Simvastatin induces a central hypotensive effect via Ras-mediated signalling to cause eNOS up-regulation. - Wikidata - awgldk - TriplyDB

Simvastatin induces a central hypotensive effect via Ras-mediated signalling to cause eNOS up-regulation.

Simvastatin induces a central hypotensive effect via Ras-mediated signalling to cause eNOS up-regulation.

http://www.wikidata.org/entity/Q37237168

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Endothelial nitric oxide synthase: a potential therapeutic target for cerebrovascular diseases Renal Denervation Improves the Baroreflex and GABA System in Chronic Kidney Disease-induced Hypertension Endothelial and neuronal nitric oxide synthases variably modulate the oestrogen-mediated control of blood pressure and cardiovascular autonomic control Mitochondria and Reactive Oxygen Species Contribute to Neurogenic Hypertension.The potential of stimulating nitric oxide formation in the treatment of hypertension.Effects of ticagrelor in a mouse model of ischemic stroke.Vascular injury in diabetic db/db mice is ameliorated by atorvastatin: role of Rac1/2-sensitive Nox-dependent pathways.Simvastatin and atorvastatin facilitates amyloid β-protein degradation in extracellular spaces by increasing neprilysin secretion from astrocytes through activation of MAPK/Erk1/2 pathways.

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Simvastatin induces a central hypotensive effect via Ras-mediated signalling to cause eNOS up-regulation.

http://www.wikidata.org/entity/Q37237168

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article científic

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article scientifique

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artigo científico

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bilimsel makale

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scientific article published on October 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Simvastatin induces a central ...... g to cause eNOS up-regulation.

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British Journal of Pharmacology

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Simvastatin induces a central ...... g to cause eNOS up-regulation.

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Chien-Feng Chang

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Ching-Jiunn Tseng

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Gwo-Ching Sun

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Ling-Zong Hong

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Michael Hsiao

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Pei-Jung Lu

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Pei-Wen Cheng

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Tung-Chen Yeh

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Wen-Han Cheng

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Wen-Yu Ho

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Ras activation of a Rac1 exchange factor, Tiam1, mediates neurotrophin-3-induced Schwann cell migration

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The sympathetic control of blood pressure.

The HMG-CoA reductase inhibitor simvastatin activates the protein kinase Akt and promotes angiogenesis in normocholesterolemic animals.

Animal research: reporting in vivo experiments: the ARRIVE guidelines.

K- and N-Ras are geranylgeranylated in cells treated with farnesyl protein transferase inhibitors.

Relative lipophilicities, solubilities, and structure-pharmacological considerations of 3-hydroxy-3-methylglutaryl-coenzyme A (HMG-CoA) reductase inhibitors pravastatin, lovastatin, mevastatin, and simvastatin.

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847-858

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10.1111/BPH.12317

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2013-10-01T00:00:00Z

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257603229

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23889671

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3799598

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