Substance P promotes expansion of human mesenteric preadipocytes through proliferative and antiapoptotic pathways. - Wikidata - awgldk - TriplyDB

Substance P promotes expansion of human mesenteric preadipocytes through proliferative and antiapoptotic pathways.

Substance P promotes expansion of human mesenteric preadipocytes through proliferative and antiapoptotic pathways.

http://www.wikidata.org/entity/Q37227039

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Mechanisms and metabolic implications of regional differences among fat depots Winning a Won Game: Caffeine Panacea for Obesity Syndemic A Western-like fat diet is sufficient to induce a gradual enhancement in fat mass over generations Fat tissue, aging, and cellular senescence.Substance P reduces TNF-α-induced apoptosis in human tenocytes through NK-1 receptor stimulation Lymphatic system: a vital link between metabolic syndrome and inflammation The Role of the Mesentery in Crohn's Disease: The Contributions of Nerves, Vessels, Lymphatics, and Fat to the Pathogenesis and Disease Course.Substance P (SP)-neurokinin-1 receptor (NK-1R) alters adipose tissue responses to high-fat diet and insulin action Role of substance P in the regulation of glucose metabolism via insulin signaling-associated pathways.Adipose tissue and inflammatory bowel disease pathogenesis Substance P mediates pro-inflammatory cytokine release form mesenteric adipocytes in Inflammatory Bowel Disease patients Substance P enhances the activation of AMPK and cellular lipid accumulation in 3T3‑L1 cells in response to high levels of glucose Akt-mediated anti-apoptotic effects of substance P in Anti-Fas-induced apoptosis of human tenocytes.Metabolic inflammation in inflammatory bowel disease: crosstalk between adipose tissue and bowel.Substance P decreases fat storage and increases adipocytokine production in 3T3-L1 adipocytes.Role of G protein-coupled receptors-microRNA interactions in gastrointestinal pathophysiology.

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P2860

Substance P promotes expansion of human mesenteric preadipocytes through proliferative and antiapoptotic pathways.

http://www.wikidata.org/entity/Q37227039

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artigo científico

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bilimsel makale

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scientific article published on 12 March 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Substance P promotes expansion ...... ve and antiapoptotic pathways.

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Substance P promotes expansion ...... ve and antiapoptotic pathways.

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type

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Substance P promotes expansion ...... ve and antiapoptotic pathways.

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Substance P promotes expansion ...... ve and antiapoptotic pathways.

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Substance P promotes expansion ...... ve and antiapoptotic pathways.

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Substance P promotes expansion ...... ve and antiapoptotic pathways.

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AJPGI.90351.2008

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American Journal of Physiology - Gastrointestinal and Liver Physiology

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Substance P promotes expansion ...... ve and antiapoptotic pathways.

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Charalabos Pothoulakis

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Collin Bowe

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Ho Kim

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Hon Wai Koon

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Iordanes Karagiannides

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James L Kirkland

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Kara Gross

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Nino Giorgadze

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Tamara Pirtskhalava

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Tamara Tchkonia

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P2860

Insulin-dependent stimulation of protein synthesis by phosphorylation of a regulator of 5'-cap function

The alpha v beta 3 integrin "vitronectin receptor"

Direct regulation of the Akt proto-oncogene product by phosphatidylinositol-3,4-bisphosphate

The protein kinase encoded by the Akt proto-oncogene is a target of the PDGF-activated phosphatidylinositol 3-kinase

The perils of portliness: causes and consequences of visceral adiposity.

Multiple signaling pathways of the insulin-like growth factor 1 receptor in protection from apoptosis.

Protein kinase C isozymes and the regulation of diverse cell responses.

Metalloproteinases and transforming growth factor-alpha mediate substance P-induced mitogen-activated protein kinase activation and proliferation in human colonocytes.

The modular phosphorylation and activation of p70s6k.

Induction of colitis causes inflammatory responses in fat depots: evidence for substance P pathways in human mesenteric preadipocytes

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10.1152/AJPGI.90351.2008

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