Prenatal ethanol exposure persistently impairs NMDA receptor-dependent activation of extracellular signal-regulated kinase in the mouse dentate gyrus.

Prenatal ethanol exposure persistently impairs NMDA receptor-dependent activation of extracellular signal-regulated kinase in the mouse dentate gyrus.

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http://www.wikidata.org/entity/Q37218819
Effect of alcohol consumption in prenatal life, childhood, and adolescence on child development.Significant long-term, but not short-term, hippocampal-dependent memory impairment in adult rats exposed to alcohol in early postnatal life.Neurological and neuropsychological effects of low and moderate prenatal alcohol exposure.Neonatal alcohol exposure and the hippocampus in developing male rats: effects on behaviorally induced CA1 c-Fos expression, CA1 pyramidal cell number, and contextual fear conditioningDoes moderate drinking harm the fetal brain? Insights from animal models.Moderate prenatal alcohol exposure reduces plasticity and alters NMDA receptor subunit composition in the dentate gyrusA limited access mouse model of prenatal alcohol exposure that produces long-lasting deficits in hippocampal-dependent learning and memory.Hippocampal N-methyl-D-aspartate receptor subunit expression profiles in a mouse model of prenatal alcohol exposure.Prenatal choline supplementation mitigates behavioral alterations associated with prenatal alcohol exposure in rats.Focus on: neurotransmitter systems.Differential effects of the histamine H(3) receptor agonist methimepip on dentate granule cell excitability, paired-pulse plasticity and long-term potentiation in prenatal alcohol-exposed rats.Neuregulin1β improves cognitive dysfunction and up-regulates expression of p-ERK1/2 in rats with chronic omethoate poisoning.Imbalanced synaptic plasticity induced spatial cognition impairment in male offspring rats treated with chronic prenatal ethanol exposure.Prenatal Alcohol Exposure Increases Histamine H3 Receptor-Mediated Inhibition of Glutamatergic Neurotransmission in Rat Dentate Gyrus.Prenatal ethanol exposure enhances NMDAR-dependent long-term potentiation in the adolescent female dentate gyrus.Monoacylglycerol lipase inhibitors reverse paclitaxel-induced nociceptive behavior and proinflammatory markers in a mouse model of chemotherapy-induced neuropathy.
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Prenatal ethanol exposure persistently impairs NMDA receptor-dependent activation of extracellular signal-regulated kinase in the mouse dentate gyrus.

Item
http://www.wikidata.org/entity/Q37218819
description
article científic
@ca
article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
@tr
scientific article published on 20 March 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
@cs
name
Prenatal ethanol exposure pers ...... se in the mouse dentate gyrus.
@en
Prenatal ethanol exposure pers ...... se in the mouse dentate gyrus.
@nl
type
label
Prenatal ethanol exposure pers ...... se in the mouse dentate gyrus.
@en
Prenatal ethanol exposure pers ...... se in the mouse dentate gyrus.
@nl
prefLabel
Prenatal ethanol exposure pers ...... se in the mouse dentate gyrus.
@en
Prenatal ethanol exposure pers ...... se in the mouse dentate gyrus.
@nl
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Prenatal ethanol exposure pers ...... se in the mouse dentate gyrus.
@en
P2093
P2860
P304
P31
P356
10.1111/J.1471-4159.2009.06049.X
P407
P433
P478
P577
P5875
P698
P932