C3a receptor deficiency accelerates the onset of renal injury in the MRL/lpr mouse. - Wikidata - awgldk - TriplyDB

C3a receptor deficiency accelerates the onset of renal injury in the MRL/lpr mouse.

C3a receptor deficiency accelerates the onset of renal injury in the MRL/lpr mouse.

http://www.wikidata.org/entity/Q37230515

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Complement in Lupus Nephritis: New Perspectives Mechanisms of tissue injury in lupus nephritis Heat shock protein 90 inhibition by 17-DMAG lessens disease in the MRL/lpr mouse model of systemic lupus erythematosus Complement factor H deficiency accelerates development of lupus nephritis.C3a and C5a promote renal ischemia-reperfusion injury.Genetic Deficiency of Complement Component 3 Does Not Alter Disease Progression in a Mouse Model of Huntington's Disease.Treatment targets in systemic lupus erythematosus: biology and clinical perspective.Targeting the complement cascade: novel treatments coming down the pike.Complement regulation and kidney diseases: recent knowledge of the double-edged roles of complement activation in nephrology.Many drugs for many targets: novel treatments for complement-mediated glomerular disease.Monitoring C3aR Expression Using a Floxed tdTomato-C3aR Reporter Knock-in Mouse.The C3aR promotes macrophage infiltration and regulates ANCA production but does not affect glomerular injury in experimental anti-myeloperoxidase glomerulonephritis.Donor pretreatment with nebulized complement C3a receptor antagonist mitigates brain-death induced immunological injury post-lung transplant.

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C3a receptor deficiency accelerates the onset of renal injury in the MRL/lpr mouse.

http://www.wikidata.org/entity/Q37230515

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 23 January 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

C3a receptor deficiency accelerates the onset of renal injury in the MRL/lpr mouse.

@en

C3a receptor deficiency accelerates the onset of renal injury in the MRL/lpr mouse.

@nl

type

label

C3a receptor deficiency accelerates the onset of renal injury in the MRL/lpr mouse.

@en

C3a receptor deficiency accelerates the onset of renal injury in the MRL/lpr mouse.

@nl

prefLabel

C3a receptor deficiency accelerates the onset of renal injury in the MRL/lpr mouse.

@en

C3a receptor deficiency accelerates the onset of renal injury in the MRL/lpr mouse.

@nl

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J.MOLIMM.2008.12.004

P1433

Molecular Immunology

P1476

C3a receptor deficiency accelerates the onset of renal injury in the MRL/lpr mouse

@en

P2093

Baozhen Ke

http://www.w3.org/2001/XMLSchema#string

Hongyu Wang

http://www.w3.org/2001/XMLSchema#string

Michael C Braun

http://www.w3.org/2001/XMLSchema#string

Rick A Wetsel

http://www.w3.org/2001/XMLSchema#string

P2860

The C5a chemoattractant receptor mediates mucosal defence to infection

Detection of anaphylatoxin receptors on CD83+ dendritic cells derived from human skin

Lungkine, a novel CXC chemokine, specifically expressed by lung bronchoepithelial cells

Identification of a selective nonpeptide antagonist of the anaphylatoxin C3a receptor that demonstrates antiinflammatory activity in animal models

Modulation of C3a activity: internalization of the human C3a receptor and its inhibition by C5a

Surface phenotype and antigenic specificity of human interleukin 17-producing T helper memory cells

The CC chemokine CCL20 and its receptor CCR6.

Disruption of the C5a receptor gene increases resistance to acute Gram-negative bacteremia and endotoxic shock: opposing roles of C3a and C5a.

Genetics of systemic autoimmunity in mouse models of lupus.

Genetic basis of autoimmune disease in MRL/lpr mice: dissection of the complex pathological manifestations and their susceptibility loci.

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1397-1404

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scholarly article

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10.1016/J.MOLIMM.2008.12.004

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7

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46

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Scott E Wenderfer

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2009-01-23T00:00:00Z

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23938072

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19167760

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2697606

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