BMP signaling and podocyte markers are decreased in human diabetic nephropathy in association with CTGF overexpression.
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Epithelial-to-Mesenchymal Transition in Diabetic Nephropathy: Fact or Fiction?Potential Renoprotective Agents through Inhibiting CTGF/CCN2 in Diabetic NephropathyDysregulated nephrin in diabetic nephropathy of type 2 diabetes: a cross sectional studyTargeted proteomics of isolated glomeruli from the kidneys of diabetic rats: sorbin and SH3 domain containing 2 is a novel protein associated with diabetic nephropathy.Urine matrix metalloproteinase-7 and risk of kidney disease progression and mortality in type 2 diabetes.TCM-Mesh: The database and analytical system for network pharmacology analysis for TCM preparations.Urinary excretion of RAS, BMP, and WNT pathway components in diabetic kidney disease.Identification of nephropathy candidate genes by comparing sclerosis-prone and sclerosis-resistant mouse strain kidney transcriptomes.Blocking αVβ3 integrin ligand occupancy inhibits the progression of albuminuria in diabetic rats.Connective tissue growth factor (CTGF) expression modulates response to high glucose.Transcriptome analysis of human diabetic kidney diseaseConditioned mesenchymal stem cells attenuate progression of chronic kidney disease through inhibition of epithelial-to-mesenchymal transition and immune modulation.BMP9 Crosstalk with the Hippo Pathway Regulates Endothelial Cell Matricellular and Chemokine Responses.Elevated Urinary Connective Tissue Growth Factor in Diabetic Nephropathy Is Caused by Local Production and Tubular Dysfunction.Sost and its paralog Sostdc1 coordinate digit number in a Gli3-dependent manner.The role of metabolic and haemodynamic factors in podocyte injury in diabetes.Creatine pretreatment prevents birth asphyxia-induced injury of the newborn spiny mouse kidney.Nodes with high centrality in protein interaction networks are responsible for driving signaling pathways in diabetic nephropathy.Transcriptional profiling of bovine intervertebral disc cells: implications for identification of normal and degenerate human intervertebral disc cell phenotypes.Mastering a mediator: blockade of CCN-2 shows early promise in human diabetic kidney diseaseIdentification of key genes for diabetic kidney disease using biological informatics methods.Disparate phospho-Smad2 levels in advanced type 2 diabetes patients with diabetic nephropathy and early experimental db/db mouse model.Direct visualization of Smad1/5/8-mediated transcriptional activity identifies podocytes and collecting ducts as major targets of BMP signalling in healthy and diseased kidneys.Erythropoietin ameliorates podocyte injury in advanced diabetic nephropathy in the db/db mouse.BMP signaling in rats with TNBS-induced colitis following BMP7 therapy.
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P2860
BMP signaling and podocyte markers are decreased in human diabetic nephropathy in association with CTGF overexpression.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 02 March 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
BMP signaling and podocyte mar ...... tion with CTGF overexpression.
@en
BMP signaling and podocyte mar ...... tion with CTGF overexpression.
@nl
type
label
BMP signaling and podocyte mar ...... tion with CTGF overexpression.
@en
BMP signaling and podocyte mar ...... tion with CTGF overexpression.
@nl
prefLabel
BMP signaling and podocyte mar ...... tion with CTGF overexpression.
@en
BMP signaling and podocyte mar ...... tion with CTGF overexpression.
@nl
P2093
P2860
P356
P1476
BMP signaling and podocyte mar ...... tion with CTGF overexpression.
@en
P2093
Jan Willem Leeuwis
Julia Gray
Karen M Lyons
Roel Goldschmeding
Suzy V Torti
P2860
P304
P356
10.1369/JHC.2009.953224
P577
2009-03-02T00:00:00Z