Global cellular response to chemotherapy-induced apoptosis.
about
Spreading the word: non-autonomous effects of apoptosis during development, regeneration and diseaseCGG Repeats in the 5'UTR of FMR1 RNA Regulate Translation of Other RNAs Localized in the Same RNA GranulesThe use of duplex-specific nuclease in ribosome profiling and a user-friendly software package for Ribo-seq data analysis.Circulating proteolytic signatures of chemotherapy-induced cell death in humans discovered by N-terminal labeling.Cross-species proteomics reveals specific modulation of signaling in cancer and stromal cells by phosphoinositide 3-kinase (PI3K) inhibitorsUnraveling the mechanism of cell death induced by chemical fibrils.Global analysis of cellular proteolysis by selective enzymatic labeling of protein N-termini.The Pan-Cancer analysis of pseudogene expression reveals biologically and clinically relevant tumour subtypes.MYC regulates the non-coding transcriptome.Attenuation of nonsense-mediated mRNA decay facilitates the response to chemotherapeutics.Oxygen and glucose deprivation induces widespread alterations in mRNA translation within 20 minutesThe Role of Translational Regulation in Survival after Radiation Damage; an Opportunity for Proteomics Analysis.Proteoform-Specific Insights into Cellular Proteome Regulation.Antibody-drug conjugate targeting CD46 eliminates multiple myeloma cells.Interactome disassembly during apoptosis occurs independent of caspase cleavage.Bortezomib-induced heat shock response protects multiple myeloma cells and is activated by heat shock factor 1 serine 326 phosphorylation.When Cancer Fights Back: Multiple Myeloma, Proteasome Inhibition, and the Heat-Shock Response.Comparative Analysis of Mitochondrial N-Termini from Mouse, Human, and Yeast.Time-Resolved Proteomics Extends Ribosome Profiling-Based Measurements of Protein Synthesis Dynamics.Evaluation of the Anti-proliferative Effects of Ophiocoma erinaceus Methanol Extract Against Human Cervical Cancer Cells.Caspases and their substrates.Translational reprogramming of colorectal cancer cells induced by 5-fluorouracil through a miRNA-dependent mechanism.eIF1 modulates the recognition of suboptimal translation initiation sites and steers gene expression via uORFsDissecting apoptosis the omics way.GWIPS-viz: 2018 update.Hyaluronan and proteoglycan link protein 1 (HAPLN1) activates bortezomib-resistant NF-κB activity and increases drug resistance in multiple myeloma.Targeting RAS-driven human cancer cells with antibodies to upregulated and essential cell-surface proteins.
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Global cellular response to chemotherapy-induced apoptosis.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 29 October 2013
@en
vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Global cellular response to chemotherapy-induced apoptosis.
@en
Global cellular response to chemotherapy-induced apoptosis.
@nl
type
label
Global cellular response to chemotherapy-induced apoptosis.
@en
Global cellular response to chemotherapy-induced apoptosis.
@nl
prefLabel
Global cellular response to chemotherapy-induced apoptosis.
@en
Global cellular response to chemotherapy-induced apoptosis.
@nl
P2093
P2860
P50
P356
P1433
P1476
Global cellular response to chemotherapy-induced apoptosis.
@en
P2093
Anatoly Urisman
Arun P Wiita
James A Wells
P2860
P304
P356
10.7554/ELIFE.01236
P407
P577
2013-10-29T00:00:00Z