhibit MIF/CD74 binding and downstream effects

Partial MHC class II constructs inhibit MIF/CD74 binding and downstream effects

http://www.wikidata.org/entity/Q37210758

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CD74 in Kidney Disease The use of flow cytometry to assess a novel drug efficacy in multiple sclerosis HLA-DRα1 constructs block CD74 expression and MIF effects in experimental autoimmune encephalomyelitis.Sex-dependent treatment of chronic EAE with partial MHC class II constructs.Novel humanized recombinant T cell receptor ligands protect the female brain after experimental stroke Preclinical evaluation of recombinant T cell receptor ligand RTL1000 as a therapeutic agent in ischemic stroke Role of macrophage migration inhibitory factor (MIF) in pollen-induced allergic conjunctivitis and pollen dermatitis in mice.HLA-DRα1-mMOG-35-55 treatment of experimental autoimmune encephalomyelitis reduces CNS inflammation, enhances M2 macrophage frequency, and promotes neuroprotection.Sex differences in the immune response to experimental stroke: Implications for translational research.Predicted structure of MIF/CD74 and RTL1000/CD74 complexes A novel HLA-DRα1-MOG-35-55 construct treats experimental stroke Upregulation of CD74 and its potential association with disease severity in subjects with ischemic stroke.Partial MHC class II constructs as novel immunomodulatory therapy for stroke.Invariant Chain Complexes and Clusters as Platforms for MIF Signaling.CD74/DQA1 dimers predispose to the development of arthritis in humanized mice.Activation status of human microglia is dependent on lesion formation stage and remyelination in multiple sclerosis.Macrophage migration inhibitory factor (MIF) modulates trophic signaling through interaction with serine protease HTRA1.MIF and D-DT are potential disease severity modifiers in male MS subjects.MIF inhibition interferes with the inflammatory and T cell-stimulatory capacity of NOD macrophages and delays autoimmune diabetes onset.Blockade of MIF-CD74 Signalling on Macrophages and Dendritic Cells Restores the Antitumour Immune Response Against Metastatic Melanoma.

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Partial MHC class II constructs inhibit MIF/CD74 binding and downstream effects

http://www.wikidata.org/entity/Q37210758

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 10 April 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

@cs

name

Partial MHC class II constructs inhibit MIF/CD74 binding and downstream effects

@en

Partial MHC class II constructs inhibit MIF/CD74 binding and downstream effects.

@nl

type

label

Partial MHC class II constructs inhibit MIF/CD74 binding and downstream effects

@en

Partial MHC class II constructs inhibit MIF/CD74 binding and downstream effects.

@nl

prefLabel

Partial MHC class II constructs inhibit MIF/CD74 binding and downstream effects

@en

Partial MHC class II constructs inhibit MIF/CD74 binding and downstream effects.

@nl

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European Journal of Immunology

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Partial MHC class II constructs inhibit MIF/CD74 binding and downstream effects

@en

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Arthur A Vandenbark

http://www.w3.org/2001/XMLSchema#string

Dennis Bourdette

http://www.w3.org/2001/XMLSchema#string

Gil Benedek

http://www.w3.org/2001/XMLSchema#string

Gregory G Burrows

http://www.w3.org/2001/XMLSchema#string

Halina Offner

http://www.w3.org/2001/XMLSchema#string

Lin Leng

http://www.w3.org/2001/XMLSchema#string

Richard Bucala

http://www.w3.org/2001/XMLSchema#string

Roberto Meza-Romero

http://www.w3.org/2001/XMLSchema#string

Shayne Andrew

http://www.w3.org/2001/XMLSchema#string

P2860

A proinflammatory cytokine inhibits p53 tumor suppressor activity

Recommended diagnostic criteria for multiple sclerosis: Guidelines from the international panel on the diagnosis of multiple sclerosis

MIF signal transduction initiated by binding to CD74

CD44 is the signaling component of the macrophage migration inhibitory factor-CD74 receptor complex

A functional heteromeric MIF receptor formed by CD74 and CXCR4

Macrophage migration inhibitory factor: a regulator of innate immunity

MIF is a pituitary-derived cytokine that potentiates lethal endotoxaemia

Death receptors: signaling and modulation

MIF is a noncognate ligand of CXC chemokine receptors in inflammatory and atherogenic cell recruitment

Macrophage migration inhibitory factor (MIF) sustains macrophage proinflammatory function by inhibiting p53: regulatory role in the innate immune response

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1309-1321

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scholarly article

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10.1002/EJI.201243162

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5

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43

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2013-04-10T00:00:00Z

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23576302

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3788583

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