New perspective of ClC-Kb/2 Cl- channel physiology in the distal renal tubule. - Wikidata - awgldk - TriplyDB

New perspective of ClC-Kb/2 Cl- channel physiology in the distal renal tubule.

New perspective of ClC-Kb/2 Cl- channel physiology in the distal renal tubule.

http://www.wikidata.org/entity/Q37211833

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Role and mechanisms of regulation of the basolateral Kir 4.1/Kir 5.1K+ channels in the distal tubules.WNK Kinases in Development and Disease.Alkaline activation of ClC-K2 chloride channels switches renal cells from reabsorbing to secreting.PGF2α regulates the basolateral K channels in the distal convoluted tubule.Dietary K+ and Cl- independently regulate basolateral conductance in principal and intercalated cells of the collecting duct.Role of ClC-K and barttin in low potassium-induced sodium chloride cotransporter activation and hypertension in mouse kidney.A selective class of inhibitors for the CLC-Ka chloride ion channel.

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New perspective of ClC-Kb/2 Cl- channel physiology in the distal renal tubule.

http://www.wikidata.org/entity/Q37211833

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 20 January 2016

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

New perspective of ClC-Kb/2 Cl- channel physiology in the distal renal tubule.

@en

New perspective of ClC-Kb/2 Cl- channel physiology in the distal renal tubule.

@nl

type

label

New perspective of ClC-Kb/2 Cl- channel physiology in the distal renal tubule.

@en

New perspective of ClC-Kb/2 Cl- channel physiology in the distal renal tubule.

@nl

prefLabel

New perspective of ClC-Kb/2 Cl- channel physiology in the distal renal tubule.

@en

New perspective of ClC-Kb/2 Cl- channel physiology in the distal renal tubule.

@nl

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AJPRENAL.00577.2015

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American Journal of Physiology - Renal Physiology

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New perspective of ClC-Kb/2 Cl- channel physiology in the distal renal tubule.

@en

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Mykola Mamenko

http://www.w3.org/2001/XMLSchema#string

Oleg Zaika

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Oleh Pochynyuk

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Viktor Tomilin

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Vivek Bhalla

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P2860

Gitelman's variant of Bartter's syndrome, inherited hypokalaemic alkalosis, is caused by mutations in the thiazide-sensitive Na-Cl cotransporter

Bartter's syndrome, hypokalaemic alkalosis with hypercalciuria, is caused by mutations in the Na-K-2Cl cotransporter NKCC2

Two highly homologous members of the ClC chloride channel family in both rat and human kidney

Mutations in the chloride channel gene, CLCNKB, cause Bartter's syndrome type III

Epilepsy, ataxia, sensorineural deafness, tubulopathy, and KCNJ10 mutations

Barttin modulates trafficking and function of ClC-K channels

Collecting duct intercalated cell function and regulation

X-ray structure of a ClC chloride channel at 3.0 A reveals the molecular basis of anion selectivity

Gating the selectivity filter in ClC chloride channels

Structure of a eukaryotic CLC transporter defines an intermediate state in the transport cycle

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scholarly article

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10.1152/AJPRENAL.00577.2015

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10

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2016-01-20T00:00:00Z

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5002062

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