Increased susceptibility of spinal muscular atrophy fibroblasts to camptothecin is p53-independent
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Proteomic assessment of a cell model of spinal muscular atrophyIdentification of the phosphorylation sites in the survival motor neuron protein by protein kinase A.Declines in Drp1 and parkin expression underlie DNA damage-induced changes in mitochondrial length and neuronal death.DNA Damage Response and DNA Repair in Skeletal Myocytes From a Mouse Model of Spinal Muscular Atrophy.Selective growth inhibition by glycogen synthase kinase-3 inhibitors in tumorigenic HeLa hybrid cells is mediated through NF-κB-dependent GLUT3 expression.
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Increased susceptibility of spinal muscular atrophy fibroblasts to camptothecin is p53-independent
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 16 May 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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Increased susceptibility of sp ...... amptothecin is p53-independent
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Increased susceptibility of sp ...... mptothecin is p53-independent.
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Increased susceptibility of sp ...... amptothecin is p53-independent
@en
Increased susceptibility of sp ...... mptothecin is p53-independent.
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Increased susceptibility of sp ...... amptothecin is p53-independent
@en
Increased susceptibility of sp ...... mptothecin is p53-independent.
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Increased susceptibility of sp ...... amptothecin is p53-independent
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Chia-Yen Wu
Ilsa Gómez-Curet
Mena Scavina
Vicky L Funanage
Wenlan Wang
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P2888
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10.1186/1471-2121-10-40
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2009-05-16T00:00:00Z
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P6179
1032277874