Treatment with anti-HMGB1 monoclonal antibody does not affect lupus nephritis in MRL/lpr mice. - Wikidata - awgldk - TriplyDB

Treatment with anti-HMGB1 monoclonal antibody does not affect lupus nephritis in MRL/lpr mice.

Treatment with anti-HMGB1 monoclonal antibody does not affect lupus nephritis in MRL/lpr mice.

http://www.wikidata.org/entity/Q37217002

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Anti-high Mobility Group Box 1 Antibody Ameliorates Albuminuria in MRL/lpr Lupus-Prone Mice.Autoantibodies to box A of high mobility group box 1 in systemic lupus erythematosus.Damage-Associated Molecular Patterns in Inflammatory Diseases

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Treatment with anti-HMGB1 monoclonal antibody does not affect lupus nephritis in MRL/lpr mice.

http://www.wikidata.org/entity/Q37217002

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 13 January 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Treatment with anti-HMGB1 mono ...... pus nephritis in MRL/lpr mice.

@en

Treatment with anti-HMGB1 mono ...... pus nephritis in MRL/lpr mice.

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type

label

Treatment with anti-HMGB1 mono ...... pus nephritis in MRL/lpr mice.

@en

Treatment with anti-HMGB1 mono ...... pus nephritis in MRL/lpr mice.

@nl

prefLabel

Treatment with anti-HMGB1 mono ...... pus nephritis in MRL/lpr mice.

@en

Treatment with anti-HMGB1 mono ...... pus nephritis in MRL/lpr mice.

@nl

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MOLMED.2015.00176

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Molecular Medicine

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Treatment with anti-HMGB1 mono ...... pus nephritis in MRL/lpr mice.

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Arjen Petersen

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Fleur Schaper

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Gerda Horst

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Johanna Westra

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Marc Bijl

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Mirjan M van Timmeren

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Pieter C Limburg

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P2860

A critical cysteine is required for HMGB1 binding to Toll-like receptor 4 and activation of macrophage cytokine release

HMG-1 as a late mediator of endotoxin lethality in mice

HMG proteins: dynamic players in gene regulation and differentiation

Toll-like receptor 9-dependent activation by DNA-containing immune complexes is mediated by HMGB1 and RAGE

Elevated plasma level of HMGB1 is associated with disease activity and combined alterations with IFN-α and TNF-α in systemic lupus erythematosus.

The classification of glomerulonephritis in systemic lupus erythematosus revisited.

Genetics of systemic autoimmunity and glomerulonephritis in mouse models of lupus.

HMGB1 mediates splenomegaly and expansion of splenic CD11b+ Ly-6C(high) inflammatory monocytes in murine sepsis survivors.

MD-2 is required for disulfide HMGB1-dependent TLR4 signaling.

Anti-HMGB1 monoclonal antibody ameliorates immunosuppression after peripheral tissue trauma: attenuated T-lymphocyte response and increased splenic CD11b (+) Gr-1 (+) myeloid-derived suppressor cells require HMGB1.

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molmed.2015.00176

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