Induction of monocyte chemoattractant protein-1 (MCP-1) and its receptor CCR2 in primary sensory neurons contributes to paclitaxel-induced peripheral neuropathy
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Cytokines as Mediators of Pain-Related Process in Breast CancerMechanisms involved in the development of chemotherapy-induced neuropathyMechanisms of chemotherapy-induced behavioral toxicitiesBasic science and clinical management of painful and non-painful chemotherapy-related neuropathy.[EXPRESS] Ion channels and neuronal hyperexcitability in chemotherapy-induced peripheral neuropathy; cause and effect?Toll-like receptor 4 signaling contributes to Paclitaxel-induced peripheral neuropathy.Chronic cannabinoid receptor 2 activation reverses paclitaxel neuropathy without tolerance or cannabinoid receptor 1-dependent withdrawalA quantitative sensory analysis of peripheral neuropathy in colorectal cancer and its exacerbation by oxaliplatin chemotherapyRole of nociceptor estrogen receptor GPR30 in a rat model of endometriosis pain.Role for monocyte chemoattractant protein-1 in the induction of chronic muscle pain in the ratNovel Therapeutic Targets in Neuroinflammation and Neuropathic PainUrinary N telopeptide levels in predicting the anti-nociceptive responses of zoledronic acid and paclitaxel in a rat model of bone metastases.MAPK signaling downstream to TLR4 contributes to paclitaxel-induced peripheral neuropathy.The Cancer Chemotherapeutic Paclitaxel Increases Human and Rodent Sensory Neuron Responses to TRPV1 by Activation of TLR4New Insights into Potential Prevention and Management Options for Chemotherapy-Induced Peripheral Neuropathy.Characterisation of Immune and Neuroinflammatory Changes Associated with Chemotherapy-Induced Peripheral Neuropathy.Nerve growth factor alters microtubule targeting agent-induced neurotransmitter release but not MTA-induced neurite retraction in sensory neurons.Enhanced performance of macrophage-encapsulated nanoparticle albumin-bound-paclitaxel in hypo-perfused cancer lesionsDorsal Root Ganglion Infiltration by Macrophages Contributes to Paclitaxel Chemotherapy-Induced Peripheral NeuropathyFunctional inhibition of chemokine receptor CCR2 by dicer-substrate-siRNA prevents pain developmentCD8+ T Cells and Endogenous IL-10 Are Required for Resolution of Chemotherapy-Induced Neuropathic Pain.Genetic inactivation and pharmacological blockade of sigma-1 receptors prevent paclitaxel-induced sensory-nerve mitochondrial abnormalities and neuropathic pain in mice.Monocyte chemoattractant protein-1 and the blood-brain barrier.Minimizing chemotherapy-induced peripheral neuropathy: preclinical and clinical development of new perspectives.The therapeutic potential of targeting chemokine signalling in the treatment of chronic pain.Platelet-rich plasma modulates the secretion of inflammatory/angiogenic proteins by inflamed tenocytes.Paclitaxel alters the evoked release of calcitonin gene-related peptide from rat sensory neurons in culture.The possible involvement of JNK activation in the spinal dorsal horn in bortezomib-induced allodynia: the role of TNF-α and IL-1β.A Genome-Wide Association Study Identifies a Novel Locus for Bortezomib-Induced Peripheral Neuropathy in European Patients with Multiple Myeloma.Effects of JWH015 in cytokine secretion in primary human keratinocytes and fibroblasts and its suitability for topical/transdermal delivery.DNA damage mediates changes in neuronal sensitivity induced by the inflammatory mediators, MCP-1 and LPS, and can be reversed by enhancing the DNA repair function of APE1.The Therapeutic Potential of Monocyte/Macrophage Manipulation in the Treatment of Chemotherapy-Induced Painful Neuropathy.DRG Voltage-gated Sodium Channel 1.7 is Up-regulated in Paclitaxel-Induced Neuropathy in Rats and in Humans with Neuropathic Pain.CCR2 upregulation in DRG neurons plays a crucial role in gastric hyperalgesia associated with diabetic gastropathy.Blocking of cytokines signalling attenuates evoked and spontaneous neuropathic pain behaviours in the paclitaxel rat model of chemotherapy-induced neuropathy.Divergent roles of immune cells and their mediators in pain.Levo-Corydalmine Alleviates Neuropathic Cancer Pain Induced by Tumor Compression via the CCL2/CCR2 Pathway.Dorsal root ganglion neurons become hyperexcitable and increase expression of voltage-gated T-type calcium channels (Cav3.2) in paclitaxel-induced peripheral neuropathy.A novel interaction between CX3CR1 and CCR2 signalling in monocytes constitutes an underlying mechanism for persistent vincristine-induced pain.Chemokine CCL2 and its receptor CCR2 in the dorsal root ganglion contribute to oxaliplatin-induced mechanical hypersensitivity.
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P2860
Induction of monocyte chemoattractant protein-1 (MCP-1) and its receptor CCR2 in primary sensory neurons contributes to paclitaxel-induced peripheral neuropathy
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 31 May 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Induction of monocyte chemoatt ...... -induced peripheral neuropathy
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type
label
Induction of monocyte chemoatt ...... -induced peripheral neuropathy
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prefLabel
Induction of monocyte chemoatt ...... -induced peripheral neuropathy
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P2093
P2860
P50
P1433
P1476
Induction of monocyte chemoatt ...... -induced peripheral neuropathy
@en
P2093
Alyssa K Kosturakis
Haijun Zhang
Jessica A Boyette-Davis
Seo-Yeon Yoon
P2860
P304
P356
10.1016/J.JPAIN.2013.03.012
P577
2013-05-31T00:00:00Z