Signaling by hypoxia-inducible factors is critical for ovulation in mice
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Control of ovulation in mice by progesterone receptor-regulated gene networksEndocrine and local control of the primate corpus luteumConversion of a disulfide bond into a thioacetal group during echinomycin biosynthesisThe Ovarian Antral Follicle: Living on the Edge of Hypoxia or Not?Control of oocyte release by progesterone receptor-regulated gene expression.Hypoxia promotes progesterone synthesis during luteinization in bovine granulosa cellsThe presence of the Y-chromosome, not the absence of the second X-chromosome, alters the mRNA levels stored in the fully grown XY mouse oocyte.Endothelins in regulating ovarian and oviductal functionDynamics of the transcriptome in the primate ovulatory follicle.Suitable housekeeping genes for normalization of transcript abundance analysis by real-time RT-PCR in cultured bovine granulosa cells during hypoxia and differential cell plating density.Immunohistochemical assessment of intrinsic and extrinsic markers of hypoxia in reproductive tissue: differential expression of HIF1α and HIF2α in rat oviduct and endometrium.Disruptions in follicle cell functions in the ovaries of rhesus monkeys during summer.X-linked lymphocyte regulated gene 5c-like (Xlr5c-like) is a novel target of progesterone action in granulosa cells of periovulatory rat ovariesEffects of echinomycin on endothelin-2 expression and ovulation in immature rats primed with gonadotropins.Endothelin-2, the forgotten isoform: emerging role in the cardiovascular system, ovarian development, immunology and cancer.In vivo imaging reveals an essential role of vasoconstriction in rupture of the ovarian follicle at ovulation.Ulipristal blocks ovulation by inhibiting progesterone receptor-dependent pathways intrinsic to the ovary.Cytokines and angiogenesis in the corpus luteumInduction of altered gene expression profiles in cultured bovine granulosa cells at high cell density.Hypoxia-inducible factor 1 mediates hypoxia-enhanced synthesis of progesterone during luteinization of granulosa cells.The critical roles of progesterone receptor (PGR) in ovulation, oocyte developmental competence and oviductal transport in mammalian reproduction.Transcriptomic signatures for ovulation in vertebrates.Expression and clinical significance of the HIF-1a/ET-2 signaling pathway during the development and treatment of polycystic ovary syndrome.Transcriptome analysis of human cumulus cells reveals hypoxia as the main determinant of follicular senescence.Contribution of hypoxia-inducible factor-1α to transcriptional regulation of vascular endothelial growth factor in bovine developing luteal cells.Growth and regression in bovine corpora lutea: regulation by local survival and death pathways.Hemoglobin: a gas transport molecule that is hormonally regulated in the ovarian follicle in mice and humans.BNIP3 expression in bovine follicle and corpus luteum.Oxidized low-density lipoprotein suppresses mouse granulosa cell differentiation through disruption of the hypoxia-inducible factor 1 pathway.Lactate promotes specific differentiation in bovine granulosa cells depending on lactate uptake thus mimicking an early post-LH stage.The expression of CXCR4 is induced by the luteinizing hormone surge and mediated by progesterone receptors in human preovulatory granulosa cells.Mechanistic target of rapamycin (MTOR) signaling during ovulation in mice.Low Oxygen Levels Induce Early Luteinization Associated Changes in Bovine Granulosa Cells
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Signaling by hypoxia-inducible factors is critical for ovulation in mice
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 26 March 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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Signaling by hypoxia-inducible factors is critical for ovulation in mice
@en
Signaling by hypoxia-inducible factors is critical for ovulation in mice.
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Signaling by hypoxia-inducible factors is critical for ovulation in mice
@en
Signaling by hypoxia-inducible factors is critical for ovulation in mice.
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Signaling by hypoxia-inducible factors is critical for ovulation in mice
@en
Signaling by hypoxia-inducible factors is critical for ovulation in mice.
@nl
P2093
P2860
P356
P1433
P1476
Signaling by hypoxia-inducible factors is critical for ovulation in mice
@en
P2093
Indrani C Bagchi
Jaeyeon Kim
Milan K Bagchi
P2860
P304
P356
10.1210/EN.2008-0948
P407
P577
2009-03-26T00:00:00Z