Biphasic regulation of ENaC by TGF-{alpha} and EGF in renal epithelial cells.
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Roles of interstitial fluid pH in diabetes mellitus: Glycolysis and mitochondrial functionRegulation of sodium transport by ENaC in the kidneyAldosterone sensitizes connecting tubule glomerular feedback via the aldosterone receptor GPR30.Role of Rho GDP dissociation inhibitor α in control of epithelial sodium channel (ENaC)-mediated sodium reabsorptionErlotinib preserves renal function and prevents salt retention in doxorubicin treated nephrotic rats.Impaired epithelial Na+ channel activity contributes to cystogenesis and development of autosomal recessive polycystic kidney disease in PCK rats.H-Ras mediates the inhibitory effect of epidermal growth factor on the epithelial Na+ channelProlactin stimulates sodium and chloride ion channels in A6 renal epithelial cells.Hypotonic stress upregulates β- and γ-ENaC expression through suppression of ERK by inducing MKP-1Phosphatidylinositol phosphate-dependent regulation of Xenopus ENaC by MARCKS protein.Regulation of transport in the connecting tubule and cortical collecting ductROS production as a common mechanism of ENaC regulation by EGF, insulin, and IGF-1.Deficiency of renal cortical EGF increases ENaC activity and contributes to salt-sensitive hypertension.Epidermal growth factors in the kidney and relationship to hypertension.The inhibitory effect of Gβγ and Gβ isoform specificity on ENaC activity.Estradiol activates epithelial sodium channels in rat alveolar cells through the G protein-coupled estrogen receptor.Regulation of epithelial sodium channel trafficking by ubiquitination.Epidermal growth factor-mediated proliferation and sodium transport in normal and PKD epithelial cells.Animal toxins and renal ion transport: Another dimension in tropical nephrology.Involvement of ENaC in the development of salt-sensitive hypertension.EGF Receptor Inhibition by Erlotinib Increases Aquaporin 2-Mediated Renal Water Reabsorption.Using myc genes to search for stem cells in the ciliary margin of the Xenopus retina.Cycloheximide and lipopolysaccharide downregulate αENaC mRNA via different mechanisms in alveolar epithelial cells.
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Biphasic regulation of ENaC by TGF-{alpha} and EGF in renal epithelial cells.
description
article científic
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article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 18 March 2009
@en
vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Biphasic regulation of ENaC by TGF-{alpha} and EGF in renal epithelial cells.
@en
Biphasic regulation of ENaC by TGF-{alpha} and EGF in renal epithelial cells.
@nl
type
label
Biphasic regulation of ENaC by TGF-{alpha} and EGF in renal epithelial cells.
@en
Biphasic regulation of ENaC by TGF-{alpha} and EGF in renal epithelial cells.
@nl
prefLabel
Biphasic regulation of ENaC by TGF-{alpha} and EGF in renal epithelial cells.
@en
Biphasic regulation of ENaC by TGF-{alpha} and EGF in renal epithelial cells.
@nl
P2093
P2860
P1476
Biphasic regulation of ENaC by TGF-{alpha} and EGF in renal epithelial cells.
@en
P2093
Bela Malik
Billie Jeanne Duke
Douglas C Eaton
P2860
P304
P356
10.1152/AJPRENAL.90337.2008
P577
2009-03-18T00:00:00Z