De novo expression of connexin hemichannels in denervated fast skeletal muscles leads to atrophy
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The pannexins: past and presentEffects of Nandrolone in the Counteraction of Skeletal Muscle Atrophy in a Mouse Model of Muscle Disuse: Molecular Biology and Functional EvaluationThe Signature of MicroRNA Dysregulation in Muscle Paralyzed by Spinal Cord Injury Includes Downregulation of MicroRNAs that Target Myostatin SignalingDeletion of connexin43 in osteoblasts/osteocytes leads to impaired muscle formation in mice.Amelioration of ischemia-reperfusion-induced muscle injury by the recombinant human MG53 proteinThe absence of dysferlin induces the expression of functional connexin-based hemichannels in human myotubesMechanism of gating by calcium in connexin hemichannelsRegulation of hemichannels and gap junction channels by cytokines in antigen-presenting cells.Neuronal involvement in muscular atrophy.Regulation of pannexin and connexin channels and their functional role in skeletal muscles.Role of reactive oxygen species in age-related neuromuscular deficits.Pannexin 1 channels in skeletal musclesConnexins and their channels in inflammation.Regulation of Skeletal Muscle Myoblast Differentiation and Proliferation by Pannexins.Connexins and Pannexins in Bone and Skeletal Muscle.On Biophysical Properties and Sensitivity to Gap Junction Blockers of Connexin 39 Hemichannels Expressed in HeLa Cells.Denervation-related alterations and biological activity of miRNAs contained in exosomes released by skeletal muscle fibers.Fast skeletal myofibers of mdx mouse, model of Duchenne muscular dystrophy, express connexin hemichannels that lead to apoptosis.Joint diseases: from connexins to gap junctions.The influence of isoflavone for denervation-induced muscle atrophy.Connexins in Cardiovascular and Neurovascular Health and Disease: Pharmacological Implications.miRNA targeted signaling pathway in the early stage of denervated fast and slow muscle atrophy.High glucocorticoid levels during gestation activate the inflammasome in hippocampal oligodendrocytes of the offspring.Angiotensin II-Induced Mesangial Cell Damaged Is Preceded by Cell Membrane Permeabilization Due to Upregulation of Non-Selective Channels.
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P2860
De novo expression of connexin hemichannels in denervated fast skeletal muscles leads to atrophy
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 16 September 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
De novo expression of connexin ...... letal muscles leads to atrophy
@en
De novo expression of connexin ...... etal muscles leads to atrophy.
@nl
type
label
De novo expression of connexin ...... letal muscles leads to atrophy
@en
De novo expression of connexin ...... etal muscles leads to atrophy.
@nl
prefLabel
De novo expression of connexin ...... letal muscles leads to atrophy
@en
De novo expression of connexin ...... etal muscles leads to atrophy.
@nl
P2093
P2860
P356
P1476
De novo expression of connexin ...... letal muscles leads to atrophy
@en
P2093
Bruno A Cisterna
Carlos Puebla
Christopher Cardozo
Juan C Sáez
Klaus Willecke
Luis A Cea
Marina Frank
Ramón Latorre
Xavier F Figueroa
P2860
P304
16229-16234
P356
10.1073/PNAS.1312331110
P407
P577
2013-09-16T00:00:00Z