Embryonic lethality of fortilin-null mutant mice by BMP-pathway overactivation - Wikidata - awgldk - TriplyDB

Embryonic lethality of fortilin-null mutant mice by BMP-pathway overactivation

Embryonic lethality of fortilin-null mutant mice by BMP-pathway overactivation

http://www.wikidata.org/entity/Q37226391

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Core transcription factors, Oct4, Sox2 and Nanog, individually form complexes with nucleophosmin (Npm1) to control embryonic stem (ES) cell fate determination Massively parallel sequencing of human urinary exosome/microvesicle RNA reveals a predominance of non-coding RNA.Elevation of serum fortilin levels is specific for apoptosis and signifies cell death in vivo.Physical and functional antagonism between tumor suppressor protein p53 and fortilin, an anti-apoptotic protein Histamine-releasing factor has a proinflammatory role in mouse models of asthma and allergy.Fortilin potentiates the peroxidase activity of Peroxiredoxin-1 and protects against alcohol-induced liver damage in mice.Fortilin reduces apoptosis in macrophages and promotes atherosclerosis.Fortilin: A Potential Target for the Prevention and Treatment of Human Diseases.The Translationally Controlled Tumor Protein and the Cellular Response to Ionizing Radiation-Induced DNA Damage.Tumorigenesis as a process of gradual loss of original cell identity and gain of properties of neural precursor/progenitor cells.The Translational Controlled Tumour Protein TCTP: Biological Functions and Regulation.Characterization of Translationally Controlled Tumour Protein from the Sea Anemone Anemonia viridis and Transcriptome Wide Identification of Cnidarian Homologues.Genes of the transforming growth factor-beta signalling pathway are associated with pre-implantation embryonic development in cattle.

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Embryonic lethality of fortilin-null mutant mice by BMP-pathway overactivation

http://www.wikidata.org/entity/Q37226391

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on 07 February 2009

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

@cs

name

Embryonic lethality of fortilin-null mutant mice by BMP-pathway overactivation

@en

Embryonic lethality of fortilin-null mutant mice by BMP-pathway overactivation.

@nl

type

label

Embryonic lethality of fortilin-null mutant mice by BMP-pathway overactivation

@en

Embryonic lethality of fortilin-null mutant mice by BMP-pathway overactivation.

@nl

prefLabel

Embryonic lethality of fortilin-null mutant mice by BMP-pathway overactivation

@en

Embryonic lethality of fortilin-null mutant mice by BMP-pathway overactivation.

@nl

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J.BBAGEN.2009.01.012

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Biochimica et Biophysica Acta

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Embryonic lethality of fortilin-null mutant mice by BMP-pathway overactivation

@en

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Amornrat Phongdara

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Decha Pinkaew

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Ken Fujise

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Moltira Tonganunt

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Nongporn Hutadilok-Towatana

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Tomomi Kiyota

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Yoichi Kato

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Yuichi Koide

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Zhihe Liu

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P2860

Characterization of fortilin, a novel antiapoptotic protein

Bone morphogenetic proteins (BMPs) as regulators of dorsal forebrain development

The Spemann organizer signal noggin binds and inactivates bone morphogenetic protein 4

Interaction between fortilin and transforming growth factor-beta stimulated clone-22 (TSC-22) prevents apoptosis via the destabilization of TSC-22

Biological models and genes of tumor reversion: cellular reprogramming through tpt1/TCTP and SIAH-1

MCL1, a gene expressed in programmed myeloid cell differentiation, has sequence similarity to BCL2

Nucleotide sequence of a major messenger RNA for a 21 kilodalton polypeptide that is under translational control in mouse tumor cells

Noggin-mediated antagonism of BMP signaling is required for growth and patterning of the neural tube and somite

Xenopus chordin: a novel dorsalizing factor activated by organizer-specific homeobox genes

cDNA sequence coding for a translationally controlled human tumor protein

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10.1016/J.BBAGEN.2009.01.012

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