REVIEW paper: pathophysiology of myocardial reperfusion injury: the role of genetically engineered mouse models. - Wikidata - awgldk - TriplyDB

REVIEW paper: pathophysiology of myocardial reperfusion injury: the role of genetically engineered mouse models.

REVIEW paper: pathophysiology of myocardial reperfusion injury: the role of genetically engineered mouse models.

http://www.wikidata.org/entity/Q37251495

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Inhibition of the Mitochondrial Permeability Transition for Cytoprotection: Direct versus Indirect Mechanisms Genetics of peripheral artery disease Alterations in Circulating Adhesion Molecules in Acute Myocardial Infarction before and after Thrombolysis with Streptokinase.Tanshinone‑IIA attenuates the deleterious effects of oxidative stress in osteoporosis through the NF‑κB signaling pathway.

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REVIEW paper: pathophysiology of myocardial reperfusion injury: the role of genetically engineered mouse models.

http://www.wikidata.org/entity/Q37251495

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on September 2008

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

REVIEW paper: pathophysiology ...... cally engineered mouse models.

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REVIEW paper: pathophysiology ...... cally engineered mouse models.

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type

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REVIEW paper: pathophysiology ...... cally engineered mouse models.

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REVIEW paper: pathophysiology ...... cally engineered mouse models.

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REVIEW paper: pathophysiology ...... cally engineered mouse models.

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REVIEW paper: pathophysiology ...... cally engineered mouse models.

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Veterinary Pathology

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REVIEW paper: pathophysiology ...... cally engineered mouse models.

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A Ramachandran

http://www.w3.org/2001/XMLSchema#string

D J Lefer

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S Jha

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P2860

Molecular characterization of mitochondrial apoptosis-inducing factor

Caspase-12 mediates endoplasmic-reticulum-specific apoptosis and cytotoxicity by amyloid-beta

Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule

Immunolocalization of von Willebrand protein in Weibel-Palade bodies of human endothelial cells

X-linked IAP is a direct inhibitor of cell-death proteases

The c-IAP-1 and c-IAP-2 proteins are direct inhibitors of specific caspases

IAPs block apoptotic events induced by caspase-8 and cytochrome c by direct inhibition of distinct caspases

GMP-140, a platelet alpha-granule membrane protein, is also synthesized by vascular endothelial cells and is localized in Weibel-Palade bodies

The Fas signaling pathway: more than a paradigm

Role of the arginine-nitric oxide pathway in the regulation of vascular smooth muscle cell proliferation

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698-706

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scholarly article

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10.1354/VP.45-5-698

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5

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45

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2008-09-01T00:00:00Z

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23192258

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18725477

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pathophysiology

reperfusion injury