Neisseria gonorrhoeae suppresses the oxidative burst of human polymorphonuclear leukocytes
about
Vaccines for gonorrhea: can we rise to the challenge?Two lytic transglycosylases in Neisseria gonorrhoeae impart resistance to killing by lysozyme and human neutrophils.Neisseria gonorrhoeae Evades Calprotectin-Mediated Nutritional Immunity and Survives Neutrophil Extracellular Traps by Production of TdfH.Neisserial Opa Protein-CEACAM Interactions: Competition for Receptors as a Means of Bacterial Invasion and Pathogenesis.An improved method for differentiating cell-bound from internalized particles by imaging flow cytometry.A thermonuclease of Neisseria gonorrhoeae enhances bacterial escape from killing by neutrophil extracellular trapsThe lipooligosaccharide-modifying enzyme LptA enhances gonococcal defence against human neutrophils.Opa+ Neisseria gonorrhoeae exhibits reduced survival in human neutrophils via Src family kinase-mediated bacterial trafficking into mature phagolysosomesNeisseria gonorrhoeae elicits extracellular traps in primary neutrophil culture while suppressing the oxidative burst.Assembly of NADPH oxidase in human neutrophils is modulated by the opacity-associated protein expression State of Neisseria gonorrhoeaeNeisseria gonorrhoeae phagosomes delay fusion with primary granules to enhance bacterial survival inside human neutrophils.Constitutively Opa-expressing and Opa-deficient neisseria gonorrhoeae strains differentially stimulate and survive exposure to human neutrophilsA bacterial siren song: intimate interactions between Neisseria and neutrophils.Resistance of Neisseria gonorrhoeae to neutrophils.Resistance of Neisseria gonorrhoeae to non-oxidative killing by adherent human polymorphonuclear leucocytes.IL-10 administration reduces PGE-2 levels and promotes CR3-mediated clearance of Escherichia coli K1 by phagocytes in meningitisMultiple mechanisms of NADPH oxidase inhibition by type A and type B Francisella tularensis.Deciphering the roles of outer membrane protein A extracellular loops in the pathogenesis of Escherichia coli K1 meningitisThe DNA-binding activity of the Neisseria gonorrhoeae LexA orthologue NG1427 is modulated by oxidation.Phase variation leads to the misidentification of a Neisseria gonorrhoeae virulence geneAttenuation of human neutrophil migration and function by uropathogenic bacteria.Estradiol-Treated Female Mice as Surrogate Hosts for Neisseria gonorrhoeae Genital Tract Infections.New concepts in immunity to Neisseria gonorrhoeae: innate responses and suppression of adaptive immunity favor the pathogen, not the hostA novel mechanism of high-level, broad-spectrum antibiotic resistance caused by a single base pair change in Neisseria gonorrhoeaeNeisseria gonorrhoeae-mediated inhibition of apoptotic signalling in polymorphonuclear leukocytes.Innate immunity against Granulibacter bethesdensis, an emerging gram-negative bacterial pathogenNeisseria gonorrhoeae virulence factor NG1686 is a bifunctional M23B family metallopeptidase that influences resistance to hydrogen peroxide and colony morphology.Neisseria gonorrhoeae selectively suppresses the development of Th1 and Th2 cells, and enhances Th17 cell responses, through TGF-β-dependent mechanisms.gp96 expression in neutrophils is critical for the onset of Escherichia coli K1 (RS218) meningitisSuppression of host adaptive immune responses by Neisseria gonorrhoeae: role of interleukin 10 and type 1 regulatory T cellsStructure-function studies of the Neisseria gonorrhoeae major outer membrane porinThe role of reactive-oxygen-species in microbial persistence and inflammation.Opacity proteins of neisseria gonorrhoeae in lipooligosaccharide mutants lost ability to interact with neutrophil-restricted CEACAM3 (CD66d).Neutrophils to the ROScue: Mechanisms of NADPH Oxidase Activation and Bacterial Resistance.Inactivation of NMB0419, Encoding a Sel1-Like Repeat (SLR) Protein, in Neisseria meningitidis Is Associated with Differential Expression of Genes Belonging to the Fur Regulon and Reduced Intraepithelial Replication.Neisseria meningitidis Lacking the Major Porins PorA and PorB Is Viable and Modulates Apoptosis and the Oxidative Burst of Neutrophils.Glutamate utilization promotes meningococcal survival in vivo through avoidance of the neutrophil oxidative burst.Genetic characterization of the nucleotide excision repair system of Neisseria gonorrhoeae.Filifactor alocis modulates human neutrophil antimicrobial functional responses.Neisseria gonorrhoeae-Induced Inflammatory Pyroptosis in Human Macrophages is Dependent on Intracellular Gonococci and Lipooligosaccharide.
P2860
Q21131153-8246A896-975C-4828-BECC-7D86CC1660B5Q30275502-C66E76A6-E4BC-4F76-9194-E8AD4BBEE576Q30276223-C0A20939-2045-4B26-AB1D-416D566BBEA0Q30276311-9B56F692-78A0-407E-BAD5-E8D922E05490Q30299956-3D238166-AEAD-439F-8E1C-A2A7F3148BE6Q30300918-6B84A46D-C506-4685-8733-EE5B365C3135Q30301074-07002A61-81B5-4436-924D-364BCA3251ADQ30303049-DBD62890-6340-40BF-A347-D0703FBB70A6Q30407881-2E94F9A7-C16B-40EE-9B32-3A19DD1EA8C9Q30411015-1AB46CEC-3DC7-4975-A6B3-97FDB1B5280EQ30411495-10160C73-7BA7-4E7D-8636-BBC33155DA4DQ30415490-C09D8445-779C-4907-9122-306900AA9A85Q30422438-06A77175-D2DF-48CE-A49D-64A8243AE502Q30431442-5E73B5B3-7542-4B56-BEB5-2BF0D20C95B0Q30437971-BC175569-AB09-41C8-A206-8A81823F23D9Q33902365-1E04DDBB-119E-49FC-BB1A-4DC687CFAA33Q34281699-A23DFB0E-0DA2-4F3D-A312-E30F8FE10FB3Q34503241-E18E08E7-45F6-4974-B1FA-565E87051841Q34823139-83EF10F0-6999-4354-A35A-12AA07B1EF78Q34973599-2C103E0C-9D67-4EB1-886D-90DFCB2935DEQ34975465-2CBAAA07-0A57-4F66-BE3C-1E4282581575Q35086017-806E3587-1135-4143-8E3B-49D74791C26AQ35155273-E113C8C8-5A7E-4DD2-A9B8-C3759FA30958Q35219997-6219DC9E-979B-44F0-ACFD-F10B9B27E2A6Q35671589-8A2D05B7-A918-4DAE-B9EA-FD48DA7CA435Q35805503-E48190BA-3213-46E6-AFC1-D48D1A4DEB4AQ35879588-A8E62B82-843F-4660-AA25-717C9C86A582Q35894782-A4D2CF07-4CA5-4B0C-8607-F42DFB790A08Q36510811-0213FF05-535F-4FA3-9D96-911F20D8DBF5Q37266237-AC68DD1B-F594-4584-B0C7-6D1F49858624Q37336078-2131636A-163B-4C5D-AB50-8524D1054DBEQ37845672-882D5AB5-238E-4402-9DAE-B9F3D45AAF6FQ39633488-58328A62-4A94-4A51-9419-3BE811F4C1A7Q40054491-C4744E95-D3F2-4FB6-8DA0-30A899862FCAQ40306129-C7FF1FFD-EBAD-405C-ABD2-D526EA928E18Q40447748-4884020A-928B-4534-B73C-82FBDA5C2986Q42076585-B9356C29-0270-4D74-9A21-7AFCE7997C33Q42929340-4BDC9C13-5C10-4FDA-9D22-436F6C7BAE3EQ47645434-0FB9B036-6F53-433C-93D2-0DA735158DA8Q50054782-235F50F7-0B7A-40E6-AADD-B1C0554635A6
P2860
Neisseria gonorrhoeae suppresses the oxidative burst of human polymorphonuclear leukocytes
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 05 August 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Neisseria gonorrhoeae suppress ...... n polymorphonuclear leukocytes
@en
Neisseria gonorrhoeae suppress ...... polymorphonuclear leukocytes.
@nl
type
label
Neisseria gonorrhoeae suppress ...... n polymorphonuclear leukocytes
@en
Neisseria gonorrhoeae suppress ...... polymorphonuclear leukocytes.
@nl
prefLabel
Neisseria gonorrhoeae suppress ...... n polymorphonuclear leukocytes
@en
Neisseria gonorrhoeae suppress ...... polymorphonuclear leukocytes.
@nl
P2860
P1476
Neisseria gonorrhoeae suppress ...... n polymorphonuclear leukocytes
@en
P2860
P304
P356
10.1111/J.1462-5822.2008.01205.X
P577
2008-08-05T00:00:00Z