Neuroinflammation in Alzheimer's disease: mechanisms, pathologic consequences, and potential for therapeutic manipulation
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Amyloid Proteins and Their Role in Multiple Sclerosis. Considerations in the Use of Amyloid-PET ImagingTargeting New Candidate Genes by Small Molecules Approaching Neurodegenerative DiseasesThe choreography of neuroinflammation in Huntington's diseaseNeuroinflammation in Alzheimer's diseaseNeuropsychiatric Disturbances in Alzheimer's Disease: What Have We Learned from Neuropathological Studies?Network Topology Analysis of Post-Mortem Brain Microarrays Identifies More Alzheimer's Related Genes and MicroRNAs and Points to Novel Routes for Fighting with the DiseaseTranslational research involving oxidative stress and diseases of aging.Mitochondria-Derived Damage-Associated Molecular Patterns in Neurodegeneration.Synaptic and cognitive improvements by inhibition of 2-AG metabolism are through upregulation of microRNA-188-3p in a mouse model of Alzheimer's disease.Upregulation of TREM2 ameliorates neuropathology and rescues spatial cognitive impairment in a transgenic mouse model of Alzheimer's diseaseAstrocytic and microglial nicotinic acetylcholine receptors: an overlooked issue in Alzheimer's disease.Differential general anesthetic effects on microglial cytokine expression.Silk Nanofiber Hydrogels with Tunable Modulus to Regulate Nerve Stem Cell Fate.Presenilin 2 influences miR146 level and activity in microglia.The induction of neuronal death by up-regulated microglial cathepsin H in LPS-induced neuroinflammation.Anti-aβ oligomer IgG and surface sialic acid in intravenous immunoglobulin: measurement and correlation with clinical outcomes in Alzheimer's disease treatment.Evaluation of inflammation-related genes polymorphisms in Mexican with Alzheimer's disease: a pilot study.Noradrenergic dysfunction in Alzheimer's disease.Active immunization against complement factor C5a: a new therapeutic approach for Alzheimer's diseaseRedox regulation of autophagy in healthy brain and neurodegeneration.Statins Inhibit Fibrillary β-Amyloid Induced Inflammation in a Model of the Human Blood Brain Barrier.The antimicrobial protein, CAP37, is upregulated in pyramidal neurons during Alzheimer's disease.Up-regulation of microglial cathepsin C expression and activity in lipopolysaccharide-induced neuroinflammation.Indomethacin-loaded lipid-core nanocapsules reduce the damage triggered by Aβ1-42 in Alzheimer's disease models.Fine-mapping of the human leukocyte antigen locus as a risk factor for Alzheimer disease: A case-control study.Epistasis analysis links immune cascades and cerebral amyloidosis.Monoacylglycerol lipase is a therapeutic target for Alzheimer's diseaseDok2 mediates the CD200Fc attenuation of Aβ-induced changes in glia.Astrocyte-Microglia Cross Talk through Complement Activation Modulates Amyloid Pathology in Mouse Models of Alzheimer's Disease.Signaling pathways regulating neuron-glia interaction and their implications in Alzheimer's disease.HMGB1 and thrombin mediate the blood-brain barrier dysfunction acting as biomarkers of neuroinflammation and progression to neurodegeneration in Alzheimer's disease.Neutrophil Gelatinase-Associated Lipocalin and its Receptors in Alzheimer's Disease (AD) Brain Regions: Differential Findings in AD with and without Depression.A pilot study on the use of interferon beta-1a in early Alzheimer's disease subjects.Oxidative and Nitrosative Stress and Immune-Inflammatory Pathways in Patients with Myalgic Encephalomyelitis (ME)/Chronic Fatigue Syndrome (CFS).Cannabinoids for treatment of Alzheimer's disease: moving toward the clinic.Rhodiola rosea L. and Alzheimer's Disease: From Farm to Pharmacy.Neuronal Pentraxin 2 predicts medial temporal atrophy and memory decline across the Alzheimer's disease spectrum.Mitochondrial lysates induce inflammation and Alzheimer's disease-relevant changes in microglial and neuronal cells.Modulators of IgG penetration through the blood-brain barrier: Implications for Alzheimer's disease immunotherapy.Towards Alzheimer's root cause: ECSIT as an integrating hub between oxidative stress, inflammation and mitochondrial dysfunction. Hypothetical role of the adapter protein ECSIT in familial and sporadic Alzheimer's disease pathogenesis.
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Neuroinflammation in Alzheimer's disease: mechanisms, pathologic consequences, and potential for therapeutic manipulation
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 2010
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Neuroinflammation in Alzheimer ...... l for therapeutic manipulation
@en
Neuroinflammation in Alzheimer ...... for therapeutic manipulation.
@nl
type
label
Neuroinflammation in Alzheimer ...... l for therapeutic manipulation
@en
Neuroinflammation in Alzheimer ...... for therapeutic manipulation.
@nl
prefLabel
Neuroinflammation in Alzheimer ...... l for therapeutic manipulation
@en
Neuroinflammation in Alzheimer ...... for therapeutic manipulation.
@nl
P2860
P356
P1476
Neuroinflammation in Alzheimer ...... l for therapeutic manipulation
@en
P2093
Kenneth Hensley
P2860
P356
10.3233/JAD-2010-1414
P577
2010-01-01T00:00:00Z